Bilateral lesion of hypothalamic paraventricular nucleus abolishes fever induced by endotoxin and bradykinin in rats (1998)
- Authors:
- USP affiliated authors: PELA, IRENE ROSEMIR - FCFRP ; FRANCI, CELSO RODRIGUES - FMRP
- Unidades: FCFRP; FMRP
- Assunto: FARMACOLOGIA
- Language: Inglês
- Imprenta:
- Source:
- Título do periódico: Annals of the New York Academy of Sciences
- Volume/Número/Paginação/Ano: v. 856, p. 294-297, 1998
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ABNT
CALDEIRA, Jérri C e FRANCI, Celso Rodrigues e PELÁ, Irene Rosemir. Bilateral lesion of hypothalamic paraventricular nucleus abolishes fever induced by endotoxin and bradykinin in rats. Annals of the New York Academy of Sciences, v. 856, p. 294-297, 1998Tradução . . Acesso em: 19 set. 2024. -
APA
Caldeira, J. C., Franci, C. R., & Pelá, I. R. (1998). Bilateral lesion of hypothalamic paraventricular nucleus abolishes fever induced by endotoxin and bradykinin in rats. Annals of the New York Academy of Sciences, 856, 294-297. -
NLM
Caldeira JC, Franci CR, Pelá IR. Bilateral lesion of hypothalamic paraventricular nucleus abolishes fever induced by endotoxin and bradykinin in rats. Annals of the New York Academy of Sciences. 1998 ; 856 294-297.[citado 2024 set. 19 ] -
Vancouver
Caldeira JC, Franci CR, Pelá IR. Bilateral lesion of hypothalamic paraventricular nucleus abolishes fever induced by endotoxin and bradykinin in rats. Annals of the New York Academy of Sciences. 1998 ; 856 294-297.[citado 2024 set. 19 ] - CRH plays a role in the platelet-derived growth factor (PDGF)-induced fever
- Lesões bilaterais e seletiva do núcleo paraventricular (NPV) bloqueia a febre induzida por endotoxina e bradicinina em ratos
- Lesion of PVN parvocelullar neurons blocks fever induced by endotoxin in rats
- Mechanisms involved in the development of tolerance to the pyrogenic effect of endotoxin in rats
- Central involvement of kinin 'B IND.1' and 'B IND.2' receptors in the febrile response induced by endotoxin in rats
- Antinociceptive effect of carbomazepine in different experimental models of acute nociception
- Effect of galactosamine, an inhibitor of hepatic protein synthesis, on endotoxin-induced fever and pyrogenic tolerance
- Febrile response induced by platelet-derived growth factor (PDGF) in rats
- Effect of galactosamine, an inhibitor of hepatic protein synthesis, on endotoxin-induced fever and pyrogenic tolerance
- Novas evidencias sobre a participacao dos receptores para a bradicinina na febre induzida por endotoxina de e. Coli em ratos
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