Intravenous glutamine administration modulates TNF-α/IL-10 ratio and attenuates NFkB phosphorylation in a protein malnutrition model (2016)
- Authors:
- USP affiliated authors: GARCIA, PRIMAVERA BORELLI - FCF ; FOCK, RICARDO AMBROSIO - FCF
- Unidade: FCF
- DOI: 10.1007/s10753-016-0424-1
- Subjects: GLUTAMINA; DESNUTRIÇÃO PROTEICO-ENERGÉTICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Inflammation
- ISSN: 0360-3997
- Volume/Número/Paginação/Ano: v. 39, n. 6, p. 1883-1891, 2016
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão submetida (Pré-print)
- Acessar versão aberta:
-
ABNT
SANTOS, Andressa Cristina Antunes et al. Intravenous glutamine administration modulates TNF-α/IL-10 ratio and attenuates NFkB phosphorylation in a protein malnutrition model. Inflammation, v. 39, n. 6, p. 1883-1891, 2016Tradução . . Disponível em: https://doi.org/10.1007/s10753-016-0424-1. Acesso em: 08 abr. 2026. -
APA
Santos, A. C. A., Correia, C. A., Oliveira, D. C. de, Nogueira-Pedro, A., Borelli, P., & Fock, R. A. (2016). Intravenous glutamine administration modulates TNF-α/IL-10 ratio and attenuates NFkB phosphorylation in a protein malnutrition model. Inflammation, 39( 6), 1883-1891. doi:10.1007/s10753-016-0424-1 -
NLM
Santos ACA, Correia CA, Oliveira DC de, Nogueira-Pedro A, Borelli P, Fock RA. Intravenous glutamine administration modulates TNF-α/IL-10 ratio and attenuates NFkB phosphorylation in a protein malnutrition model [Internet]. Inflammation. 2016 ; 39( 6): 1883-1891.[citado 2026 abr. 08 ] Available from: https://doi.org/10.1007/s10753-016-0424-1 -
Vancouver
Santos ACA, Correia CA, Oliveira DC de, Nogueira-Pedro A, Borelli P, Fock RA. Intravenous glutamine administration modulates TNF-α/IL-10 ratio and attenuates NFkB phosphorylation in a protein malnutrition model [Internet]. Inflammation. 2016 ; 39( 6): 1883-1891.[citado 2026 abr. 08 ] Available from: https://doi.org/10.1007/s10753-016-0424-1 - TNF receptor expression by macrophages in protein malnourished mice
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- Bone marrow endhotelial-like cells produce G-CSF in order to maintain hematopoiesis during protein-energy malnutrition states
- Protein malnutrition: effects on hematopoietic stem cell frequency, cell cycle and chemokine receptors expression in the endosteal niche regulation
- Bone marrow multipotent mesenchymal stem cells are committed to adipogenic differentiation in protein malnutrition states
- Femoral microarchitecture and endosteal niche alterations in protein malnutrition: possible effects on hematopoiesis?
- Induction of autophagy by Akt/Mtor pathway in the bone marrow due to protein malnutrition
- A célula-tronco e seu nicho
- Malnourished mice display an impaired hematologic response to granulocyte colony-stimulating factor administration
- L-Glutamine in vitro modulates some immunomodulatory properties of bone marrow mesenchymal stem cells
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