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Artigo de periodico
Mechanisms regulating host cell death during Leishmania infection (2024)
Fernandes, Juliane Cristina Ribeiro; Zamboni, Dario Simões
Source: mBio. Unidades: ICB, FMRP
Subjects: CÉLULAS, LEISHMANIA, DOENÇAS PARASITÁRIAS, PROTOZOA
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FERNANDES, Juliane Cristina Ribeiro e ZAMBONI, Dario Simões. Mechanisms regulating host cell death during Leishmania infection. mBio, v. 15, n. 11, p. 1-11, 2024Tradução . . Disponível em: https://doi.org/10.1128/mbio.01980-23. Acesso em: 02 jul. 2025.
APA
Fernandes, J. C. R., & Zamboni, D. S. (2024). Mechanisms regulating host cell death during Leishmania infection. mBio, 15( 11), 1-11. doi:10.1128/mbio.01980-23
NLM
Fernandes JCR, Zamboni DS. Mechanisms regulating host cell death during Leishmania infection [Internet]. mBio. 2024 ; 15( 11): 1-11.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1128/mbio.01980-23
Vancouver
Fernandes JCR, Zamboni DS. Mechanisms regulating host cell death during Leishmania infection [Internet]. mBio. 2024 ; 15( 11): 1-11.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1128/mbio.01980-23
Artigo de periodico
Neutrophil virucidal activity against SARS-CoV-2 is mediated by neutrophil extracellular traps (2024)
Almeida, Cícero José Luíz dos Ramos; Veras, Flávio Protásio ; Paiva, Isadora Marques ; Schneider, Ayda Henriques; Silva, Juliana da Costa; Gomes, Giovanni Freitas; Costa, Victor Ferreira; Silva, Bruna Manuella Souza; Caetité, Diego ; Silva, Camila Meirelles Souza; Salina, Ana Carolina Guerta ; Martins, Ronaldo; Bonilha, Caio Santos ; Cunha, Larissa Dias da ; Jamur, Maria Celia ; Silva, Luis Lamberti Pinto da ; Arruda, Eurico ; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidades: FMRP, FM
Subjects: COVID-19, NEUTRÓFILOS, CORONAVIRUS, IMUNIDADE
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ALMEIDA, Cícero José Luíz dos Ramos et al. Neutrophil virucidal activity against SARS-CoV-2 is mediated by neutrophil extracellular traps. The Journal of Infectious Diseases, v. 229, n. 5, p. 1352-1365, 2024Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiad526. Acesso em: 02 jul. 2025.
APA
Almeida, C. J. L. dos R., Veras, F. P., Paiva, I. M., Schneider, A. H., Silva, J. da C., Gomes, G. F., et al. (2024). Neutrophil virucidal activity against SARS-CoV-2 is mediated by neutrophil extracellular traps. The Journal of Infectious Diseases, 229( 5), 1352-1365. doi:10.1093/infdis/jiad526
NLM
Almeida CJL dos R, Veras FP, Paiva IM, Schneider AH, Silva J da C, Gomes GF, Costa VF, Silva BMS, Caetité D, Silva CMS, Salina ACG, Martins R, Bonilha CS, Cunha LD da, Jamur MC, Silva LLP da, Arruda E, Zamboni DS, Louzada Júnior P, Oliveira RDR de, Alves Filho JCF, Cunha TM, Cunha F de Q. Neutrophil virucidal activity against SARS-CoV-2 is mediated by neutrophil extracellular traps [Internet]. The Journal of Infectious Diseases. 2024 ; 229( 5): 1352-1365.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/infdis/jiad526
Vancouver
Almeida CJL dos R, Veras FP, Paiva IM, Schneider AH, Silva J da C, Gomes GF, Costa VF, Silva BMS, Caetité D, Silva CMS, Salina ACG, Martins R, Bonilha CS, Cunha LD da, Jamur MC, Silva LLP da, Arruda E, Zamboni DS, Louzada Júnior P, Oliveira RDR de, Alves Filho JCF, Cunha TM, Cunha F de Q. Neutrophil virucidal activity against SARS-CoV-2 is mediated by neutrophil extracellular traps [Internet]. The Journal of Infectious Diseases. 2024 ; 229( 5): 1352-1365.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/infdis/jiad526
Artigo de periodico
The unique Legionella longbeachae capsule favors intracellular replication and immune evasion (2024)
Schmidt, Silke; Mondino, Sonia; Gomez-Valero, Laura; Escoll, Pedro; Mascarenhas, Danielle Pini Alves ; Gonçalves, Augusto Cesar ; Camara, Pedro Henrique M.; Garcia Rodriguez, Francisco J.; Rusniok, Christophe; Sachse, Martin; Moya-Nilges, Maryse; Fontaine, Thierry; Zamboni, Dario Simões ; Buchrieser, Carmen
Source: PLOS Pathogens. Unidade: FMRP
Assunto: LEGIONELLA
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SCHMIDT, Silke et al. The unique Legionella longbeachae capsule favors intracellular replication and immune evasion. PLOS Pathogens, v. 20, n. 9, p. 1-35, 2024Tradução . . Disponível em: https://doi.org/10.1371/journal.ppat.1012534. Acesso em: 02 jul. 2025.
