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  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      FONTES, M. T. et al. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, v. 94, n. 1, p. 24-29, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.017. Acesso em: 04 out. 2024.
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      Fontes, M. T., Silva, T. L. B. T., Mota, M. M., Barreto, A. S., Rossoni, L. V., & Santos, M. R. V. (2014). Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, 94( 1), 24-29. doi:10.1016/j.lfs.2013.11.017
    • NLM

      Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
    • Vancouver

      Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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      SILVA, Ariel S. da et al. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, v. 91, n. 24-26, p. 1174-1179, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.05.001. Acesso em: 04 out. 2024.
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      Silva, A. S. da, Dias, L. D., Borges, J. F., Markoski, M. M., Souza, M. S. de, Irigoyen, M. C., et al. (2013). Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, 91( 24-26), 1174-1179. doi:10.1016/j.lfs.2013.05.001
    • NLM

      Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001
    • Vancouver

      Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001
  • Source: Life Sciences. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      VILLELA, Darine et al. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland. Life Sciences, v. 94, n. 2, p. 122-129, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.004. Acesso em: 04 out. 2024.
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      Villela, D., Lima, L. de S., Peres, R., Peliciari-Garcia, R. A., Amaral, F. G. do, Cipolla-Neto, J., et al. (2013). Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland. Life Sciences, 94( 2), 122-129. doi:10.1016/j.lfs.2013.11.004
    • NLM

      Villela D, Lima L de S, Peres R, Peliciari-Garcia RA, Amaral FG do, Cipolla-Neto J, Scavone C, Afeche SC. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland [Internet]. Life Sciences. 2013 ; 94( 2): 122-129.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004
    • Vancouver

      Villela D, Lima L de S, Peres R, Peliciari-Garcia RA, Amaral FG do, Cipolla-Neto J, Scavone C, Afeche SC. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland [Internet]. Life Sciences. 2013 ; 94( 2): 122-129.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004
  • Source: Life Sciences. Unidade: ICB

    Assunto: FARMACOLOGIA

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      MAZIERO, Aline Mendes et al. Inhibition of human platelet aggregation by eosinophils. Life Sciences, v. 93, n. 9-11, p. 416-422, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.07.012. Acesso em: 04 out. 2024.
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      Maziero, A. M., Lorenzetti, R., Donato, J. L., Lilla, S., & De Nucci, G. (2013). Inhibition of human platelet aggregation by eosinophils. Life Sciences, 93( 9-11), 416-422. doi:10.1016/j.lfs.2013.07.012
    • NLM

      Maziero AM, Lorenzetti R, Donato JL, Lilla S, De Nucci G. Inhibition of human platelet aggregation by eosinophils [Internet]. Life Sciences. 2013 ; 93( 9-11): 416-422.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.07.012
    • Vancouver

      Maziero AM, Lorenzetti R, Donato JL, Lilla S, De Nucci G. Inhibition of human platelet aggregation by eosinophils [Internet]. Life Sciences. 2013 ; 93( 9-11): 416-422.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.07.012
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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      DAVID-SILVA, Aline et al. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats. Life Sciences, v. 93, n. 22, p. 805-813, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.10.011. Acesso em: 04 out. 2024.
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      David-Silva, A., Freitas, H. S. de, Okamoto, M. M., Sabino-Silva, R., Schaan, B. D., & Machado, U. F. (2013). Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats. Life Sciences, 93( 22), 805-813. doi:10.1016/j.lfs.2013.10.011
    • NLM

      David-Silva A, Freitas HS de, Okamoto MM, Sabino-Silva R, Schaan BD, Machado UF. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats [Internet]. Life Sciences. 2013 ; 93( 22): 805-813.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.10.011
    • Vancouver

      David-Silva A, Freitas HS de, Okamoto MM, Sabino-Silva R, Schaan BD, Machado UF. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats [Internet]. Life Sciences. 2013 ; 93( 22): 805-813.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.10.011
  • Source: Life Sciences. Unidade: ICB

    Assunto: FARMACOLOGIA

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      RAMOS-FILHO, Antonio Celso S. et al. The renin–angiotensin system plays a major role in voiding dysfunction of ovariectomized rats. Life Sciences, v. 93, n. 22, p. 820-829, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.09.008. Acesso em: 04 out. 2024.
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      Ramos-Filho, A. C. S., Faria, J. A., Calmasini, F. B., Teixeira, S. A., Mónica, F. Z., Muscará, M. N., et al. (2013). The renin–angiotensin system plays a major role in voiding dysfunction of ovariectomized rats. Life Sciences, 93( 22), 820-829. doi:10.1016/j.lfs.2013.09.008
    • NLM

