Defective catabolism of d - glucose and alfaglutamine in islet, maintained in culture after streptozotocin exposure (1988)
- Authors:
- Autor USP: EIZIRIK, DECIO LAKS - FMRP
- Unidade: FMRP
- Assunto: MEDICINA
- Language: Português
- Source:
- Título do periódico: Diabetologia
- Volume/Número/Paginação/Ano: v.31, p.488a, 1988
-
ABNT
EIZIRIK, D L et al. Defective catabolism of d - glucose and alfaglutamine in islet, maintained in culture after streptozotocin exposure. Diabetologia, v. 31, p. 488a, 1988Tradução . . Acesso em: 19 abr. 2024. -
APA
Eizirik, D. L., Sandler, S., Sener, A., & Malaisse, W. J. (1988). Defective catabolism of d - glucose and alfaglutamine in islet, maintained in culture after streptozotocin exposure. Diabetologia, 31, 488a. -
NLM
Eizirik DL, Sandler S, Sener A, Malaisse WJ. Defective catabolism of d - glucose and alfaglutamine in islet, maintained in culture after streptozotocin exposure. Diabetologia. 1988 ;31 488a.[citado 2024 abr. 19 ] -
Vancouver
Eizirik DL, Sandler S, Sener A, Malaisse WJ. Defective catabolism of d - glucose and alfaglutamine in islet, maintained in culture after streptozotocin exposure. Diabetologia. 1988 ;31 488a.[citado 2024 abr. 19 ] - Functional restoration of cultured mouse pancreatic islets after in vitro-exposure to alloxan
- Functional characteristics of cultured mouse pancreatic islets following exposure to different streptozotocin concentrations
- Functional restoration of cultured mouse pancreatic islets six days after exposure to alloxan on heat shock
- Functional characteristics of rat pancreatic islets maintained in culture following exposure to human intelenkin 1
- Culture of mouse pancreatic islets in different glucose concentrations modify beta-cells sensitivity to streptozotocin
- High protein diet does not prevent the development of low-dose streptozolin induced diabetes in mice
- Cold-acclimation improves cold-tolerance of diabetic rats
- Interleukin - 1 induced impairment in pancreatic islet oxidative metabolism of glucose is potentiated by tumor necrosis factor
- Diabetes mellitus tipo i (insulino-dependente): a busca de um tratamento etiologico
- Partial protectivie effect of branched chain amino acids against streptozotocin-induced xytotocity to mouse pancreatic islets in vitro
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