APA
Schmidt, S., Mondino, S., Gomez-Valero, L., Escoll, P., Mascarenhas, D. P. A., Gonçalves, A. C., et al. (2024). The unique Legionella longbeachae capsule favors intracellular replication and immune evasion. PLOS Pathogens, 20( 9), 1-35. doi:10.1371/journal.ppat.1012534
NLM
Schmidt S, Mondino S, Gomez-Valero L, Escoll P, Mascarenhas DPA, Gonçalves AC, Camara PHM, Garcia Rodriguez FJ, Rusniok C, Sachse M, Moya-Nilges M, Fontaine T, Zamboni DS, Buchrieser C. The unique Legionella longbeachae capsule favors intracellular replication and immune evasion [Internet]. PLOS Pathogens. 2024 ; 20( 9): 1-35.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1371/journal.ppat.1012534
Vancouver
Schmidt S, Mondino S, Gomez-Valero L, Escoll P, Mascarenhas DPA, Gonçalves AC, Camara PHM, Garcia Rodriguez FJ, Rusniok C, Sachse M, Moya-Nilges M, Fontaine T, Zamboni DS, Buchrieser C. The unique Legionella longbeachae capsule favors intracellular replication and immune evasion [Internet]. PLOS Pathogens. 2024 ; 20( 9): 1-35.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1371/journal.ppat.1012534
Artigo de periodico
Innate immune responses and monocyte-derived phagocyte recruitment in protective immunity to pathogenic bacteria: insights from Legionella pneumophila (2024)
Mascarenhas, Danielle Pini Alves ; Zamboni, Dario Simões
Source: Current Opinion in Microbiology. Unidade: FMRP
Subjects: LEGIONELLA PNEUMOPHILA, IMUNIDADE, BACTÉRIAS PATOGÊNICAS, FAGÓCITOS
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MASCARENHAS, Danielle Pini Alves e ZAMBONI, Dario Simões. Innate immune responses and monocyte-derived phagocyte recruitment in protective immunity to pathogenic bacteria: insights from Legionella pneumophila. Current Opinion in Microbiology, v. 80, 2024Tradução . . Disponível em: https://doi.org/10.1016/j.mib.2024.102495. Acesso em: 02 jul. 2025.
APA
Mascarenhas, D. P. A., & Zamboni, D. S. (2024). Innate immune responses and monocyte-derived phagocyte recruitment in protective immunity to pathogenic bacteria: insights from Legionella pneumophila. Current Opinion in Microbiology, 80. doi:10.1016/j.mib.2024.102495
NLM
Mascarenhas DPA, Zamboni DS. Innate immune responses and monocyte-derived phagocyte recruitment in protective immunity to pathogenic bacteria: insights from Legionella pneumophila [Internet]. Current Opinion in Microbiology. 2024 ; 80[citado 2025 jul. 02 ] Available from: https://doi.org/10.1016/j.mib.2024.102495
Vancouver
Mascarenhas DPA, Zamboni DS. Innate immune responses and monocyte-derived phagocyte recruitment in protective immunity to pathogenic bacteria: insights from Legionella pneumophila [Internet]. Current Opinion in Microbiology. 2024 ; 80[citado 2025 jul. 02 ] Available from: https://doi.org/10.1016/j.mib.2024.102495
Artigo de periodico
Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity (2023)
Albornoz, Sandra Patrícia Palma ; Silva, Thais Fernanda de Campo Fraga da ; Carvalho, Renan Villanova Homem de ; Rodrigues, Tamara Silva; Gembre, Ana Flávia; Oliveira, Rômulo Silva de; Souza, Fernanda Mesquita de; Corrêa, Giseli Furlan ; Ramalho, Leandra Naira Zambelli ; Sartori, Daniela Carlos ; Câmara, Niels Olsen Saraiva ; Zamboni, Dario Simões ; Takahashi, Viviani Nardini ; Sorgi, Carlos Artério ; Faccioli, Lucia Helena ; Medeiros, Alexandra Ivo de ; Costa, Diego Luís ; Bonato, Vânia Luiza Deperon
Source: Journal of Pathology. Unidades: FMRP, ICB, FCFRP, FFCLRP, ESALQ
Assunto: IMUNOLOGIA
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ALBORNOZ, Sandra Patrícia Palma et al. Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity. Journal of Pathology, v. 259, p. 291-303, 2023Tradução . . Disponível em: https://doi.org/10.1002/path.6041. Acesso em: 02 jul. 2025.
APA
Albornoz, S. P. P., Silva, T. F. de C. F. da, Carvalho, R. V. H. de, Rodrigues, T. S., Gembre, A. F., Oliveira, R. S. de, et al. (2023). Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity. Journal of Pathology, 259, 291-303. doi:10.1002/path.6041
NLM
Albornoz SPP, Silva TF de CF da, Carvalho RVH de, Rodrigues TS, Gembre AF, Oliveira RS de, Souza FM de, Corrêa GF, Ramalho LNZ, Sartori DC, Câmara NOS, Zamboni DS, Takahashi VN, Sorgi CA, Faccioli LH, Medeiros AI de, Costa DL, Bonato VLD. Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity [Internet]. Journal of Pathology. 2023 ; 259 291-303.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1002/path.6041
Vancouver
Albornoz SPP, Silva TF de CF da, Carvalho RVH de, Rodrigues TS, Gembre AF, Oliveira RS de, Souza FM de, Corrêa GF, Ramalho LNZ, Sartori DC, Câmara NOS, Zamboni DS, Takahashi VN, Sorgi CA, Faccioli LH, Medeiros AI de, Costa DL, Bonato VLD. Cell death induced by NLRP3–palmitate axis impairs pulmonary damage tolerance and aggravates immunopathology during obesity-tuberculosis comorbidity [Internet]. Journal of Pathology. 2023 ; 259 291-303.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1002/path.6041
Artigo de periodico
Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients (2023)
Amaral, Natália Brasil do; Rodrigues, Tamara Silva; Giannini, Marcela Cavichioli; Lopes, Maria Isabel Fernandes; Bonjorno, Letícia Pastorelli; Menezes, Pamella Indira da Silva Oliveira; Dib, Saulo Musse; Gigante, Samara Libich Gusmão; Benatti, Maíra Nilson; Rezek, Uebe C.; Emrich-Filho, Laerte L.; Sousa, Betania A. A.; Almeida, Sérgio Couto Luna de; Luppino-Assad, Rodrigo; Veras, Flávio Protásio ; Schneider, Ayda Henriques; Leiria, Luiz Osório Silveira ; Cunha, Larissa Dias da; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Miranda, Carlos Henrique ; Pazin Filho, Antônio ; Martins, Maria Auxiliadora ; Borges, Marcos de Carvalho ; Fonseca, Benedito Antonio Lopes da ; Bollela, Valdes Roberto ; Del-Ben, Cristina Marta ; Cunha, Fernando de Queiroz ; Santana, Rodrigo de Carvalho ; Vilar, Fernando Crivelenti; Zamboni, Dario Simões ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de
Source: Inflammation Research. Unidade: FMRP
Subjects: COVID-19, CITOCINAS, PROTEÍNAS, ALCALOIDES
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AMARAL, Natália Brasil do et al. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, v. 72, n. 5, p. 895-899, 2023Tradução . . Disponível em: https://doi.org/10.1007/s00011-023-01718-y. Acesso em: 02 jul. 2025.
