T cell clonality and autoantigen recogniton in rheumatic heart disease (2002)
- Autor:
- Autor USP: KALIL FILHO, JORGE ELIAS - FM
- Unidade: FM
- Assunto: CARDIOPATIA REUMÁTICA (IMUNOLOGIA)
- Language: Inglês
- Imprenta:
- Source:
- Título: International journal on immunorehabilitation
- ISSN: 1562-3629
- Volume/Número/Paginação/Ano: v. 4, n. 3, 2002
- Conference titles: Proceedings of the world congress on immunopathology
-
ABNT
GUILHERME, L. T cell clonality and autoantigen recogniton in rheumatic heart disease. International journal on immunorehabilitation. Moscou: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 04 out. 2024. , 2002 -
APA
Guilherme, L. (2002). T cell clonality and autoantigen recogniton in rheumatic heart disease. International journal on immunorehabilitation. Moscou: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Guilherme L. T cell clonality and autoantigen recogniton in rheumatic heart disease. International journal on immunorehabilitation. 2002 ; 4( 3):[citado 2024 out. 04 ] -
Vancouver
Guilherme L. T cell clonality and autoantigen recogniton in rheumatic heart disease. International journal on immunorehabilitation. 2002 ; 4( 3):[citado 2024 out. 04 ] - Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target
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- Locally produced IL-15 may underlie the predominance of CD8+IL2RALPHA- T cells ar heart lesions of human Chagas Disease cardiomyopathy
- Transplante cardíaco para tratamento da miocardiopatia chagásica
- Distribution of primary immunodeficiency diseases at University of Sao Paulo Medical School, Brazil
- Comparison between indirect allo-proliferative response and binding prediction to HLA-DR molecules by tepitope analysis
- Sensitization to aeroallergens: urban/rural differences in São Paulo-Brazil
- Rheumatic heart disease: intralesional T cells recognize peptides from cardiac myosin beta chain
- Can heat shock protein be used to induce transplantation tolerance to murine allograft?
- Identification of public expansions in the peripheral T cell repertoire reactive to "Self" H-2'K POT. 1'peptides, following rejection of grafts without H-2 disparaty, in tapi mutant mice
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