Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells (1993)
- Authors:
- USP affiliated authors: ARMELIN, HUGO AGUIRRE - IQ ; KIMURA, EICO - IQ
- Unidade: IQ
- Subjects: BIOQUÍMICA; BIOLOGIA MOLECULAR
- Language: Inglês
- Imprenta:
- Publisher place: Washington
- Date published: 1993
- Source:
- Título do periódico: Biochemistry
- Volume/Número/Paginação/Ano: v.12, p.103-8, 1993
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ABNT
KIMURA, E e FRIGERI, C K e ARMELIN, Hugo Aguirre. Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells. Biochemistry, v. 12, p. 103-8, 1993Tradução . . Acesso em: 16 abr. 2024. -
APA
Kimura, E., Frigeri, C. K., & Armelin, H. A. (1993). Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells. Biochemistry, 12, 103-8. -
NLM
Kimura E, Frigeri CK, Armelin HA. Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells. Biochemistry. 1993 ;12 103-8.[citado 2024 abr. 16 ] -
Vancouver
Kimura E, Frigeri CK, Armelin HA. Kinetics of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth, dcamp and phorbol ester in mouse adrenocortical cells. Biochemistry. 1993 ;12 103-8.[citado 2024 abr. 16 ] - Kinetcs of inhibition in c-myc proto-oncogene expression and dna synthesis caused by acth dibuthyl camp and phorbol ester in mouse adrenocortical cells
- Relevance of c-fos proto-oncogene induction for the steroidogenic response to acth, da amp and phorbol ester in adrenocortical cells
- Transcriptional pattern of c-fos oncogene in mouse malignant adrenocortical cell cultures
- Phorbol ester mimicks adrenocorticotropic hormone (acth) action in adrenocortical mouse cells
- Role of proto-oncogene c-ki-ras amplification and overexpression in the malignancy of y-1 adrenocortical tumor cells
- Amplified ki-ras oncogene elimination as the basis for normal reversion in malignant adrenocortical cells
- Phorbol ester mimics acth action in corticoadrenal cells stimulating steroidogenesis blocking cell cycle, changing cell shape, and inducing c-fos proto-oncogene expression
- Growth control in mouse y-1 adrenocortical tumor cells: role of c-ki-ras amplification and effects of alth
- C-fos oncogene transcription pattern in mouse malignant adrenocortical cell cultures
- Dna synthesis inhibition by adrenocorticotropic hormone (acth) and a phorbol ester in y-1 corticoadrenal cells
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