ReP USP - Resultado da busca

Artigo de periodico
Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis (2022)
Vale, Aline Halfeld Fernandes ; Nascimento, Daniele Carvalho ; Alvarez, Annie Rocío Piñeros ; Ferreira, Raphael G. ; Santos, Jeimison Duarte ; Aragon, Davi Casale ; Cunha, Fernando de Queiroz ; Ramalho, Fernando Silva ; Alves Filho, José Carlos Farias ; Carlotti, Ana Paula de Carvalho Panzeri
Source: Brazilian Journal of Medical and Biological Research. Unidade: FMRP
Subjects: ANTI-INFLAMATÓRIOS, ANTIOXIDANTES, INFERÊNCIA BAYESIANA, QUIMIOCINAS, CREATINA, FARMACOTERAPIA
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ABNT
VALE, Aline Halfeld Fernandes et al. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis. Brazilian Journal of Medical and Biological Research, v. 55, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1590/1414-431X2022e12107. Acesso em: 16 nov. 2024.
APA
Vale, A. H. F., Nascimento, D. C., Alvarez, A. R. P., Ferreira, R. G., Santos, J. D., Aragon, D. C., et al. (2022). Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis. Brazilian Journal of Medical and Biological Research, 55, 1-8. doi:10.1590/1414-431X2022e12107
NLM
Vale AHF, Nascimento DC, Alvarez ARP, Ferreira RG, Santos JD, Aragon DC, Cunha F de Q, Ramalho FS, Alves Filho JCF, Carlotti AP de CP. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis [Internet]. Brazilian Journal of Medical and Biological Research. 2022 ; 55 1-8.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1590/1414-431X2022e12107
Vancouver
Vale AHF, Nascimento DC, Alvarez ARP, Ferreira RG, Santos JD, Aragon DC, Cunha F de Q, Ramalho FS, Alves Filho JCF, Carlotti AP de CP. Riboflavin did not provide anti-inflammatory or antioxidant effects in an experimental model of sepsis [Internet]. Brazilian Journal of Medical and Biological Research. 2022 ; 55 1-8.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1590/1414-431X2022e12107
Artigo de periodico
The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth (2021)
Teixeira, Silvia Aparecida; Viapiano, Mariano S.; Andrade, Augusto F.; Nandhu, Mohan S.; Pezuk, Julia Alejandra; Bidinotto, Lucas T.; Sant'Ana, Veridiana Kiill Suazo de; Serafini, Luciano Neder; Carlotti Júnior, Carlos Gilberto ; Becker, Aline P.; Tone, Luiz Gonzaga ; Scrideli, Carlos Alberto
Source: Molecular Neurobiology. Unidade: FMRP
Subjects: ANIDRASE CARBÔNICA, METABOLISMO CELULAR, NEOPLASIAS CEREBRAIS, FARMACOTERAPIA, LINHAGEM CELULAR, CICLO CELULAR, SULFONAMIDAS
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ABNT
TEIXEIRA, Silvia Aparecida et al. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, v. 58, n. 9, p. 4520-4534, 2021Tradução . . Disponível em: https://doi.org/10.1007/s12035-021-02437-3. Acesso em: 16 nov. 2024.
APA
Teixeira, S. A., Viapiano, M. S., Andrade, A. F., Nandhu, M. S., Pezuk, J. A., Bidinotto, L. T., et al. (2021). The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth. Molecular Neurobiology, 58( 9), 4520-4534. doi:10.1007/s12035-021-02437-3
NLM
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s12035-021-02437-3
Vancouver
Teixeira SA, Viapiano MS, Andrade AF, Nandhu MS, Pezuk JA, Bidinotto LT, Sant'Ana VKS de, Serafini LN, Carlotti Júnior CG, Becker AP, Tone LG, Scrideli CA. The carbonic anhydrase inhibitor E7070 sensitizes glioblastoma cells to radio and chemotherapy and reduces tumor growth [Internet]. Molecular Neurobiology. 2021 ; 58( 9): 4520-4534.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s12035-021-02437-3
Artigo de periodico
Use of Copaifera multijuga for acute corneal repair after chemical injury: A clinical, histopathological and toxicogenetic study (2017)
Dias, Fernanda Gosuen Gonçalves; Jorge, Adriana Torrecilhas; Pereira, Lucas de Freitas; Furtado, Ricardo Andrade; Ambrósio, Sérgio Ricardo; Bastos, Jairo Kenupp; Ramos, Salvador Boccaletti; Chahud, Fernando; Dias, Luis Gustavo Gosuen Gonçalves; Honsho, Cristiane dos Santos; Tavares, Denise Crispim
Source: Biomedicine and Pharmacotherapy. Unidades: FCFRP, FMRP
Subjects: BIOMEDICINA, FARMACOTERAPIA, COPAÍBA, CIÊNCIAS DA SAÚDE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DIAS, Fernanda Gosuen Gonçalves et al. Use of Copaifera multijuga for acute corneal repair after chemical injury: A clinical, histopathological and toxicogenetic study. Biomedicine and Pharmacotherapy, v. 96, p. 1193-1198, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.biopha.2017.11.099. Acesso em: 16 nov. 2024.
