Increased microvascular permeability in the hamster cheek pouch induced by oxidized low density lipoprotein (oxLDL) and some fragmented apolipoprotein B proteins (2003)
- Authors:
- USP affiliated authors: NEGRO, SONIA JANCAR - ICB ; GIDLUND, MAGNUS AKE - ICB
- Unidade: ICB
- DOI: 10.1007/s000110300074
- Assunto: IMUNOLOGIA
- Language: Inglês
- Source:
- Título do periódico: Inflammation Research
- ISSN: 1023-3830
- Volume/Número/Paginação/Ano: v.52, p. 215-220, 2003
- Este periódico é de assinatura
- Este artigo NÃO é de acesso aberto
- Cor do Acesso Aberto: closed
-
ABNT
SVENSJÖ, E. et al. Increased microvascular permeability in the hamster cheek pouch induced by oxidized low density lipoprotein (oxLDL) and some fragmented apolipoprotein B proteins. Inflammation Research, v. 52, p. 215-220, 2003Tradução . . Disponível em: https://doi.org/10.1007/s000110300074. Acesso em: 25 abr. 2024. -
APA
Svensjö, E., Boschcov, P., Ketelhuth, D. F. J., Passaglia, R. de cassia A. T., & Carvalho, M. H. C. (2003). Increased microvascular permeability in the hamster cheek pouch induced by oxidized low density lipoprotein (oxLDL) and some fragmented apolipoprotein B proteins. Inflammation Research, 52, 215-220. doi:10.1007/s000110300074 -
NLM
Svensjö E, Boschcov P, Ketelhuth DFJ, Passaglia R de cassia AT, Carvalho MHC. Increased microvascular permeability in the hamster cheek pouch induced by oxidized low density lipoprotein (oxLDL) and some fragmented apolipoprotein B proteins [Internet]. Inflammation Research. 2003 ;52 215-220.[citado 2024 abr. 25 ] Available from: https://doi.org/10.1007/s000110300074 -
Vancouver
Svensjö E, Boschcov P, Ketelhuth DFJ, Passaglia R de cassia AT, Carvalho MHC. Increased microvascular permeability in the hamster cheek pouch induced by oxidized low density lipoprotein (oxLDL) and some fragmented apolipoprotein B proteins [Internet]. Inflammation Research. 2003 ;52 215-220.[citado 2024 abr. 25 ] Available from: https://doi.org/10.1007/s000110300074 - Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxide production in RAW264.7 cells - effects of a PAF-antagonist
- Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides
- Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL)
- Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR
- OxLDL inibe a produção de óxido nítrico induzida por LPS e IFN-gamma em células RAW264.7 via receptor de PAF
- Oxidized low density lipoprotein (LDL) can increase the expression of scavenger receptor (CD36) and FCgRII by interaction with platelet-activating factor (PAF) receptor
- Oxidized low density lipoprotein (oxLDL) and purified oxLDL products inhibits nitric oxide releasem iNOS and COX-2 expression in macrophages
- Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxideproduction in RAW264.7 cells - effect of a PAF-antagonist
- Degree of modification of low density lipoprotein (LDL) displays different properties in size and eletric charge and induction of CD11B and CD36 in THP-1 cells
- Vascular permeability is increased by oxidized low density lopoprotein (oxLDL) and selected apolipoprotein B derived synthetic polypeptides
Informações sobre o DOI: 10.1007/s000110300074 (Fonte: oaDOI API)
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