Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth (2008)
- Autores:
- Autor USP: CALICH, VERA LUCIA GARCIA - ICB
- Unidade: ICB
- Assunto: IMUNOLOGIA
- Idioma: Inglês
- Imprenta:
- Editora: Comissão de Cultura e Extensão Universitária do ICB/USP
- Local: São Paulo
- Data de publicação: 2008
- Fonte:
- Título do periódico: Resumos
- Nome do evento: Congresso do Instituto de Ciências Biomédicas da Universidade de São Paulo
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ABNT
ARAUJO, E. F.; CALICH, Vera Lucia Garcia. Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. Anais.. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP, 2008. -
APA
Araujo, E. F., & Calich, V. L. G. (2008). Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. In Resumos. São Paulo: Comissão de Cultura e Extensão Universitária do ICB/USP. -
NLM
Araujo EF, Calich VLG. Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. Resumos. 2008 ; -
Vancouver
Araujo EF, Calich VLG. Suceptible and resistant mice to p. brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth. Resumos. 2008 ; - Type I and type 2 cytokines: from basic science to fungal infections
- Exarcebation of murine paracoccidioidomycosis induces type 2 cytokines, whereas the protective model of the disease is associated to both, type 1 and type 2 T cell responses
- Anti-CD25 treatment depletes treg cells and decreases disease severity in susceptible and resistant mice infected with Paracoccidioides brasiliensis
- CD28 exerts protective and detrimental effects in a pulmonary model of paracoccidioidomycosis
- TLR4 is used by a fungal pathogen to infect macrophages but also to induce protective immunity
- CD28 costimulatory molecule deficiency results in more severe Paracoccidioides brasiliensis infection but protects mice from life-threatening immunopathology
- Paracoccidioides brasiliensis-TLR-4 interaction induces macrophage activation but results in more severe paracoccidioimycosis
- Genetic deficiency of CD28 costimulatory molecule results in more severe Paracoccicioidomycosis (PCM) associated with decreased antibodies and cytokines production
- Genetic deficiency of IL-10 determines a less severe paracoccidioidomycosis associated with increased IL-12 secretion and enhanced T cell activation
- Susceptible and resistant mice to P. Brasiliensis infection use an indoleamine 2,3-dioxygenase mechanism to control fungal growth
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