Regulation of trypanosoma cruzi infection by type I interferons (2000)
- Autores:
- Autor USP: ABRAHAMSOHN, ISES DE ALMEIDA - ICB
- Unidade: ICB
- Assunto: IMMUNOLOGIA
- Idioma: Inglês
- Imprenta:
- Local: Florianópolis
- Data de publicação: 2000
- Fonte:
- Título do periódico: Abstract
- Nome do evento: Meeting of the Brazilian Society of Immunology
-
ABNT
COSTA, V M A e MENDONÇA, R. Z. e ABRAHAMSOHN, Ises de Almeida. Regulation of trypanosoma cruzi infection by type I interferons. 2000, Anais.. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2000. . Acesso em: 19 abr. 2024. -
APA
Costa, V. M. A., Mendonça, R. Z., & Abrahamsohn, I. de A. (2000). Regulation of trypanosoma cruzi infection by type I interferons. In Abstract. Florianópolis: Instituto de Ciências Biomédicas, Universidade de São Paulo. -
NLM
Costa VMA, Mendonça RZ, Abrahamsohn I de A. Regulation of trypanosoma cruzi infection by type I interferons. Abstract. 2000 ;[citado 2024 abr. 19 ] -
Vancouver
Costa VMA, Mendonça RZ, Abrahamsohn I de A. Regulation of trypanosoma cruzi infection by type I interferons. Abstract. 2000 ;[citado 2024 abr. 19 ] - Endogenously produced IL-10 and prostaglandins regulate peripheral blood mononuclear cells proliferation to Trypanosoma cruzi antigens
- Nitric oxide produced by macrophages and regulation of resistance to "Trypanosoma cruzi" at the initial phase of infection
- Egulation of Trypanosoma cruzi infection by type interferons
- Il-12 regulation of ifn-gama synthesis in the acute phase of trypanosoma cruzi infection
- IL-12 regulation of IFN-'gama' synthesis in the acute phase of Trypanosoma cruzi infection
- Prostaglandins mediate suppression of lymphoproliferation and cytokine synthesis during the early phase of Trypanosoma cruzi infection
- Il-12 enhances proliferation of peripheral blood mononuclear cells from chagasic patients to trypanosoma cruzi antigen
- Il-10 and ifn-gamma production by spleen cells in trypanosoma cruzi infected mice
- Bordetella pertussis and innate immune response: inhibition of NO production by murine BMDMO through TGF-ß arginase and TLR4
- Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation
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