Humanization of the anti-CD18 antibody 6.7:: an unexpected effect of a framework residue in binding to antigen (2003)
- Authors:
- USP affiliated authors: KALIL FILHO, JORGE ELIAS - FM ; COELHO, VERONICA PORTO CARREIRO DE VASCONCELLOS - FM
- Unidade: FM
- DOI: 10.1016/s0161-5890(03)00022-1
- Assunto: ANTÍGENOS
- Language: Inglês
- Imprenta:
- Source:
- Título: Molecular Immunology.
- ISSN: 0161-5890
- Volume/Número/Paginação/Ano: v. 1240, p. 1-12, 2003
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
CALDAS, Cristina et al. Humanization of the anti-CD18 antibody 6.7:: an unexpected effect of a framework residue in binding to antigen. Molecular Immunology., v. 1240, p. 1-12, 2003Tradução . . Disponível em: https://doi.org/10.1016/s0161-5890(03)00022-1. Acesso em: 03 nov. 2024. -
APA
Caldas, C., Coelho, V., Kalil, J., Moro, A. M., Maranhão, A. Q., & Brígido, M. M. (2003). Humanization of the anti-CD18 antibody 6.7:: an unexpected effect of a framework residue in binding to antigen. Molecular Immunology., 1240, 1-12. doi:10.1016/s0161-5890(03)00022-1 -
NLM
Caldas C, Coelho V, Kalil J, Moro AM, Maranhão AQ, Brígido MM. Humanization of the anti-CD18 antibody 6.7:: an unexpected effect of a framework residue in binding to antigen [Internet]. Molecular Immunology. 2003 ; 1240 1-12.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/s0161-5890(03)00022-1 -
Vancouver
Caldas C, Coelho V, Kalil J, Moro AM, Maranhão AQ, Brígido MM. Humanization of the anti-CD18 antibody 6.7:: an unexpected effect of a framework residue in binding to antigen [Internet]. Molecular Immunology. 2003 ; 1240 1-12.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/s0161-5890(03)00022-1 - Rejection of grafts with no H-2 disparity in TAP1 mutant mice:: CD4 T cells are important effector cells and self H-2b class I molecules are target
- T cell reactivity to HSP60 in murine model of vascularized heart allografting
- Locally produced IL-15 may underlie the predominance of CD8+IL2RALPHA- T cells ar heart lesions of human Chagas Disease cardiomyopathy
- Transplante cardíaco para tratamento da miocardiopatia chagásica
- Distribution of primary immunodeficiency diseases at University of Sao Paulo Medical School, Brazil
- Comparison between indirect allo-proliferative response and binding prediction to HLA-DR molecules by tepitope analysis
- Sensitization to aeroallergens: urban/rural differences in São Paulo-Brazil
- Rheumatic heart disease: intralesional T cells recognize peptides from cardiac myosin beta chain
- Can heat shock protein be used to induce transplantation tolerance to murine allograft?
- Identification of public expansions in the peripheral T cell repertoire reactive to "Self" H-2'K POT. 1'peptides, following rejection of grafts without H-2 disparaty, in tapi mutant mice
Informações sobre o DOI: 10.1016/s0161-5890(03)00022-1 (Fonte: oaDOI API)
How to cite
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas