The B2 subunit of vascular H+-ATPase is chronically regulated by angiotensin II in immortalized rat proximal tubule cells (2007)
- Authors:
- USP affiliated authors: SOUZA, MARIA OLIVEIRA DE - ICB ; MALNIC, GERHARD - ICB
- School: ICB
- Subject: FISIOLOGIA
- Language: Inglês
- Imprenta:
- Publisher: Sociedade Brasileira de Nefrologia
- Place of publication: Rio de Janeiro
- Date published: 2007
- Source:
- Título do periódico: Abstracts
- Conference title: World Congress of Nephrology
-
ABNT
CARRARO-LACROIX, L. R.; BEZERRA, C. N. A.; OLIVEIRA-SOUZA, Maria; MALNIC, Gerhard. The B2 subunit of vascular H+-ATPase is chronically regulated by angiotensin II in immortalized rat proximal tubule cells. Anais.. Rio de Janeiro: Sociedade Brasileira de Nefrologia, 2007. -
APA
Carraro-Lacroix, L. R., Bezerra, C. N. A., Oliveira-Souza, M., & Malnic, G. (2007). The B2 subunit of vascular H+-ATPase is chronically regulated by angiotensin II in immortalized rat proximal tubule cells. In Abstracts. Rio de Janeiro: Sociedade Brasileira de Nefrologia. -
NLM
Carraro-Lacroix LR, Bezerra CNA, Oliveira-Souza M, Malnic G. The B2 subunit of vascular H+-ATPase is chronically regulated by angiotensin II in immortalized rat proximal tubule cells. Abstracts. 2007 ; -
Vancouver
Carraro-Lacroix LR, Bezerra CNA, Oliveira-Souza M, Malnic G. The B2 subunit of vascular H+-ATPase is chronically regulated by angiotensin II in immortalized rat proximal tubule cells. Abstracts. 2007 ; - Colchicine impairs the dose-dependent stimulatory effect of ANG II or AVP on acid subcellular vesicle traafficking in MDCK cells
- ANP impairs the dose-dependent stimulatory effect of ANG II or AVP on H+-ATPase subcellular vesicle trafficking
- Arginine vasopressin stimulates H+ - ATPase in MDCK cells via V1 (cell Ca2+) and V2 (cAMP) receptors
- Genomic and nongenomic dose-dependent biphasic effect of aldosterone on Na+/H+ exchanger in proximal S3 segment: role of cytosolic calcium
- Colchicina inibe ação da AVP no tráfego de vesículas ácidas subcelulares, em células MDCK
- Activation of the AT1 receptor-beta-arrestin signaling pathway inhibits NHE3 activity in the renal proximal tubule
- Proximal tubule NHE3 activity is inhibited by beta-arrestin-biased angiotensin II type 1 receptor signaling
- The role of β-adrenergic overstimulation in the early stages of renal injury
- Efeito prolongado da angiotensina II (ANG II) sobre as cascatas de sinalização e internalização do receptor AT1-
- Efeito da angiotensina II e do peptídeo atrial natriurético na modulação do PH intracelular de células mdck, em cultura
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