Skipping of exon 30 in C5 gene results in complete human C5 deficiency and demonstrates the importance of C5d and CUB domains for stability (2009)
- Authors:
- USP affiliated authors: FARAH, SHAKER CHUCK - IQ ; ISAAC, LOURDES - ICB
- Unidades: IQ; ICB
- DOI: 10.1016/j.molimm.2008.10.035
- Subjects: MENINGITE; IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular Immunology
- ISSN: 0161-5890
- Volume/Número/Paginação/Ano: v. 46, n. 10, p. 2116-2123, 2009
- Este periódico é de acesso aberto
- Este artigo NÃO é de acesso aberto
-
ABNT
AGUILAR-RAMIREZ, Priscilia et al. Skipping of exon 30 in C5 gene results in complete human C5 deficiency and demonstrates the importance of C5d and CUB domains for stability. Molecular Immunology, v. 46, n. 10, p. 2116-2123, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2008.10.035. Acesso em: 26 jan. 2026. -
APA
Aguilar-Ramirez, P., Reis, E. da S., Florido, M. P. C., Barbosa, A. S., Farah, C. S., Costa-Carvalho, B. T., & Isaac, L. (2009). Skipping of exon 30 in C5 gene results in complete human C5 deficiency and demonstrates the importance of C5d and CUB domains for stability. Molecular Immunology, 46( 10), 2116-2123. doi:10.1016/j.molimm.2008.10.035 -
NLM
Aguilar-Ramirez P, Reis E da S, Florido MPC, Barbosa AS, Farah CS, Costa-Carvalho BT, Isaac L. Skipping of exon 30 in C5 gene results in complete human C5 deficiency and demonstrates the importance of C5d and CUB domains for stability [Internet]. Molecular Immunology. 2009 ; 46( 10): 2116-2123.[citado 2026 jan. 26 ] Available from: https://doi.org/10.1016/j.molimm.2008.10.035 -
Vancouver
Aguilar-Ramirez P, Reis E da S, Florido MPC, Barbosa AS, Farah CS, Costa-Carvalho BT, Isaac L. Skipping of exon 30 in C5 gene results in complete human C5 deficiency and demonstrates the importance of C5d and CUB domains for stability [Internet]. Molecular Immunology. 2009 ; 46( 10): 2116-2123.[citado 2026 jan. 26 ] Available from: https://doi.org/10.1016/j.molimm.2008.10.035 - C1s deficiency associated with systemic lupus erythematosus highlights alternative splicing of anormal C1s gene
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Informações sobre o DOI: 10.1016/j.molimm.2008.10.035 (Fonte: oaDOI API)
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