Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides (2002)
- Authors:
- USP affiliated authors: GIDLUND, MAGNUS AKE - ICB ; NEGRO, SONIA JANCAR - ICB
- Unidade: ICB
- Assunto: IMMUNOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título: Anais
- Conference titles: Meeting of the Brazilian Society of Immunology
-
ABNT
SVENSJO, E. et al. Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides. 2002, Anais.. Salvador: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2002. . Acesso em: 30 dez. 2025. -
APA
Svensjo, E., Ketelhuth, D. F. J., Boschcov, P., Jancar, S., & Gidlund, M. A. (2002). Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides. In Anais. Salvador: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Svensjo E, Ketelhuth DFJ, Boschcov P, Jancar S, Gidlund MA. Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides. Anais. 2002 ;[citado 2025 dez. 30 ] -
Vancouver
Svensjo E, Ketelhuth DFJ, Boschcov P, Jancar S, Gidlund MA. Vascular permeability is increased by oxidized low density lipoprotein (oxLDL), size-inclusion subfractions and slelcted immunogenic apolipoprotein B (apoB) derived synthetic peptides. Anais. 2002 ;[citado 2025 dez. 30 ] - Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxide production in RAW264.7 cells - effects of a PAF-antagonist
- OxLDL inibe a produção de óxido nítrico induzida por LPS e IFN-gamma em células RAW264.7 via receptor de PAF
- Oxidized low density lipoprotein (LDL) can increase the expression of scavenger receptor (CD36) and FCgRII by interaction with platelet-activating factor (PAF) receptor
- Myeloperoxidase, hydrogen peroxide and tumor necrosis factor 'alfa' production as methods to access platelet aggregating factor - like activity of oxidazed low density lipoprotein (OXLDL)
- Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR
- Increased microvascular permeability in the hamster cheek pouch induced by oxidized low density lipoprotein (oxLDL) and some fragmented apolipoprotein B proteins
- Vascular permeability is increased by oxidized low density lipoprotein (oxLDL) and selected apolipoprotein B derived synthetic polypeptides
- Oxidized low density lipoprotein inhibits LPS/IFN-induced nitric oxideproduction in RAW264.7 cells - effect of a PAF-antagonist
- Role of PPAR-gamma in the modulation of CD36 and FcgammaRII induced by LDL with low and high degrees of oxidation during the differentiation of the monocytic THP-1 cell line
- Pivotal role for platelet-activating factor receptor in CD36 expression and oxLDL uptake by human monocytes/macrophages
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