BCL2A1a over-expression in murine hematopoietic stem and progenitor cells decreases apoptosis and results in hematopoietic transformation (2012)
- Authors:
- Autor USP: RODRIGUES, RODRIGO DO TOCANTINS CALADO DE SALOMA - FMRP
- Unidade: FMRP
- DOI: 10.1371/journal.pone.0048267
- Subjects: APOPTOSE; EXPRESSÃO GÊNICA; HEMATOPOESE; LEUCEMIA (GENÉTICA)
- Language: Inglês
- Imprenta:
- Publisher place: San Francisco
- Date published: 2012
- Source:
- Este periódico é de acesso aberto
- Este artigo é de acesso aberto
- URL de acesso aberto
- Cor do Acesso Aberto: gold
- Licença: cc-by
-
ABNT
MÉTAIS, Jean-Yves et al. BCL2A1a over-expression in murine hematopoietic stem and progenitor cells decreases apoptosis and results in hematopoietic transformation. PLoS One, v. 7, n. 10, p. e48267-1 - e48267-12, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0048267. Acesso em: 27 dez. 2025. -
APA
Métais, J. -Y., Winkler, T., Geyer, J. T., Calado, R. T., Aplan, P. D., Eckhaus, M. A., & Dunbar, C. E. (2012). BCL2A1a over-expression in murine hematopoietic stem and progenitor cells decreases apoptosis and results in hematopoietic transformation. PLoS One, 7( 10), e48267-1 - e48267-12. doi:10.1371/journal.pone.0048267 -
NLM
Métais J-Y, Winkler T, Geyer JT, Calado RT, Aplan PD, Eckhaus MA, Dunbar CE. BCL2A1a over-expression in murine hematopoietic stem and progenitor cells decreases apoptosis and results in hematopoietic transformation [Internet]. PLoS One. 2012 ; 7( 10): e48267-1 - e48267-12.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1371/journal.pone.0048267 -
Vancouver
Métais J-Y, Winkler T, Geyer JT, Calado RT, Aplan PD, Eckhaus MA, Dunbar CE. BCL2A1a over-expression in murine hematopoietic stem and progenitor cells decreases apoptosis and results in hematopoietic transformation [Internet]. PLoS One. 2012 ; 7( 10): e48267-1 - e48267-12.[citado 2025 dez. 27 ] Available from: https://doi.org/10.1371/journal.pone.0048267 - Androgen treatment mitigates hematopoietic cell telomere attrition in vivo
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Informações sobre o DOI: 10.1371/journal.pone.0048267 (Fonte: oaDOI API)
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