Functional restoration of cultured mouse pancreatic islets after in vitro-exposure to alloxan (1988)
- Authors:
- Autor USP: EIZIRIK, DECIO LAKS - FMRP
- Unidade: FMRP
- Assunto: FARMACOLOGIA
- Language: Português
- Source:
- Título do periódico: Pharmacology e Toxicology
- Volume/Número/Paginação/Ano: v.63, n.5 , p.396-9, 1988
-
ABNT
EIZIRICK, D L e SANDLER, S. Functional restoration of cultured mouse pancreatic islets after in vitro-exposure to alloxan. Pharmacology e Toxicology, v. 63, n. 5 , p. 396-9, 1988Tradução . . Acesso em: 13 maio 2024. -
APA
Eizirick, D. L., & Sandler, S. (1988). Functional restoration of cultured mouse pancreatic islets after in vitro-exposure to alloxan. Pharmacology e Toxicology, 63( 5 ), 396-9. -
NLM
Eizirick DL, Sandler S. Functional restoration of cultured mouse pancreatic islets after in vitro-exposure to alloxan. Pharmacology e Toxicology. 1988 ;63( 5 ): 396-9.[citado 2024 maio 13 ] -
Vancouver
Eizirick DL, Sandler S. Functional restoration of cultured mouse pancreatic islets after in vitro-exposure to alloxan. Pharmacology e Toxicology. 1988 ;63( 5 ): 396-9.[citado 2024 maio 13 ] - Functional characteristics of cultured mouse pancreatic islets following exposure to different streptozotocin concentrations
- Diabetes mellitus tipo i (insulino-dependente): a busca de um tratamento etiologico
- Partial protectivie effect of branched chain amino acids against streptozotocin-induced xytotocity to mouse pancreatic islets in vitro
- Functional restoration of cultured mouse pancreatic islets six days after exposure to alloxan on heat shock
- Defective catabolism of d - glucose and alfaglutamine in islet, maintained in culture after streptozotocin exposure
- Functional characteristics of rat pancreatic islets maintained in culture following exposure to human intelenkin 1
- Culture of mouse pancreatic islets in different glucose concentrations modify beta-cells sensitivity to streptozotocin
- High protein diet does not prevent the development of low-dose streptozolin induced diabetes in mice
- Cold-acclimation improves cold-tolerance of diabetic rats
- Interleukin - 1 induced impairment in pancreatic islet oxidative metabolism of glucose is potentiated by tumor necrosis factor
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