Tempol ameliorates murine viral encephalomyelitis by preserving the blood-brain barrier, reducing viral load and lessening inflammation (2009)
- Authors:
- USP affiliated authors: AUGUSTO, OHARA - IQ ; LINARES, EDLAINE - IQ ; CHADI, GERSON - FM
- Unidades: IQ; FM
- Subjects: ENCEFALOMIELITE ANIMAL; BIOQUÍMICA; DOENÇAS DO SISTEMA NERVOSO CENTRAL
- Language: Inglês
- Imprenta:
- Source:
- Título: Free Radical Biology & Medicine
- Volume/Número/Paginação/Ano: v. 47, suppl. 1, p. S136 res. 362, 2009
- Conference titles: Annual Meeting of the Society for Free Radical and Medicine
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ABNT
AUGUSTO, Ohara et al. Tempol ameliorates murine viral encephalomyelitis by preserving the blood-brain barrier, reducing viral load and lessening inflammation. Free Radical Biology & Medicine. New York: Instituto de Química, Universidade de São Paulo. . Acesso em: 09 nov. 2024. , 2009 -
APA
Augusto, O., Tsuhako, M. H., Linares, E., Chadi, G., & Pereira, C. A. (2009). Tempol ameliorates murine viral encephalomyelitis by preserving the blood-brain barrier, reducing viral load and lessening inflammation. Free Radical Biology & Medicine. New York: Instituto de Química, Universidade de São Paulo. -
NLM
Augusto O, Tsuhako MH, Linares E, Chadi G, Pereira CA. Tempol ameliorates murine viral encephalomyelitis by preserving the blood-brain barrier, reducing viral load and lessening inflammation. Free Radical Biology & Medicine. 2009 ; 47 S136 res. 362.[citado 2024 nov. 09 ] -
Vancouver
Augusto O, Tsuhako MH, Linares E, Chadi G, Pereira CA. Tempol ameliorates murine viral encephalomyelitis by preserving the blood-brain barrier, reducing viral load and lessening inflammation. Free Radical Biology & Medicine. 2009 ; 47 S136 res. 362.[citado 2024 nov. 09 ] - Tempol ameliorates murine viral encephalomylitis by preserving the blood brain barrier, reducing viral load and lessening inflammation
- Tempol ameliorates murine viral encephalomyelitis by preserving the blood–brain barrier, reducing viral load, and lessening inflammation
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- EPR studies of in vivo radical production by lipopolysaccharide: potential role of iron mobilized from iron-nitrosyl complexes
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