Aminoguanidine and metformin prevent the reduced rate of HDL-mediated cell cholesterol efflux induced by formation of advanced glycation end products (2006)
- Authors:
- USP affiliated authors: QUINTAO, EDER CARLOS ROCHA - FM ; PASSARELLI, MARISA - FM
- Unidade: FM
- DOI: 10.1016/j.biocel.2005.09.016
- Subjects: DIABETES MELLITUS (METABOLISMO); DIABETES MELLITUS (EFEITOS DE DROGAS); GLICEMIA (METABOLISMO)
- Language: Inglês
- Imprenta:
- Publisher place: Kidlington
- Date published: 2006
- Source:
- Título: International Journal of Biochemistry
- ISSN: 1357-2725
- Volume/Número/Paginação/Ano: v. 38, n. 3, p. 392-403, 2006
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
MACHADO, Adriana P. et al. Aminoguanidine and metformin prevent the reduced rate of HDL-mediated cell cholesterol efflux induced by formation of advanced glycation end products. International Journal of Biochemistry, v. 38, n. 3, p. 392-403, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.biocel.2005.09.016. Acesso em: 19 out. 2024. -
APA
Machado, A. P., Pinto, R. S., Moysés, Z. P., Nakandakare, E. R., Quintão, E. C. R., & Passarelli, M. (2006). Aminoguanidine and metformin prevent the reduced rate of HDL-mediated cell cholesterol efflux induced by formation of advanced glycation end products. International Journal of Biochemistry, 38( 3), 392-403. doi:10.1016/j.biocel.2005.09.016 -
NLM
Machado AP, Pinto RS, Moysés ZP, Nakandakare ER, Quintão ECR, Passarelli M. Aminoguanidine and metformin prevent the reduced rate of HDL-mediated cell cholesterol efflux induced by formation of advanced glycation end products [Internet]. International Journal of Biochemistry. 2006 ; 38( 3): 392-403.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/j.biocel.2005.09.016 -
Vancouver
Machado AP, Pinto RS, Moysés ZP, Nakandakare ER, Quintão ECR, Passarelli M. Aminoguanidine and metformin prevent the reduced rate of HDL-mediated cell cholesterol efflux induced by formation of advanced glycation end products [Internet]. International Journal of Biochemistry. 2006 ; 38( 3): 392-403.[citado 2024 out. 19 ] Available from: https://doi.org/10.1016/j.biocel.2005.09.016 - Lipoprotein desialylation simultaneously enhances the cell cholesterol uptake and impairs the reverse cholesterol transport system: in vitro evidences utlizing neuraminidase-treated lipoproteins and mouse peritoneal macrophages
- Albumina glicada reduz o efluxo de colesterol celular por diminuir a ligação de HDL ao seu receptor e por induzir glicoxidação intracelular
- Intracellular oxidative stress induced by advanced glycated albumin reduces ABCA-1, ABCG-1 and increases rage and CD-36 expression
- Impairment of the HDL-mediated cell cholesterol efflux rate by advance glycated albumin is reversed by aminoguanidine
- Intracellular oxidative stress induced by advaced glycated albumin reduces ABCA-1 ABCG-1 and increase rage and CD-36 expression
- Influence of in vitro HDL glycodesialylation and glycoxidation on the cellular cholesterol efflux from macrophage foam cells
- In vitro lipid transfer protein mediated radioactive cholesteryl oleate transfer rate from high densit lipoprotein to a oxidized is slower than to normal low density lipoprotein
- Metformin, probucol and aminoguanidine prevent the reduced rate of HDL-mediated cell cholesterol efflux induced by intracellular advanced glycation
- Estresse oxidativo, induzido pela albumina glicada, reduz a expressão do receptor ABCA-1 e a remoção de colesterol celular, mediada por HDL
- Intracellular oxidative stress induced by advanced glycated albumin reduces ABC-1 and ABCG-1 and increases rage and CD-36 expression
Informações sobre o DOI: 10.1016/j.biocel.2005.09.016 (Fonte: oaDOI API)
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