Envolvement of galectin-3 in replicative senescene of mesenchymal embryone cells exposed to glucose-dependent metabolic oxidative stress (2004)
- Authors:
- Autor USP: CHAMMAS, ROGER - FM
- Unidade: FM
- Subjects: CÉLULAS; ESTRESSE; MEDICINA
- Language: Inglês
- Imprenta:
- Publisher place: Angra dos Reis, Rio de Janeiro, Brazil
- Date published: 2004
- Conference titles: Brazilian Symposium on Extracellular Matrix
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ABNT
JUNQUEIRA, M. S. e LIU, F. T. e CHAMMA, Roger. Envolvement of galectin-3 in replicative senescene of mesenchymal embryone cells exposed to glucose-dependent metabolic oxidative stress. 2004, Anais.. Angra dos Reis, Rio de Janeiro, Brazil: Faculdade de Medicina, Universidade de São Paulo, 2004. . Acesso em: 20 jan. 2026. -
APA
Junqueira, M. S., Liu, F. T., & Chamma, R. (2004). Envolvement of galectin-3 in replicative senescene of mesenchymal embryone cells exposed to glucose-dependent metabolic oxidative stress. In . Angra dos Reis, Rio de Janeiro, Brazil: Faculdade de Medicina, Universidade de São Paulo. -
NLM
Junqueira MS, Liu FT, Chamma R. Envolvement of galectin-3 in replicative senescene of mesenchymal embryone cells exposed to glucose-dependent metabolic oxidative stress. 2004 ;[citado 2026 jan. 20 ] -
Vancouver
Junqueira MS, Liu FT, Chamma R. Envolvement of galectin-3 in replicative senescene of mesenchymal embryone cells exposed to glucose-dependent metabolic oxidative stress. 2004 ;[citado 2026 jan. 20 ] - Evidence that DNA methylation contributes to regulation of galectin-3 expression in human cancers
- Evidence for a dual role of galectin-3 within tumor
- Recrutamento de células inflamatórias por galectina-3 facilitam o crescimento de melanoma murino in vivo
- Resistência às terapias
- Ganglioside GD3 modulates chemotherapy-induced cell death in vitro
- Dual role of galectin-3 in murine melanoma growth
- Ganglioside Gd3 modulates chemotherapy-induced cell death in vitro
- Galectin-3 gene expression is silenced by methylation of its promoter in murine melanoma cells.
- Cellular prion protein protects against experimetnal metastasis in mouse model
- Epigenetic mechanisms control galectin-3 gene expression along murine melanoma progression
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