Survivin and CIAP-1 gene expression is linked to chronic myeloid leukemia progression and poor response to imatinib (2006)
- Authors:
- USP affiliated authors: LOPES, LUCIA ROSSETTI - ICB ; CASTRO, FABÍOLA ATTIÉ DE - FCFRP ; MOREIRA FILHO, CARLOS ALBERTO - ICB ; MENDES, JOAO GUSTAVO PESSINI AMARANTE - ICB
- Unidades: ICB; FCFRP
- Subjects: LEUCEMIA; EXPRESSÃO GÊNICA
- Language: Inglês
- Imprenta:
- Source:
- Título: Haematologica
- ISSN: 0390-6078
- Volume/Número/Paginação/Ano: v. 91, suppl. 1, p. 65, res. 0176, 2006
- Conference titles: Congress of The European Hematology Association
-
ABNT
CASTRO, Fabíola Attié de et al. Survivin and CIAP-1 gene expression is linked to chronic myeloid leukemia progression and poor response to imatinib. Haematologica. Pavia: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 09 out. 2024. , 2006 -
APA
Castro, F. A. de, Jacysyn, J. F., Ulbrich, A. G., Tobo, P. R., Lopes, L. R., Alves, D. I. C., et al. (2006). Survivin and CIAP-1 gene expression is linked to chronic myeloid leukemia progression and poor response to imatinib. Haematologica. Pavia: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Castro FA de, Jacysyn JF, Ulbrich AG, Tobo PR, Lopes LR, Alves DIC, Zanichelli MA, Colassanti MD, Camanho D, Moreira-Filho CA, Hamerschlak N, Amarante-Mendes JGP. Survivin and CIAP-1 gene expression is linked to chronic myeloid leukemia progression and poor response to imatinib. Haematologica. 2006 ; 91 65.[citado 2024 out. 09 ] -
Vancouver
Castro FA de, Jacysyn JF, Ulbrich AG, Tobo PR, Lopes LR, Alves DIC, Zanichelli MA, Colassanti MD, Camanho D, Moreira-Filho CA, Hamerschlak N, Amarante-Mendes JGP. Survivin and CIAP-1 gene expression is linked to chronic myeloid leukemia progression and poor response to imatinib. Haematologica. 2006 ; 91 65.[citado 2024 out. 09 ] - Genomic approach to understand the mechanisms invilved in Bcr-Abl-mediated resistance to apoptosis
- Indoleamine 2,3-dioxygenase expression in chronic myelogenous leukemia patients: association with disease progression
- Expression of pro-apoptotic bid and bik genes is decreased in chronic myeloid leukemia and low levels of bik MRNA are associated with lack of response to imatinib treatment
- Genomic approach to understand the mechanisms involved in BCR-ABL mediated resistance to apoptosis
- Down regulation of Hematopoietic SH2 domain containing (HSH2D) in the progression of the chronic myelogenous leukemia
- Development and characterization of murine models of T lymphoma to investigate the impact of oncogene expression
- Conversion of CD95 (Fas) type II into type I signaling by sub-lethal doses of cycloheximide
- Correlation between metabolic alterations and apoptosis resistance confered by BCR-ABL expression
- T lymphoma murine models to study the impact of ectopic expression of anti-apoptotic oncogenes
- Cosmomycin D, an anthracycline more potent than Doxorubicin for induction of tumor cell death, synergize with imatinib mesilate to induce apoptosis in Bcr-Abl-positive cells
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