Impairment of the hematological response and interleukin-1ß production in protein-energy malnourished mice after endotoxemia with lipopolysaccharide (2012)
- Authors:
- USP affiliated authors: FOCK, RICARDO AMBROSIO - FCF ; GARCIA, PRIMAVERA BORELLI - FCF
- Unidade: FCF
- DOI: 10.1590/S0100-879X2012007500151
- Subjects: LIPOPOLISSACARÍDEOS; CITOCININAS; INTERLEUCINA 1
- Language: Inglês
- Imprenta:
- Publisher place: Ribeirão Preto
- Date published: 2012
- Source:
- Título: Brazilian Journal of Medical and Biological Research
- ISSN: 0100-879X
- Volume/Número/Paginação/Ano: v. 45, n. 12, p. 1163-1171, 2012
- Este artigo possui versão em acesso aberto
- URL de acesso aberto
- PDF de acesso aberto
- Versão do Documento: Versão publicada (Published version)
-
Status: Artigo publicado em periódico de acesso aberto (Gold Open Access) -
ABNT
FOCK, Ricardo Ambrósio et al. Impairment of the hematological response and interleukin-1ß production in protein-energy malnourished mice after endotoxemia with lipopolysaccharide. Brazilian Journal of Medical and Biological Research, v. 45, n. 12, p. 1163-1171, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500151. Acesso em: 15 mar. 2026. -
APA
Fock, R. A., Vinolo, M. A. R., Blatt, S. L., & Borelli, P. (2012). Impairment of the hematological response and interleukin-1ß production in protein-energy malnourished mice after endotoxemia with lipopolysaccharide. Brazilian Journal of Medical and Biological Research, 45( 12), 1163-1171. doi:10.1590/S0100-879X2012007500151 -
NLM
Fock RA, Vinolo MAR, Blatt SL, Borelli P. Impairment of the hematological response and interleukin-1ß production in protein-energy malnourished mice after endotoxemia with lipopolysaccharide [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 12): 1163-1171.[citado 2026 mar. 15 ] Available from: https://doi.org/10.1590/S0100-879X2012007500151 -
Vancouver
Fock RA, Vinolo MAR, Blatt SL, Borelli P. Impairment of the hematological response and interleukin-1ß production in protein-energy malnourished mice after endotoxemia with lipopolysaccharide [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 12): 1163-1171.[citado 2026 mar. 15 ] Available from: https://doi.org/10.1590/S0100-879X2012007500151 - Intravenous glutamine administration modulates TNF-α/IL-10 ratio and attenuates NFkB phosphorylation in a protein malnutrition model
- TNF receptor expression by macrophages in protein malnourished mice
- Protein malnutrition: effects on hematopoietic stem cell frequency, cell cycle and chemokine receptors expression in the endosteal niche regulation
- Bone marrow endhotelial-like cells produce G-CSF in order to maintain hematopoiesis during protein-energy malnutrition states
- Protein malnutrition: effects on hematopoietic stem cell frequency, cell cycle and chemokine receptors expression in the endosteal niche regulation
- Bone marrow multipotent mesenchymal stem cells are committed to adipogenic differentiation in protein malnutrition states
- Femoral microarchitecture and endosteal niche alterations in protein malnutrition: possible effects on hematopoiesis?
- Induction of autophagy by Akt/Mtor pathway in the bone marrow due to protein malnutrition
- A célula-tronco e seu nicho
- Malnourished mice display an impaired hematologic response to granulocyte colony-stimulating factor administration
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