APA
Amaral, N. B. do, Rodrigues, T. S., Giannini, M. C., Lopes, M. I. F., Bonjorno, L. P., Menezes, P. I. da S. O., et al. (2023). Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients. Inflammation Research, 72( 5), 895-899. doi:10.1007/s00011-023-01718-y
NLM
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Vancouver
Amaral NB do, Rodrigues TS, Giannini MC, Lopes MIF, Bonjorno LP, Menezes PI da SO, Dib SM, Gigante SLG, Benatti MN, Rezek UC, Emrich-Filho LL, Sousa BAA, Almeida SCL de, Luppino-Assad R, Veras FP, Schneider AH, Leiria LOS, Cunha LD da, Alves Filho JCF, Cunha TM, Arruda Neto E de, Miranda CH, Pazin Filho A, Martins MA, Borges M de C, Fonseca BAL da, Bollela VR, Del-Ben CM, Cunha F de Q, Santana R de C, Vilar FC, Zamboni DS, Louzada Júnior P, Oliveira RDR de. Colchicine reduces the activation of NLRP3 inflammasome in COVID-19 patients [Internet]. Inflammation Research. 2023 ; 72( 5): 895-899.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1007/s00011-023-01718-y
Artigo de periodico
C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps (2023)
Silva, Bruna Manuella Souza; Gomes, Giovanni Freitas; Veras, Flávio Protásio ; Cambier, Seppe; Silva, Gabriel Victor Lucena da; Quadros, Andreza Urba de; Caetité, Diego Brito; Nascimento, Daniele Carvalho ; Silva, Camilla M.; Silva, Juliana C. ; Damasceno, Samara; Schneider, Ayda Henriques ; Beretta, Fabio; Batah, Sabrina Setembre ; Castro, Icaro M. S.; Paiva, Isadora Marques ; Rodrigues, Tamara Silva ; Salina, Ana Carolina Guerta ; Martins Júnior, Ronaldo Bragança; Cebinelli, Guilherme Cesar Martelossi ; Bibo, Naira Lopes; Jorge, Daniel M.; Nakaya, Helder Takashi Imoto ; Zamboni, Dario Simões ; Leiria, Luiz Osório Silveira ; Fabro, Alexandre Todorovic ; Alves Filho, José Carlos Farias ; Arruda Neto, Eurico de; Louzada Júnior, Paulo ; Oliveira, Renê Donizete Ribeiro de; Cunha, Larissa Dias da; Mol, Pierre Van; Vanderbeke, Lore; Feys, Simon; Wauters, Els; Brandolini, Laura; Aramini, Andrea; Cunha, Fernando Q.; Köhl, Jörg; Allegretti, Marcello; Lambrechts, Diether; Wauters, Joost; Proost, Paul; Cunha, Thiago Mattar
Source: Journal of Clinical Investigation. Unidades: FCFRP, FMRP
Subjects: COVID-19, PULMÃO, INFLAMAÇÃO, IMUNIDADE
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SILVA, Bruna Manuella Souza et al. C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps. Journal of Clinical Investigation, v. 133, n. 12, p. 1-18, 2023Tradução . . Disponível em: https://doi.org/10.1172/JCI163105. Acesso em: 02 jul. 2025.
APA
Silva, B. M. S., Gomes, G. F., Veras, F. P., Cambier, S., Silva, G. V. L. da, Quadros, A. U. de, et al. (2023). C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps. Journal of Clinical Investigation, 133( 12), 1-18. doi:10.1172/JCI163105
NLM
Silva BMS, Gomes GF, Veras FP, Cambier S, Silva GVL da, Quadros AU de, Caetité DB, Nascimento DC, Silva CM, Silva JC, Damasceno S, Schneider AH, Beretta F, Batah SS, Castro IMS, Paiva IM, Rodrigues TS, Salina ACG, Martins Júnior RB, Cebinelli GCM, Bibo NL, Jorge DM, Nakaya HTI, Zamboni DS, Leiria LOS, Fabro AT, Alves Filho JCF, Arruda Neto E de, Louzada Júnior P, Oliveira RDR de, Cunha LD da, Mol PV, Vanderbeke L, Feys S, Wauters E, Brandolini L, Aramini A, Cunha FQ, Köhl J, Allegretti M, Lambrechts D, Wauters J, Proost P, Cunha TM. C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps [Internet]. Journal of Clinical Investigation. 2023 ; 133( 12): 1-18.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1172/JCI163105
Vancouver
Silva BMS, Gomes GF, Veras FP, Cambier S, Silva GVL da, Quadros AU de, Caetité DB, Nascimento DC, Silva CM, Silva JC, Damasceno S, Schneider AH, Beretta F, Batah SS, Castro IMS, Paiva IM, Rodrigues TS, Salina ACG, Martins Júnior RB, Cebinelli GCM, Bibo NL, Jorge DM, Nakaya HTI, Zamboni DS, Leiria LOS, Fabro AT, Alves Filho JCF, Arruda Neto E de, Louzada Júnior P, Oliveira RDR de, Cunha LD da, Mol PV, Vanderbeke L, Feys S, Wauters E, Brandolini L, Aramini A, Cunha FQ, Köhl J, Allegretti M, Lambrechts D, Wauters J, Proost P, Cunha TM. C5aR1 signaling triggers lung immunopathology in COVID-19 through neutrophil extracellular traps [Internet]. Journal of Clinical Investigation. 2023 ; 133( 12): 1-18.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1172/JCI163105
Artigo de periodico
Inflammasome activation and CCR2-mediated monocyte-derived dendritic cell recruitment restrict Legionella pneumophila infection (2023)
Ataíde, Marco A.; Manin, Graziele Zenaro; Oliveira, Samuel S.; Guerra, Rhanoica Oliveira ; Zamboni, Dario Simões
Source: European Journal of Immunology. Unidade: FMRP
Subjects: IMUNIDADE, CÉLULAS DENDRÍTICAS, MONÓCITOS, LEGIONELLA
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ATAÍDE, Marco A. et al. Inflammasome activation and CCR2-mediated monocyte-derived dendritic cell recruitment restrict Legionella pneumophila infection. European Journal of Immunology, v. 53, n. 2, p. 1-13, 2023Tradução . . Disponível em: https://doi.org/10.1002/eji.202249985. Acesso em: 02 jul. 2025.
