iNOS-derived nitric oxide modulates infection-stimulated bone loss (2008)
- Authors:
- USP affiliated authors: GARLET, GUSTAVO POMPERMAIER - FOB ; CAMPOS, MARIO JULIO AVILA - ICB ; SILVA, JOÃO SANTANA DA - FMRP ; CUNHA, FERNANDO DE QUEIROZ - FMRP
- Unidades: FOB; ICB; FMRP
- Subjects: ÓXIDO NÍTRICO; LESÕES PERIAPICAIS; RATOS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Journal of Dental Research
- ISSN: 0022-0345
- Volume/Número/Paginação/Ano: v. 87, n. 12, p. 1155-1159, Dec. 2008
-
ABNT
FUKADA, S. Y. et al. iNOS-derived nitric oxide modulates infection-stimulated bone loss. Journal of Dental Research, v. 87, n. 12, p. 1155-1159, 2008Tradução . . Disponível em: http://jdr.iadrjournals.org/cgi/reprint/87/12/1155?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&andorexactfulltext=and&searchid=1&FIRSTINDEX=0&sortspec=relevance&volume=87&firstpage=1155&resourcetype=HWCIT. Acesso em: 19 abr. 2024. -
APA
Fukada, S. Y., Silva, T. A., Saconato, I. F., Garlet, G. P., Avila Campos, M. J., Silva, J. S. da, & Cunha, F. de Q. (2008). iNOS-derived nitric oxide modulates infection-stimulated bone loss. Journal of Dental Research, 87( 12), 1155-1159. Recuperado de http://jdr.iadrjournals.org/cgi/reprint/87/12/1155?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&andorexactfulltext=and&searchid=1&FIRSTINDEX=0&sortspec=relevance&volume=87&firstpage=1155&resourcetype=HWCIT -
NLM
Fukada SY, Silva TA, Saconato IF, Garlet GP, Avila Campos MJ, Silva JS da, Cunha F de Q. iNOS-derived nitric oxide modulates infection-stimulated bone loss [Internet]. Journal of Dental Research. 2008 ; 87( 12): 1155-1159.[citado 2024 abr. 19 ] Available from: http://jdr.iadrjournals.org/cgi/reprint/87/12/1155?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&andorexactfulltext=and&searchid=1&FIRSTINDEX=0&sortspec=relevance&volume=87&firstpage=1155&resourcetype=HWCIT -
Vancouver
Fukada SY, Silva TA, Saconato IF, Garlet GP, Avila Campos MJ, Silva JS da, Cunha F de Q. iNOS-derived nitric oxide modulates infection-stimulated bone loss [Internet]. Journal of Dental Research. 2008 ; 87( 12): 1155-1159.[citado 2024 abr. 19 ] Available from: http://jdr.iadrjournals.org/cgi/reprint/87/12/1155?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&andorexactfulltext=and&searchid=1&FIRSTINDEX=0&sortspec=relevance&volume=87&firstpage=1155&resourcetype=HWCIT - The dual role of p55 tumour necrosis factor-a receptor in Actinobacillus actinomycetemcomitans-induced experimental periodontitis: host protection and tissue destruction
- Cytokine pattern determines the progression of experimental periodontal disease induced by Actinobacillus actinomycetemcomitans through the modulation of MMPs, RANKL, and their physiological inhibitors
- CCR2 deficiency results in increased osteolysis in experimental periapical lesions in mice
- Chemokines in oral inflammatory diseases: apical periodontitis and periodontal disease
- The cooperative role of CCR1 and CCR5 in the migration of osteoclast precursors and RANKL+ Cells throughout inflammatory bone loss after periodontal infection
- Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice
- TNF-alpha/CCL3-5/CCR5 cascade mediates RANKL plus cells migration in inflammatory bone resorption
- Evidences of the cooperative role of the chemokines CCL3, CCL4 and CCL5 and its receptors CCR1+ and CCR5+ in RANKL+ cell migration throughout experimental periodontitis in mice
- Association of human T lymphotropic virus 1 amplification of periodontitis severity with altered cytokine expression in response to a standard periodontopathogen infection
- The conflicting roles of TNF-'alfa'-mediated cell migration in experimental periodontal disease: from host protection to tissue destruction
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
