Mild mitochondrial uncoupling as a therapeutic strategy (2011)
- Authors:
- USP affiliated authors: CUNHA, FERNANDA MARQUES DA - EACH ; KOWALTOWSKI, ALICIA JULIANA - IQ
- Unidades: EACH; IQ
- Subjects: MITOCÔNDRIAS; ESTRESSE OXIDATIVO
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Current Drug Targets
- ISSN: 1389-4501
- Volume/Número/Paginação/Ano: v. 12, n. 6, p. 783-789, 2011
-
ABNT
CUNHA, Fernanda Marques da et al. Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets, v. 12, n. 6, p. 783-789, 2011Tradução . . Acesso em: 19 set. 2024. -
APA
Cunha, F. M. da, Caldeira da Silva, C. C., Cerqueira, F. M., & Kowaltowski, A. J. (2011). Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets, 12( 6), 783-789. -
NLM
Cunha FM da, Caldeira da Silva CC, Cerqueira FM, Kowaltowski AJ. Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets. 2011 ; 12( 6): 783-789.[citado 2024 set. 19 ] -
Vancouver
Cunha FM da, Caldeira da Silva CC, Cerqueira FM, Kowaltowski AJ. Mild mitochondrial uncoupling as a therapeutic strategy. Current Drug Targets. 2011 ; 12( 6): 783-789.[citado 2024 set. 19 ] - Aging and calorie restriction modulate yeast redox state, oxidized protein removal, and the ubiquitin-proteasome system
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- Redox state, insulin sensitivity and aging
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- Caloric restriction protects mice against isoproterenol-induced cardiac hypertrophy by mechanisms that involve mitochondrial ATP sensitive potassium channel and modulation of redox state
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