Impaired dendritic cell differentiation and maturation in the absence of C3 (2007)
- Authors:
- USP affiliated authors: BARBUTO, JOSE ALEXANDRE MARZAGAO - ICB ; ISAAC, LOURDES - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular Immunology
- Volume/Número/Paginação/Ano: v. 44, n. 16, 2007
- Conference titles: European Meeting on Complement in Human Disease
-
ABNT
REIS, Edimara S. et al. Impaired dendritic cell differentiation and maturation in the absence of C3. Molecular Immunology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 21 fev. 2026. , 2007 -
APA
Reis, E. S., Barbuto, J. A. M., Köhl, J., & Isaac, L. (2007). Impaired dendritic cell differentiation and maturation in the absence of C3. Molecular Immunology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Reis ES, Barbuto JAM, Köhl J, Isaac L. Impaired dendritic cell differentiation and maturation in the absence of C3. Molecular Immunology. 2007 ; 44( 16):[citado 2026 fev. 21 ] -
Vancouver
Reis ES, Barbuto JAM, Köhl J, Isaac L. Impaired dendritic cell differentiation and maturation in the absence of C3. Molecular Immunology. 2007 ; 44( 16):[citado 2026 fev. 21 ] - Human monocyte-derived dendritic cells produce complement proteins
- Human monocyte-derived dendritic cells are a source of several complement proteins
- Complement components, regulators and receptors are produced by human monocyte-derived dendritic cells
- Complement components, regulators and receptors are produced by human monocyte-derived dendritic cells
- Complement is necessary for full human monocyte differentiation into dendritic cells
- Immunological and genetic characterization of deficiency of the component C5 of the human complement system
- Clinical manifestations of complement deficient patients
- Microarray based analysis of serum proteins in dried blood spot samples on filter paper
- Skipping of exon 27 in C3 gene compromises TED domain and results in complete human C3 deficiency
- Leptospirosis: aspects of innate immunity, immunopathogenesis and immune evasion from the complement system
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