APA
Ataíde, M. A., Manin, G. Z., Oliveira, S. S., Guerra, R. O., & Zamboni, D. S. (2023). Inflammasome activation and CCR2-mediated monocyte-derived dendritic cell recruitment restrict Legionella pneumophila infection. European Journal of Immunology, 53( 2), 1-13. doi:10.1002/eji.202249985
NLM
Ataíde MA, Manin GZ, Oliveira SS, Guerra RO, Zamboni DS. Inflammasome activation and CCR2-mediated monocyte-derived dendritic cell recruitment restrict Legionella pneumophila infection [Internet]. European Journal of Immunology. 2023 ; 53( 2): 1-13.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1002/eji.202249985
Vancouver
Ataíde MA, Manin GZ, Oliveira SS, Guerra RO, Zamboni DS. Inflammasome activation and CCR2-mediated monocyte-derived dendritic cell recruitment restrict Legionella pneumophila infection [Internet]. European Journal of Immunology. 2023 ; 53( 2): 1-13.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1002/eji.202249985
Artigo de periodico
Gasdermin-D activation promotes NLRP3 activation and host resistance to Leishmania infection (2023)
Sá, Keyla Santos Guedes de ; Amaral, Luana Alexandrina; Rodrigues, Tamara Silva ; Ishimoto, Adriene Yumi; Andrade, Warrison Athanásio Coelho de; Almeida, Leticia de; Freitas-Castro, Felipe ; Batah, Sabrina Setembre ; Oliveira, Sergio C.; Pastorello, Mônica Tempest; Fabro, Alexandre Todorovic ; Zamboni, Dario Simões
Source: Nature Communications. Unidade: FMRP
Subjects: PEPTÍDEOS, PROTEÍNAS, LEISHMANIA, APOPTOSE
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ABNT
SÁ, Keyla Santos Guedes de et al. Gasdermin-D activation promotes NLRP3 activation and host resistance to Leishmania infection. Nature Communications, v. 14, n. 1, p. 1-19, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41467-023-36626-6. Acesso em: 02 jul. 2025.
APA
Sá, K. S. G. de, Amaral, L. A., Rodrigues, T. S., Ishimoto, A. Y., Andrade, W. A. C. de, Almeida, L. de, et al. (2023). Gasdermin-D activation promotes NLRP3 activation and host resistance to Leishmania infection. Nature Communications, 14( 1), 1-19. doi:10.1038/s41467-023-36626-6
NLM
Sá KSG de, Amaral LA, Rodrigues TS, Ishimoto AY, Andrade WAC de, Almeida L de, Freitas-Castro F, Batah SS, Oliveira SC, Pastorello MT, Fabro AT, Zamboni DS. Gasdermin-D activation promotes NLRP3 activation and host resistance to Leishmania infection [Internet]. Nature Communications. 2023 ; 14( 1): 1-19.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1038/s41467-023-36626-6
Vancouver
Sá KSG de, Amaral LA, Rodrigues TS, Ishimoto AY, Andrade WAC de, Almeida L de, Freitas-Castro F, Batah SS, Oliveira SC, Pastorello MT, Fabro AT, Zamboni DS. Gasdermin-D activation promotes NLRP3 activation and host resistance to Leishmania infection [Internet]. Nature Communications. 2023 ; 14( 1): 1-19.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1038/s41467-023-36626-6
Artigo de periodico
AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity (2023)
Leite, Jefferson Antonio ; Menezes, Luísa ; Martins, Eloisa ; Rodrigues, Tamara Silva; Tavares, Lucas ; Ebering, Anna ; Schelmbauer, Carsten ; Cebinelli, Guilherme Cesar Martelossi ; Zinina, Valeriya ; Golden, Artemy ; Soshnikova, Natalia ; Zamboni, Dario S. ; Cunha, Fernando Q. ; Magdalena Huber ; Waisman, Ari ; Silva, João Santana da ; Sartori, Daniela Carlos ; Câmara, Niels Olsen Saraiva
Source: iScience. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, CAMUNDONGOS, PROTEÍNAS, DIFERENCIAÇÃO CELULAR, LINFÓCITOS T, CÉLULAS SANGUÍNEAS
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ABNT
LEITE, Jefferson Antonio et al. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity. iScience, v. 26, n. 11, p. art. 108134. 19 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108134. Acesso em: 02 jul. 2025.
