Molecular basis of peripheral blockade of inflammatory pain by opioids (2010)
- Authors:
- USP affiliated authors: CUNHA, THIAGO MATTAR - FMRP ; CHIAVEGATTO, SILVANA - ICB ; CUNHA, FERNANDO DE QUEIROZ - FMRP ; FERREIRA, SERGIO HENRIQUE - FMRP
- Unidades: FMRP; ICB
- Subjects: DOR (TRATAMENTO); ANALGESIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Basic and Clinical Pharmacology and Toxicology
- ISSN: 1742-7851
- Volume/Número/Paginação/Ano: v. 107, suppl. 1, p. 118, res. 2402, 2010
- Conference titles: World Congress of Basic and Clinical Pharmacology
-
ABNT
CUNHA, Thiago Mattar et al. Molecular basis of peripheral blockade of inflammatory pain by opioids. Basic and Clinical Pharmacology and Toxicology. Oxford: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 01 jan. 2026. , 2010 -
APA
Cunha, T. M., Roman-Campos, D., Lotufo, C., Duarte, H., Souza, G., Verri Júnior, W., et al. (2010). Molecular basis of peripheral blockade of inflammatory pain by opioids. Basic and Clinical Pharmacology and Toxicology. Oxford: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. -
NLM
Cunha TM, Roman-Campos D, Lotufo C, Duarte H, Souza G, Verri Júnior W, Chiavegatto S, Cruz J, Teixeira M, Hothershall J, Cunha F de Q, Ferreira SH. Molecular basis of peripheral blockade of inflammatory pain by opioids. Basic and Clinical Pharmacology and Toxicology. 2010 ; 107 118.[citado 2026 jan. 01 ] -
Vancouver
Cunha TM, Roman-Campos D, Lotufo C, Duarte H, Souza G, Verri Júnior W, Chiavegatto S, Cruz J, Teixeira M, Hothershall J, Cunha F de Q, Ferreira SH. Molecular basis of peripheral blockade of inflammatory pain by opioids. Basic and Clinical Pharmacology and Toxicology. 2010 ; 107 118.[citado 2026 jan. 01 ] - Morphine peripheral analgesia depends on activation of the PI3Kγ/AKT/nNOS/NO/KATP signaling pathway
- The activation of peripheral kappa opioid receptors directly blocks inflammatory hyperalgesia through the stimulation of PI3Kgamma/AKT?NO signaling pathway
- Acetic acid- and phenyl-p-benzoquinone-induced overt pain-like behavior depends on spinal activation of MAP kinases, P'I IND.3'K and microglia in mice
- Pharmacological beta-adrenergic receptor activation attenuates neutrophil recruitment by a mechanism dependent on nicotinic receptor and the spleen
- Acetic acid and Phenyl-P-Benzoquinone-induced overt pain-like behavior depends on spinal activation of map kinases and PI3K in mice
- Participation of nonpeptidergic nociceptors in inflammatory pain
- The role of PAF/PAFR signaling in zymosan-induced articular inflammatory hyperalgesia
- Fractalkine mediates inflammatory pain through activation of satellite glial cells
- Role of CCR2 de novo expression in neutrophils in experimental arthritis
- Kaurenoic acid from Sphagneticola trilobata inhibits inflammatory pain: effect on cytokine production and activation of the NO–Cyclic GMP–protein kinase G–ATP-sensitive potassium channel signaling pathway
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