APA
Dias, F. G. G., Jorge, A. T., Pereira, L. de F., Furtado, R. A., Ambrósio, S. R., Bastos, J. K., et al. (2017). Use of Copaifera multijuga for acute corneal repair after chemical injury: A clinical, histopathological and toxicogenetic study. Biomedicine and Pharmacotherapy, 96, 1193-1198. doi:10.1016/j.biopha.2017.11.099
NLM
Dias FGG, Jorge AT, Pereira L de F, Furtado RA, Ambrósio SR, Bastos JK, Ramos SB, Chahud F, Dias LGGG, Honsho C dos S, Tavares DC. Use of Copaifera multijuga for acute corneal repair after chemical injury: A clinical, histopathological and toxicogenetic study [Internet]. Biomedicine and Pharmacotherapy. 2017 ; 96 1193-1198.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.biopha.2017.11.099
Vancouver
Dias FGG, Jorge AT, Pereira L de F, Furtado RA, Ambrósio SR, Bastos JK, Ramos SB, Chahud F, Dias LGGG, Honsho C dos S, Tavares DC. Use of Copaifera multijuga for acute corneal repair after chemical injury: A clinical, histopathological and toxicogenetic study [Internet]. Biomedicine and Pharmacotherapy. 2017 ; 96 1193-1198.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.biopha.2017.11.099
Artigo de periodico
Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration (2010)
Dal Secco, Daniela; Freitas, Andressa; Abreu, Monica A.; Garlet, Thiago P.; Rossi, Marcos Antonio; Ferreira, Sérgio Henrique; Silva, João Santana da; Alves-Filho, José C.; Cunha, Fernando Queiroz
Source: Naunyn-Schmiedeberg's Archives of Pharmacology. Unidade: FMRP
Subjects: NEUTRÓFILOS, ÓXIDO NÍTRICO, FARMACOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAL SECCO, Daniela et al. Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration. Naunyn-Schmiedeberg's Archives of Pharmacology, v. 381, n. 6, p. 483-493, 2010Tradução . . Disponível em: https://doi.org/10.1007/s00210-010-0500-2. Acesso em: 16 nov. 2024.
APA
Dal Secco, D., Freitas, A., Abreu, M. A., Garlet, T. P., Rossi, M. A., Ferreira, S. H., et al. (2010). Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration. Naunyn-Schmiedeberg's Archives of Pharmacology, 381( 6), 483-493. doi:10.1007/s00210-010-0500-2
NLM
Dal Secco D, Freitas A, Abreu MA, Garlet TP, Rossi MA, Ferreira SH, Silva JS da, Alves-Filho JC, Cunha FQ. Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2010 ; 381( 6): 483-493.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00210-010-0500-2
Vancouver
Dal Secco D, Freitas A, Abreu MA, Garlet TP, Rossi MA, Ferreira SH, Silva JS da, Alves-Filho JC, Cunha FQ. Reduction of ICAM-1 expression by carbon monoxide via soluble guanylate cyclase activation accounts for modulation of neutrophil migration [Internet]. Naunyn-Schmiedeberg's Archives of Pharmacology. 2010 ; 381( 6): 483-493.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1007/s00210-010-0500-2

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