APA
Leite, J. A., Menezes, L., Martins, E., Rodrigues, T. S., Tavares, L., Ebering, A., et al. (2023). AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity. iScience, 26( 11), art. 108134. 19 . doi:10.1016/j.isci.2023.108134
NLM
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Sartori DC, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2025 jul. 02 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Vancouver
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Sartori DC, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2025 jul. 02 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Artigo de periodico
Tonsils are major sites of persistence of SARS-CoV-2 in children (2023)
Lima, Thais Melquiades de; Martins Júnior, Ronaldo Bragança; Miura, Carolina Sponchiado; Souza, Maria Vitória Oliveira de; Cassiano, Murilo Henrique Anzolini ; Rodrigues, Tamara Silva ; Veras, Flávio Protásio ; Sousa, Josane de Freitas ; Gomes, Rogério; Almeida, Glaucia Maria ; Melo, Stella Rezende; Silva, Gabriela Condé da; Dias, Matheus; Capato, Carlos Fabiano; Silva, Maria Lúcia; Luiz, Veridiana Ester Dias de Barros; Carenzi, Lucas Rodrigues; Zamboni, Dario Simões ; Jorge, Daniel Macedo de Melo; Cunha, Fernando de Queiroz ; Tamashiro, Edwin ; Anselmo-Lima, Wilma Terezinha ; Valera, Fabiana Cardoso Pereira ; Arruda Neto, Eurico de
Source: Microbiology Spectrum. Unidades: FCFRP, FMRP
Subjects: TONSILA, ADENOIDES, VÍRUS, DOENÇAS INFECCIOSAS, PEDIATRIA, OTORRINOLARINGOLOGIA
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ABNT
LIMA, Thais Melquiades de et al. Tonsils are major sites of persistence of SARS-CoV-2 in children. Microbiology Spectrum, v. 11, n. 5, p. 1-14, 2023Tradução . . Disponível em: https://doi.org/10.1128/spectrum.01347-23. Acesso em: 02 jul. 2025.
APA
Lima, T. M. de, Martins Júnior, R. B., Miura, C. S., Souza, M. V. O. de, Cassiano, M. H. A., Rodrigues, T. S., et al. (2023). Tonsils are major sites of persistence of SARS-CoV-2 in children. Microbiology Spectrum, 11( 5), 1-14. doi:10.1128/spectrum.01347-23
NLM
Lima TM de, Martins Júnior RB, Miura CS, Souza MVO de, Cassiano MHA, Rodrigues TS, Veras FP, Sousa J de F, Gomes R, Almeida GM, Melo SR, Silva GC da, Dias M, Capato CF, Silva ML, Luiz VED de B, Carenzi LR, Zamboni DS, Jorge DM de M, Cunha F de Q, Tamashiro E, Anselmo-Lima WT, Valera FCP, Arruda Neto E de. Tonsils are major sites of persistence of SARS-CoV-2 in children [Internet]. Microbiology Spectrum. 2023 ; 11( 5): 1-14.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1128/spectrum.01347-23
Vancouver
Lima TM de, Martins Júnior RB, Miura CS, Souza MVO de, Cassiano MHA, Rodrigues TS, Veras FP, Sousa J de F, Gomes R, Almeida GM, Melo SR, Silva GC da, Dias M, Capato CF, Silva ML, Luiz VED de B, Carenzi LR, Zamboni DS, Jorge DM de M, Cunha F de Q, Tamashiro E, Anselmo-Lima WT, Valera FCP, Arruda Neto E de. Tonsils are major sites of persistence of SARS-CoV-2 in children [Internet]. Microbiology Spectrum. 2023 ; 11( 5): 1-14.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1128/spectrum.01347-23
Artigo de periodico
Coxiella co-opts the Glutathione Peroxidase 4 to protect the host cell from oxidative stress-induced cell death (2023)
Loterio, Robson Kriiger; Thomas, David R.; Andrade, Warrison Athanásio Coelho de; Lee, Yi Wei; Santos, Leonardo Lima dos ; Mascarenhas, Danielle Pini Alves; Steiner, Thiago M.; Chiaratto, Jéssica; Fielden, Laura F.; Lopes, Letícia; Bird, Lauren E.; Goldman, Gustavo Henrique ; Stojanovski, Diana; Scott, Nichollas E.; Zamboni, Dario Simões ; Newton, Hayley J.
Source: Proceedings National Academy of Sciences. Unidades: FMRP, FCFRP, FORP
Subjects: ANTIOXIDANTES, MORTE, ESTRESSE OXIDATIVO, FOSFOLIPÍDEOS, GLUTATIONA TRANSFERASE, PEROXIDASE
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ABNT
LOTERIO, Robson Kriiger et al. Coxiella co-opts the Glutathione Peroxidase 4 to protect the host cell from oxidative stress-induced cell death. Proceedings National Academy of Sciences, v. 120, n. 36, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1073/pnas.2308752120. Acesso em: 02 jul. 2025.
APA
Loterio, R. K., Thomas, D. R., Andrade, W. A. C. de, Lee, Y. W., Santos, L. L. dos, Mascarenhas, D. P. A., et al. (2023). Coxiella co-opts the Glutathione Peroxidase 4 to protect the host cell from oxidative stress-induced cell death. Proceedings National Academy of Sciences, 120( 36), 1-11. doi:10.1073/pnas.2308752120
NLM
Loterio RK, Thomas DR, Andrade WAC de, Lee YW, Santos LL dos, Mascarenhas DPA, Steiner TM, Chiaratto J, Fielden LF, Lopes L, Bird LE, Goldman GH, Stojanovski D, Scott NE, Zamboni DS, Newton HJ. Coxiella co-opts the Glutathione Peroxidase 4 to protect the host cell from oxidative stress-induced cell death [Internet]. Proceedings National Academy of Sciences. 2023 ; 120( 36): 1-11.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1073/pnas.2308752120
Vancouver
Loterio RK, Thomas DR, Andrade WAC de, Lee YW, Santos LL dos, Mascarenhas DPA, Steiner TM, Chiaratto J, Fielden LF, Lopes L, Bird LE, Goldman GH, Stojanovski D, Scott NE, Zamboni DS, Newton HJ. Coxiella co-opts the Glutathione Peroxidase 4 to protect the host cell from oxidative stress-induced cell death [Internet]. Proceedings National Academy of Sciences. 2023 ; 120( 36): 1-11.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1073/pnas.2308752120
Artigo de periodico
The TIGIT+ T regulatory cells subset associates with nosocomial infection and fatal outcome in COVID-19 patients under mechanical ventilation (2023)
Lima, Mikhael Haruo Fernandes de ; Machado, Caio Cavalcante ; Nascimento, Daniele Carvalho ; Silva, Camila Meirelles de Souza; Toller-kawahisa, Juliana Escher ; Rodrigues, Tamara Silva ; Veras, Flávio Protásio ; Pontelli, Marjorie Cornejo ; Castro, Ítalo Araújo de ; Zamboni, Dario Simões ; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Cunha, Larissa Dias da; Oliveira, Renê Donizeti Ribeiro de; Cunha, Fernando Queiroz ; Louzada Júnior, Paulo
Source: Scientific Reports. Unidades: FMRP, FCFRP
Subjects: BACTEREMIA, COVID-19, RECEPTORES IMUNOLÓGICOS, RESPIRAÇÃO ARTIFICIAL, LINFÓCITOS T
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. The TIGIT+ T regulatory cells subset associates with nosocomial infection and fatal outcome in COVID-19 patients under mechanical ventilation. Scientific Reports, v. 13, n. 1, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1038/s41598-023-39924-7. Acesso em: 02 jul. 2025.
