Tempol slightly ameliorates amyotrophic lateral sclerosis in an animal model (2010)
- Authors:
- USP affiliated authors: LINARES, EDLAINE - IQ ; CHADI, GERSON - FM ; AUGUSTO, OHARA - IQ
- Unidades: IQ; FM
- Subjects: DOENÇAS NEURODEGENERATIVAS; SUPERÓXIDO DISMUTASE
- Language: Inglês
- Imprenta:
- Source:
- Título: Abstracts
- Conference titles: Annual Meeting of Brazilian Biochemistry and Molecular Biology (SBBq)
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ABNT
SEIXAS, L. V et al. Tempol slightly ameliorates amyotrophic lateral sclerosis in an animal model. 2010, Anais.. São Paulo: SBBq, 2010. . Acesso em: 26 jan. 2026. -
APA
Seixas, L. V., Linares, E., Chadi, G., Maximino, J. R., Duobles, T., & Augusto, O. (2010). Tempol slightly ameliorates amyotrophic lateral sclerosis in an animal model. In Abstracts. São Paulo: SBBq. -
NLM
Seixas LV, Linares E, Chadi G, Maximino JR, Duobles T, Augusto O. Tempol slightly ameliorates amyotrophic lateral sclerosis in an animal model. Abstracts. 2010 ;[citado 2026 jan. 26 ] -
Vancouver
Seixas LV, Linares E, Chadi G, Maximino JR, Duobles T, Augusto O. Tempol slightly ameliorates amyotrophic lateral sclerosis in an animal model. Abstracts. 2010 ;[citado 2026 jan. 26 ] - Tempol ameliorates murine viral encephalomyelitis by preserving the blood-brain barrier, reducing viral load and lessening inflammation
- Tempol ameliorates murine viral encephalomyelitis by preserving the blood–brain barrier, reducing viral load, and lessening inflammation
- Tempol ameliorates murine viral encephalomylitis by preserving the blood brain barrier, reducing viral load and lessening inflammation
- Maintenance of blood-brain barrier integrity and decreased severity of experimental multiple sclerosis in mice treated with tempol
- Kinetics and mechanism of the reaction of a nitroxide radical (Tempol) with a phenolic antioxidant
- Tempol inhibits macrophage leishmanicidal activity in infected mice
- Argininosuccinate synthetase is a functional target for a snake venom anti-hypertensive peptide: role in arginine and nitric oxide production
- EPR studies of radical production In vivo by lypopolysaccharide. Potential role of iron mobilized from iron-nitrosyl complexes
- Maitenance of blood-brain barrier integrity and decreased severity of experimental multiple sclerosis in tempol-treated mice
- Inhibition of in vivo leishmanicidal mechanisms by tempol: Nitric oxide down-regulation and oxidant scavenging
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