Aminoacetone induces copper-mediated oxidative damage to insulin-producing beta cells (2007)
- Authors:
- USP affiliated authors: SOGAYAR, MARI CLEIDE - IQ ; BECHARA, ETELVINO JOSE HENRIQUES - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; DIABETES MELLITUS
- Language: Inglês
- Imprenta:
- Source:
- Título: Abstracts
- Conference titles: Reunião Anual da Sociedade Brasileira de Bioquímica e Biologia Molecular
-
ABNT
SARTORI, Adriano et al. Aminoacetone induces copper-mediated oxidative damage to insulin-producing beta cells. 2007, Anais.. São Paulo: SBBq, 2007. . Acesso em: 13 fev. 2026. -
APA
Sartori, A., Dutra, F., Sogayar, M. C., & Bechara, E. J. H. (2007). Aminoacetone induces copper-mediated oxidative damage to insulin-producing beta cells. In Abstracts. São Paulo: SBBq. -
NLM
Sartori A, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone induces copper-mediated oxidative damage to insulin-producing beta cells. Abstracts. 2007 ;[citado 2026 fev. 13 ] -
Vancouver
Sartori A, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone induces copper-mediated oxidative damage to insulin-producing beta cells. Abstracts. 2007 ;[citado 2026 fev. 13 ] - Glutathione as a key regulator of skin stem cell homeostasis: From embryonic development to ageing
- Aminoacetone causes oxidative stress and dealth to insulin-producing RINM5F cells
- Effects of aminoacetone, a threonine metabolite, on insulin-producing cells
- Aminoacetone impairs prodution of insulin by RINm5f cells
- Aminoacetone increases NO levels in pancreatic beta-cell lineages and cytochrome c oxidative damage
- Oxidative damage caused by aminoacetone on insulin-producing cells
- Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells
- Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells
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