APA
Lima, M. H. F. de, Machado, C. C., Nascimento, D. C., Silva, C. M. de S., Toller-kawahisa, J. E., Rodrigues, T. S., et al. (2023). The TIGIT+ T regulatory cells subset associates with nosocomial infection and fatal outcome in COVID-19 patients under mechanical ventilation. Scientific Reports, 13( 1), 1-12. doi:10.1038/s41598-023-39924-7
NLM
Lima MHF de, Machado CC, Nascimento DC, Silva CM de S, Toller-kawahisa JE, Rodrigues TS, Veras FP, Pontelli MC, Castro ÍA de, Zamboni DS, Alves Filho JCF, Cunha TM, Arruda Neto E de, Cunha LD da, Oliveira RDR de, Cunha FQ, Louzada Júnior P. The TIGIT+ T regulatory cells subset associates with nosocomial infection and fatal outcome in COVID-19 patients under mechanical ventilation [Internet]. Scientific Reports. 2023 ; 13( 1): 1-12.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1038/s41598-023-39924-7
Vancouver
Lima MHF de, Machado CC, Nascimento DC, Silva CM de S, Toller-kawahisa JE, Rodrigues TS, Veras FP, Pontelli MC, Castro ÍA de, Zamboni DS, Alves Filho JCF, Cunha TM, Arruda Neto E de, Cunha LD da, Oliveira RDR de, Cunha FQ, Louzada Júnior P. The TIGIT+ T regulatory cells subset associates with nosocomial infection and fatal outcome in COVID-19 patients under mechanical ventilation [Internet]. Scientific Reports. 2023 ; 13( 1): 1-12.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1038/s41598-023-39924-7
Artigo de periodico
CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation (2023)
Rodrigues, Tamara Silva ; Caetano, Camila C. S.; Sá, Keyla Santos Guedes de ; Almeida, Leticia de; Becerra, Amanda de Matos; Gonçalves, Augusto Velozo ; Lopes, Letícia de Sousa; Oliveira, Samuel; Mascarenhas, Danielle Pini Alves; Batah, Sabrina Setembre ; Silva, Bruna Manuella Souza; Gomes, Giovanni Freitas; Castro, Ricardo de; Martins Júnior, Ronaldo Bragança; Avila, Jonathan; Frantz, Fabiani Gai ; Cunha, Thiago Mattar ; Arruda Neto, Eurico de; Cunha, Fernando Queiroz ; Helder Takashi Imoto Nakaya ; Cunha, Larissa Dias da; Fabro, Alexandre Todorovic ; Louzada Júnior, Paulo ; Oliveira, Renê Donizeti Ribeiro de; Zamboni, Dario Simões
Source: The Journal of Infectious Diseases. Unidades: FMRP, FCFRP
Subjects: COVID-19, MACRÓFAGOS, CORONAVIRUS
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ABNT
RODRIGUES, Tamara Silva et al. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation. The Journal of Infectious Diseases, v. 227, n. 12, p. 1364-1375, 2023Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiad037. Acesso em: 02 jul. 2025.
APA
Rodrigues, T. S., Caetano, C. C. S., Sá, K. S. G. de, Almeida, L. de, Becerra, A. de M., Gonçalves, A. V., et al. (2023). CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation. The Journal of Infectious Diseases, 227( 12), 1364-1375. doi:10.1093/infdis/jiad037
NLM
Rodrigues TS, Caetano CCS, Sá KSG de, Almeida L de, Becerra A de M, Gonçalves AV, Lopes L de S, Oliveira S, Mascarenhas DPA, Batah SS, Silva BMS, Gomes GF, Castro R de, Martins Júnior RB, Avila J, Frantz FG, Cunha TM, Arruda Neto E de, Cunha FQ, Helder Takashi Imoto Nakaya, Cunha LD da, Fabro AT, Louzada Júnior P, Oliveira RDR de, Zamboni DS. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation [Internet]. The Journal of Infectious Diseases. 2023 ; 227( 12): 1364-1375.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/infdis/jiad037
Vancouver
Rodrigues TS, Caetano CCS, Sá KSG de, Almeida L de, Becerra A de M, Gonçalves AV, Lopes L de S, Oliveira S, Mascarenhas DPA, Batah SS, Silva BMS, Gomes GF, Castro R de, Martins Júnior RB, Avila J, Frantz FG, Cunha TM, Arruda Neto E de, Cunha FQ, Helder Takashi Imoto Nakaya, Cunha LD da, Fabro AT, Louzada Júnior P, Oliveira RDR de, Zamboni DS. CASP4/11 contributes to NLRP3 activation and COVID-19 exacerbation [Internet]. The Journal of Infectious Diseases. 2023 ; 227( 12): 1364-1375.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/infdis/jiad037
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 02 jul. 2025.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection (2022)
Almeida, Leticia de; Silva, Alexandre Luiz Neves; Rodrigues, Tamara Silva; Oliveira, Samuel; Ishimoto, Adriene Yumi; Seribelli, Amanda Aparecida; Becerra, Amanda; Andrade, Warrison A; Ataíde, Marco Antônio; Caetano, Camila C. S.; Sá, Keyla Santos Guedes de; Pelisson, Natália; Martins, Ronaldo Bragança; Souza, Juliano de Paula; Arruda, Eurico; Batah, Sabrina Setembre; Castro, Ricardo; Frantz, Fabiani Gai; Cunha, Fernando Queiróz; Cunha, Thiago Mattar; Fabro, Alexandre Todorovic; Cunha, Larissa Dias; Louzada-Junior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões
Source: Science Advances. Unidades: FMRP, FCFRP
Subjects: INFECÇÕES RESPIRATÓRIAS, COVID-19, ADJUVANTES IMUNOLÓGICOS, INFLAMAÇÃO
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ABNT
ALMEIDA, Leticia de et al. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, v. 8, n. 37, 2022Tradução . . Disponível em: https://doi.org/10.1126/sciadv.abo5400. Acesso em: 02 jul. 2025.
