CCR2 deficiency results in increased osteolysis in experimental periapical lesions in mice (2010)
- Authors:
- USP affiliated authors: GARLET, GUSTAVO POMPERMAIER - FOB ; CAMPOS, MARIO JULIO AVILA - ICB ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidades: FOB; ICB; FMRP
- DOI: 10.1016/j.joen.2009.09.004
- Subjects: OSTEÓLISE ANIMAL (MICROBIOLOGIA); RATOS; LESÕES PERIAPICAIS; CITOCINAS (IMUNOLOGIA)
- Language: Inglês
- Imprenta:
- Source:
- Título: Journal of Endodontics
- ISSN: 0099-2399
- Volume/Número/Paginação/Ano: v. 36, n. 2, p. 244-250, Feb. 2010
- Status:
- Artigo possui versão em acesso aberto em repositório (Green Open Access)
- Versão do Documento:
- Versão submetida (Pré-print)
- Acessar versão aberta:
-
ABNT
GARLET, Thiago Pompermaier et al. CCR2 deficiency results in increased osteolysis in experimental periapical lesions in mice. Journal of Endodontics, v. 36, n. 2, p. 244-250, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.joen.2009.09.004. Acesso em: 31 mar. 2026. -
APA
Garlet, T. P., Fukada, S. Y., Saconato, I. F., Avila-Campos, M. J., Silva, T. A. da, Garlet, G. P., & Cunha, F. de Q. (2010). CCR2 deficiency results in increased osteolysis in experimental periapical lesions in mice. Journal of Endodontics, 36( 2), 244-250. doi:10.1016/j.joen.2009.09.004 -
NLM
Garlet TP, Fukada SY, Saconato IF, Avila-Campos MJ, Silva TA da, Garlet GP, Cunha F de Q. CCR2 deficiency results in increased osteolysis in experimental periapical lesions in mice [Internet]. Journal of Endodontics. 2010 ; 36( 2): 244-250.[citado 2026 mar. 31 ] Available from: https://doi.org/10.1016/j.joen.2009.09.004 -
Vancouver
Garlet TP, Fukada SY, Saconato IF, Avila-Campos MJ, Silva TA da, Garlet GP, Cunha F de Q. CCR2 deficiency results in increased osteolysis in experimental periapical lesions in mice [Internet]. Journal of Endodontics. 2010 ; 36( 2): 244-250.[citado 2026 mar. 31 ] Available from: https://doi.org/10.1016/j.joen.2009.09.004 - The dual role of p55 tumour necrosis factor-a receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction
- The essential role of IFN-'gama' in the control of lethal Aggregatibacter actinomycetemcomitans infection in mice
- iNOS-derived nitric oxide modulates infection-stimulated bone loss
- Periodontal pathogens directly promote autoimmune experimental arthritis by inducing a TLR2- and IL-1–driven Th17 response
- Role of CCR2 in orthodontic tooth movement
- Differential expression of osteoblast and osteoclast chemmoatractants in compression and tension sides during orthodontic movement
- Tumor necrosis factor-alpha -308G/A single nucleotide polymorphism and red-complex periodontopathogens are independently associated with increased levels of tumor necrosis factor-alpha in diseased periodontal tissues
- Experimental periodontitis in mice selected for maximal or minimal inflammatory reactions: increased inflammatory immune responsiveness drives increased alveolar bone loss without enhancing the control of periodontal infection
- The aggravation of arthritis by periodontitis is dependent of IL-17receptor A activation
- Cytokine pattern determines the progression of experimental periodontal disease induced by Actinobacillus actinomycetemcomitans through the modulation of MMPs, RANKL, and their physiological inhibitors
Informações sobre a disponibilidade de versões do artigo em acesso aberto coletadas automaticamente via oaDOI API (Unpaywall).
Por se tratar de integração com serviço externo, podem existir diferentes versões do trabalho (como preprints ou postprints), que podem diferir da versão publicada.
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
