Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi (2000)
- Authors:
- USP affiliated authors: RIZZO, LUIZ VICENTE - ICB ; ABRAHAMSOHN, ISES DE ALMEIDA - ICB
- Unidade: ICB
- Assunto: IMMUNOLOGIA
- Language: Inglês
- Imprenta:
- Publisher place: Florianópolis
- Date published: 2000
- Source:
- Título: Abstract
- Conference titles: Meeting of the Brazilian Society of Immunology
-
ABNT
TADOKORO, C E et al. Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. 2000, Anais.. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2000. . Acesso em: 25 fev. 2026. -
APA
Tadokoro, C. E., Mosca, T., Rizzo, L. V., & Abrahamsohn, I. de A. (2000). Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. In Abstract. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Tadokoro CE, Mosca T, Rizzo LV, Abrahamsohn I de A. Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. Abstract. 2000 ;[citado 2026 fev. 25 ] -
Vancouver
Tadokoro CE, Mosca T, Rizzo LV, Abrahamsohn I de A. Lack of experimental autoimmune encephalomyelitis (EAE) in mice infected with Trypanosoma cruzi. Abstract. 2000 ;[citado 2026 fev. 25 ] - The development of experimental autoimmune encephalomyelitis could be completed abrogated by immunossuppressive effect of Trypanosoma cruzi infection
- Trypanosoma cruzi infection inhibits autoimmune encephalomyelitis
- Experimental autoimmune encephalomyelitis can be prevented and cured by infection with Trypanosoma cruzi
- The effect of t. cruzi infection over eae development
- High molecular weight components from Ascaris suum exert suppressive effect on dendritic cells
- Combined immunodeficiency in a chilhood with Cd3 deficiency: case report
- Regulation by cytokines and by nitric oxide of the lymphoproliferative responses in trypanosoma cruzi infection
- Cytokine and nitric oxide dependent suppression of lymphoproliferative responses in acute trypanosoma cruzi infections
- Effect of interleukin-4 deprivation and treatment on resistance to "trypanosoma cruzi"
- Interleukin-12 stimulation of lymphoproliferative response in Trypanossoma cruzi infection
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