APA
Almeida, L. de, Silva, A. L. N., Rodrigues, T. S., Oliveira, S., Ishimoto, A. Y., Seribelli, A. A., et al. (2022). Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection. Science Advances, 8( 37). doi:10.1126/sciadv.abo5400
NLM
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2025 jul. 02 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Vancouver
Almeida L de, Silva ALN, Rodrigues TS, Oliveira S, Ishimoto AY, Seribelli AA, Becerra A, Andrade WA, Ataíde MA, Caetano CCS, Sá KSG de, Pelisson N, Martins RB, Souza J de P, Arruda E, Batah SS, Castro R, Frantz FG, Cunha FQ, Cunha TM, Fabro AT, Cunha LD, Louzada-Junior P, Oliveira RDR de, Zamboni DS. Identification of immunomodulatory drugs that inhibit multiple inflammasomes and impair SARS-CoV-2 infection [Internet]. Science Advances. 2022 ; 8( 37):[citado 2025 jul. 02 ] Available from: https://doi.org/10.1126/sciadv.abo5400
Artigo de periodico
SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients (2022)
Pontelli, Marjorie C.; Castro, Ítalo A.; Martins, Ronaldo B; La Serra, Leonardo; Veras, Flávio P.; Nascimento, Daniele C; Silva, Camila M.; Cardoso, Ricardo S; Rosales, Roberta Ribeiro Costa; Gomes, Rogério; Lima, Thais M.; Souza, Juliano P.; Vitti, Brenda C.; Caetité, Diego B.; Lima, Mikhael Haruo Fernandes de; Stumpf, Spencer D; Thompson, Cassandra E; Bloyet, Louis-Marie; Toller-Kawahisa, Juliana E.; Giannini, Marcela C.; Bonjorno, Letícia P; Lopes, Maria Isabel Fernandes; Batah, Sabrina Setembre; Siyuan, Li; Luppino-Assad, Rodrigo; Almeida, Sergio C L; Oliveira, Fabiola R; Benatti, Maíra N; Pontes, Lorena Lobo de Figueiredo; Santana, Rodrigo Carvalho; Vilar, Fernando C; Auxiliadora-Martins, Maria; Shi, Pei-Yong; Cunha, Thiago Mattar; Calado, Rodrigo Tocantins; Alves-Filho, José Carlos; Zamboni, Dario Simões; Fabro, Alexandre Todorovic; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro; Whelan, Sean P J; Cunha, Fernando Queiróz; Arruda, Eurico
Source: Journal of Molecular Cell Biology. Unidades: FCFRP, FMRP
Subjects: COVID-19, LINFÓCITOS, MONÓCITOS, APOPTOSE
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ABNT
PONTELLI, Marjorie C. et al. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, v. 14, n. 4, 2022Tradução . . Disponível em: https://doi.org/10.1093/jmcb/mjac021. Acesso em: 02 jul. 2025.
APA
Pontelli, M. C., Castro, Í. A., Martins, R. B., La Serra, L., Veras, F. P., Nascimento, D. C., et al. (2022). SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients. Journal of Molecular Cell Biology, 14( 4). doi:10.1093/jmcb/mjac021
NLM
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Vancouver
Pontelli MC, Castro ÍA, Martins RB, La Serra L, Veras FP, Nascimento DC, Silva CM, Cardoso RS, Rosales RRC, Gomes R, Lima TM, Souza JP, Vitti BC, Caetité DB, Lima MHF de, Stumpf SD, Thompson CE, Bloyet L-M, Toller-Kawahisa JE, Giannini MC, Bonjorno LP, Lopes MIF, Batah SS, Siyuan L, Luppino-Assad R, Almeida SCL, Oliveira FR, Benatti MN, Pontes LL de F, Santana RC, Vilar FC, Auxiliadora-Martins M, Shi P-Y, Cunha TM, Calado RT, Alves-Filho JC, Zamboni DS, Fabro AT, Louzada Júnior P, Oliveira RDR, Whelan SPJ, Cunha FQ, Arruda E. SARS-CoV-2 productively infects primary human immune system cells in vitro and in COVID-19 patients [Internet]. Journal of Molecular Cell Biology. 2022 ; 14( 4):[citado 2025 jul. 02 ] Available from: https://doi.org/10.1093/jmcb/mjac021
Artigo de periodico-carta/editorial
COVID-19 bimodal clinical and pathological phenotypes. [Carta] (2022)
Batah, Sabrina S.; Benatti, Maira N.; Siyuan, Li; Telini, Wagner M.; Barboza, Jamile O.; Menezes, Marcelo B.; Nadai, Tales Rubens de ; Sá, Keyla Santos Guedes de; Vaswani, Chirag M.; Gupta, Sahil; Zamboni, Dario Simões; Rodrigues, Rodrigo do Tocantins Calado de Saloma; Oliveira, Renê Donizeti Ribeiro de; Louzada Júnior, Paulo; Martins, Maria Auxiliadora; Louzada Júnior, Paulo; Veras, Flávio Protásio; Cunha, Larissa Dias da; Cunha, Thiago Mattar; Arruda Neto, Eurico de; Chahud, Fernando; Santos, Marcel Koenigkam; Cetlin, Andrea Antunes; Cunha, Fernando de Queiroz; Fabro, Alexandre Todorovic; Capelozzi, Vera Luiza
Source: Clinical and Translational Medicine. Unidades: FM, FMRP, FCFRP
Subjects: COVID-19, FENÓTIPOS, CARTAS TERAPÊUTICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BATAH, Sabrina S. et al. COVID-19 bimodal clinical and pathological phenotypes. [Carta]. Clinical and Translational Medicine. Chichester: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1002/ctm2.648. Acesso em: 02 jul. 2025. , 2022
APA
