Aminoacetone impairs prodution of insulin by RINm5f cells (2008)
- Authors:
- USP affiliated authors: MALPARTIDA, HUMBERTO MIGUEL GARAY - EACH ; SOGAYAR, MARI CLEIDE - IQ ; BECHARA, ETELVINO JOSE HENRIQUES - IQ
- Unidades: EACH; IQ
- Subjects: BIOQUÍMICA; DIABETES MELLITUS
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Abstracts
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq)
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ABNT
SARTORI, Adriano et al. Aminoacetone impairs prodution of insulin by RINm5f cells. 2008, Anais.. São Paulo: SBBq, 2008. . Acesso em: 20 abr. 2024. -
APA
Sartori, A., Garay-Malpartida, H. M., Forni, M. F., Dutra, F., Sogayar, M. C., & Bechara, E. J. H. (2008). Aminoacetone impairs prodution of insulin by RINm5f cells. In Abstracts. São Paulo: SBBq. -
NLM
Sartori A, Garay-Malpartida HM, Forni MF, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone impairs prodution of insulin by RINm5f cells. Abstracts. 2008 ;[citado 2024 abr. 20 ] -
Vancouver
Sartori A, Garay-Malpartida HM, Forni MF, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone impairs prodution of insulin by RINm5f cells. Abstracts. 2008 ;[citado 2024 abr. 20 ] - Recombinant human prolactin (rhPRL) promotes human beta cell survival by inhibiting apoptosis
- Recombinant human prolactin (rhPRL) promotes human beta-cell survival through apoptosis inhibition
- Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells
- The novel cytoline PANDER/FAM3B inhibits cell death in prostate tumor cells
- Glypican-3 reexpression regulates apoptosis in murine adenocarcinoma mammary cells modulating PI3K/Akt and p38MAPK signaling pathways
- Recombinant human prolactin (rhPRL) promotes human beta-cell surviral by inhibiting apoptosis
- The novel cytokine PANDER/FAM3B inhibits cell death in prostate tumor cells
- Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells
- Aminoacetone induces copper-mediated oxidative damage to insulin-producing beta cells
- Effects of aminoacetone, a threonine metabolite, on insulin-producing cells
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