Lipoprotein desialylation simultaneously enhances the cell cholesterol uptake and impairs the reverse cholesterol transport system: in vitro evidences utlizing neuraminidase-treated lipoproteins and mouse peritoneal macrophages (1998)
- Authors:
- USP affiliated authors: PASSARELLI, MARISA - FM ; QUINTAO, EDER CARLOS ROCHA - FM
- Unidade: FM
- DOI: 10.1016/s0021-9150(98)00057-4
- Assunto: ENDOCRINOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Atherosclerosis
- Volume/Número/Paginação/Ano: v. 139, n. 1, p. 65-75, 1998
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
HARADA, Lilia M et al. Lipoprotein desialylation simultaneously enhances the cell cholesterol uptake and impairs the reverse cholesterol transport system: in vitro evidences utlizing neuraminidase-treated lipoproteins and mouse peritoneal macrophages. Atherosclerosis, v. 139, n. 1, p. 65-75, 1998Tradução . . Disponível em: https://doi.org/10.1016/s0021-9150(98)00057-4. Acesso em: 28 dez. 2025. -
APA
Harada, L. M., Carvalho, M. D. T., Passarelli, M., & Quintão, E. C. da R. (1998). Lipoprotein desialylation simultaneously enhances the cell cholesterol uptake and impairs the reverse cholesterol transport system: in vitro evidences utlizing neuraminidase-treated lipoproteins and mouse peritoneal macrophages. Atherosclerosis, 139( 1), 65-75. doi:10.1016/s0021-9150(98)00057-4 -
NLM
Harada LM, Carvalho MDT, Passarelli M, Quintão EC da R. Lipoprotein desialylation simultaneously enhances the cell cholesterol uptake and impairs the reverse cholesterol transport system: in vitro evidences utlizing neuraminidase-treated lipoproteins and mouse peritoneal macrophages [Internet]. Atherosclerosis. 1998 ; 139( 1): 65-75.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/s0021-9150(98)00057-4 -
Vancouver
Harada LM, Carvalho MDT, Passarelli M, Quintão EC da R. Lipoprotein desialylation simultaneously enhances the cell cholesterol uptake and impairs the reverse cholesterol transport system: in vitro evidences utlizing neuraminidase-treated lipoproteins and mouse peritoneal macrophages [Internet]. Atherosclerosis. 1998 ; 139( 1): 65-75.[citado 2025 dez. 28 ] Available from: https://doi.org/10.1016/s0021-9150(98)00057-4 - Intracellular oxidative stress induced by advaced glycated albumin reduces ABCA-1 ABCG-1 and increase rage and CD-36 expression
- Influence of in vitro HDL glycodesialylation and glycoxidation on the cellular cholesterol efflux from macrophage foam cells
- In vitro lipid transfer protein mediated radioactive cholesteryl oleate transfer rate from high densit lipoprotein to a oxidized is slower than to normal low density lipoprotein
- Metformin, probucol and aminoguanidine prevent the reduced rate of HDL-mediated cell cholesterol efflux induced by intracellular advanced glycation
- Estresse oxidativo, induzido pela albumina glicada, reduz a expressão do receptor ABCA-1 e a remoção de colesterol celular, mediada por HDL
- Intracellular oxidative stress induced by advanced glycated albumin reduces ABC-1 and ABCG-1 and increases rage and CD-36 expression
- Dislipidemias
- Albumina glicada reduz o efluxo de colesterol celular por diminuir a ligação de HDL ao seu receptor e por induzir glicoxidação intracelular
- Intracellular oxidative stress induced by advanced glycated albumin reduces ABCA-1, ABCG-1 and increases rage and CD-36 expression
- Impairment of the HDL-mediated cell cholesterol efflux rate by advance glycated albumin is reversed by aminoguanidine
Informações sobre o DOI: 10.1016/s0021-9150(98)00057-4 (Fonte: oaDOI API)
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