Batah, S. S., Benatti, M. N., Siyuan, L., Telini, W. M., Barboza, J. O., Menezes, M. B., et al. (2022). COVID-19 bimodal clinical and pathological phenotypes. [Carta]. Clinical and Translational Medicine. Chichester: Faculdade de Medicina, Universidade de São Paulo. doi:10.1002/ctm2.648
NLM
Batah SS, Benatti MN, Siyuan L, Telini WM, Barboza JO, Menezes MB, Nadai TR de, Sá KSG de, Vaswani CM, Gupta S, Zamboni DS, Rodrigues R do TC de S, Oliveira RDR de, Louzada Júnior P, Martins MA, Louzada Júnior P, Veras FP, Cunha LD da, Cunha TM, Arruda Neto E de, Chahud F, Santos MK, Cetlin AA, Cunha F de Q, Fabro AT, Capelozzi VL. COVID-19 bimodal clinical and pathological phenotypes. [Carta] [Internet]. Clinical and Translational Medicine. 2022 ; 12( 1):[citado 2025 jul. 02 ] Available from: https://doi.org/10.1002/ctm2.648
Vancouver
Batah SS, Benatti MN, Siyuan L, Telini WM, Barboza JO, Menezes MB, Nadai TR de, Sá KSG de, Vaswani CM, Gupta S, Zamboni DS, Rodrigues R do TC de S, Oliveira RDR de, Louzada Júnior P, Martins MA, Louzada Júnior P, Veras FP, Cunha LD da, Cunha TM, Arruda Neto E de, Chahud F, Santos MK, Cetlin AA, Cunha F de Q, Fabro AT, Capelozzi VL. COVID-19 bimodal clinical and pathological phenotypes. [Carta] [Internet]. Clinical and Translational Medicine. 2022 ; 12( 1):[citado 2025 jul. 02 ] Available from: https://doi.org/10.1002/ctm2.648
Curadoria
São Paulo School of Advanced Science on pathogenic trypanosomatids: from basic biology to pathogenesis and new therapies (2022)
Cruz, Angela Kaysel (cur) ; Zamboni, Dario Simões (cur) ; Andricopulo, Adriano Defini (cur)
Unidades: FMRP, IFSC
Subjects: DOENÇAS NEGLIGENCIADAS, LEISHMANIA, DOENÇA DE CHAGAS, EVENTOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
São Paulo School of Advanced Science on pathogenic trypanosomatids: from basic biology to pathogenesis and new therapies. . Ribeirão Preto: Universidade de São Paulo - USP. . Acesso em: 02 jul. 2025. , 2022
APA
São Paulo School of Advanced Science on pathogenic trypanosomatids: from basic biology to pathogenesis and new therapies. (2022). São Paulo School of Advanced Science on pathogenic trypanosomatids: from basic biology to pathogenesis and new therapies. Ribeirão Preto: Universidade de São Paulo - USP.
NLM
São Paulo School of Advanced Science on pathogenic trypanosomatids: from basic biology to pathogenesis and new therapies. 2022 ;[citado 2025 jul. 02 ]
Vancouver
São Paulo School of Advanced Science on pathogenic trypanosomatids: from basic biology to pathogenesis and new therapies. 2022 ;[citado 2025 jul. 02 ]
Artigo de periodico
Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage (2022)
Costa, Tiago Januário da ; Potje, Simone Regina ; Silva, Thais Fernanda de Campos Fraga da; Silva Neto, Júlio Alves da; Barros, Paula Rodrigues de; Rodrigues, Daniel; Machado, Mirele Resende; Martins Júnior, Ronaldo Bragança; Eichler, Rosangela Aparecida dos Santos; Benatti, Maira N.; Sá, Keyla Santos Guedes de; Almado, Carlos Eduardo Lopes; Castro, Italo A.; Pontelli, Marjorie C.; Serra, Leonardo La; Carneiro, Fernando Silva ; Becari, Christiane; Louzada Júnior, Paulo; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões; Arruda, Eurico; Martins, Maria Auxiliadora; Giachini, Fernanda R. C.; Bonato, Vânia Luiza Deperon; Zachara, Natasha E.; Bomfim, Gisele F.; Tostes, Rita de Cássia Aleixo
Source: Vascular Pharmacology. Unidades: FCFRP, ICB, FMRP
Subjects: COVID-19, MITOCÔNDRIAS, CÉLULAS ENDOTELIAIS, DNA MITOCONDRIAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Tiago Januário da et al. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, v. 142, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2021.106946. Acesso em: 02 jul. 2025.
APA
Costa, T. J. da, Potje, S. R., Silva, T. F. de C. F. da, Silva Neto, J. A. da, Barros, P. R. de, Rodrigues, D., et al. (2022). Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage. Vascular Pharmacology, 142. doi:10.1016/j.vph.2021.106946
NLM
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2025 jul. 02 ] Available from: https://doi.org/10.1016/j.vph.2021.106946
Vancouver
Costa TJ da, Potje SR, Silva TF de CF da, Silva Neto JA da, Barros PR de, Rodrigues D, Machado MR, Martins Júnior RB, Eichler RA dos S, Benatti MN, Sá KSG de, Almado CEL, Castro IA, Pontelli MC, Serra LL, Carneiro FS, Becari C, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Arruda E, Martins MA, Giachini FRC, Bonato VLD, Zachara NE, Bomfim GF, Tostes R de CA. Mitochondrial DNA and TLR9 activation contribute to SARS-CoV-2-induced endothelial cell damage [Internet]. Vascular Pharmacology. 2022 ; 142[citado 2025 jul. 02 ] Available from: https://doi.org/10.1016/j.vph.2021.106946

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