Telomere length in acute promyelocytic leukemia (2013)
- Authors:
- USP affiliated authors: REGO, EDUARDO MAGALHÃES - FMRP ; RODRIGUES, RODRIGO DO TOCANTINS CALADO DE SALOMA - FMRP
- Unidade: FMRP
- Assunto: LEUCEMIA
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 2013
- Source:
- Conference titles: Annual Meeting of American Society of Hematology
-
ABNT
SCATENA, Natália Ferreira et al. Telomere length in acute promyelocytic leukemia. Blood. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 01 abr. 2026. , 2013 -
APA
Scatena, N. F., Araújo, A. R. L., Paiva, R. M. A., Rego, E. M., & Calado, R. T. (2013). Telomere length in acute promyelocytic leukemia. Blood. Washington: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Scatena NF, Araújo ARL, Paiva RMA, Rego EM, Calado RT. Telomere length in acute promyelocytic leukemia. Blood. 2013 ; 122( 21):[citado 2026 abr. 01 ] -
Vancouver
Scatena NF, Araújo ARL, Paiva RMA, Rego EM, Calado RT. Telomere length in acute promyelocytic leukemia. Blood. 2013 ; 122( 21):[citado 2026 abr. 01 ] - Defective telomere elongation and hematopoiesis from telomerase-mutant aplastic anemia iPSCs
- Cell senescence and malignant transformation in the inherited bone marrow failure syndromes: overlapping pathophysiology with therapeutic implications
- Short telomeres result in chromosomal instability in hematopoietic cells and precede malignant evolution in human aplastic anemia
- Exome sequencing reveals a thrombopoietin ligand mutation in a Micronesian family with autosomal recessive aplastic anemia
- Telomere shortening in hepatocyte proliferation and predisposition to alcohol-induced steatosis
- Telomerase enzyme deficiency promotes metabolic dysfunction in murine hepatocytes upon dietary stress
- Telomere dynamics in iPS cells derived from human patients with telomerase mutations
- Predictors of early mortality after rabbit antithymocyte globulin as first-line treatment in severe aplastic anemia
- Células reprogramadas. [Depoimento a Hérika Dias e Luis Ribeiro]
- A mutation in the CCAAT box of the TERC promoter region causes telomere disease
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