      Ramos-Filho ACS, Faria JA, Calmasini FB, Teixeira SA, Mónica FZ, Muscará MN, Gontijo JAR, Anhê GF, Zanesco A, Antunes E. The renin–angiotensin system plays a major role in voiding dysfunction of ovariectomized rats [Internet]. Life Sciences. 2013 ; 93( 22): 820-829.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.09.008
    • Vancouver

      Ramos-Filho ACS, Faria JA, Calmasini FB, Teixeira SA, Mónica FZ, Muscará MN, Gontijo JAR, Anhê GF, Zanesco A, Antunes E. The renin–angiotensin system plays a major role in voiding dysfunction of ovariectomized rats [Internet]. Life Sciences. 2013 ; 93( 22): 820-829.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2013.09.008
  • Source: Life Sciences. Unidades: ICB, FMRP

    Assunto: FARMACOLOGIA

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      FILGUEIRA, Fernando Paranaíba et al. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, v. 90, n. 17-18, p. 689-694, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2012.01.017. Acesso em: 04 out. 2024.
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      Filgueira, F. P., Lobato, N. de S., Dos Santos, R. A., Oliveira, M. A., Akamine, E. H., Tostes-Passaglia, R. de C. A., et al. (2012). Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats. Life Sciences, 90( 17-18), 689-694. doi:10.1016/j.lfs.2012.01.017
    • NLM

      Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
    • Vancouver

      Filgueira FP, Lobato N de S, Dos Santos RA, Oliveira MA, Akamine EH, Tostes-Passaglia R de CA, Fortes ZB, Carvalho MHC de. Endogenous testosterone increases leukocyte–endothelial cell interaction in spontaneously hypertensive rats [Internet]. Life Sciences. 2012 ; 90( 17-18): 689-694.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
  • Source: Life Sciences. Unidade: ICB

    Assunto: FARMACOLOGIA

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      PONZIO, Beatriz Felice et al. Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring. Life Sciences, v. 90, n. 15-16, p. 571-577, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2012.01.017. Acesso em: 04 out. 2024.
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      Ponzio, B. F., Carvalho, M. H. C., Fortes, Z. B., & Franco, M. do C. (2012). Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring. Life Sciences, 90( 15-16), 571-577. doi:10.1016/j.lfs.2012.01.017
    • NLM

      Ponzio BF, Carvalho MHC, Fortes ZB, Franco M do C. Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring [Internet]. Life Sciences. 2012 ; 90( 15-16): 571-577.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
    • Vancouver

      Ponzio BF, Carvalho MHC, Fortes ZB, Franco M do C. Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring [Internet]. Life Sciences. 2012 ; 90( 15-16): 571-577.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
  • Source: Life Sciences. Unidade: ICB

    Subjects: FARMACOLOGIA, HISTOLOGIA

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      FONSECA, Eveline Aparecida Isquierdo et al. Metformin reduces the stimulatory effect of obesity on in vivo Walker-256 tumor development and increases the area of tumor necrosis. Life Sciences, v. 88, n. 19-20, p. 846-852, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2011.03.005. Acesso em: 04 out. 2024.
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      Fonseca, E. A. I., Oliveira, M. A. de, Lobato, N. de S., Akamine, E. H., Colquhoun, A., Carvalho, M. H. C. de, et al. (2011). Metformin reduces the stimulatory effect of obesity on in vivo Walker-256 tumor development and increases the area of tumor necrosis. Life Sciences, 88( 19-20), 846-852. doi:10.1016/j.lfs.2011.03.005
    • NLM

      Fonseca EAI, Oliveira MA de, Lobato N de S, Akamine EH, Colquhoun A, Carvalho MHC de, Zyngier SB, Fortes ZB. Metformin reduces the stimulatory effect of obesity on in vivo Walker-256 tumor development and increases the area of tumor necrosis [Internet]. Life Sciences. 2011 ; 88( 19-20): 846-852.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2011.03.005
    • Vancouver

      Fonseca EAI, Oliveira MA de, Lobato N de S, Akamine EH, Colquhoun A, Carvalho MHC de, Zyngier SB, Fortes ZB. Metformin reduces the stimulatory effect of obesity on in vivo Walker-256 tumor development and increases the area of tumor necrosis [Internet]. Life Sciences. 2011 ; 88( 19-20): 846-852.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2011.03.005
  • Source: Life Sciences. Unidades: ICB, EEFE

    Assunto: FARMACOLOGIA

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      ZAMO, F. S. et al. The renin–angiotensin system is modulated by swimming training depending on the age of spontaneously hypertensive rats. Life Sciences, v. 89, n. 3-4, p. 93-99, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2011.05.004. Acesso em: 04 out. 2024.
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      Zamo, F. S., Barauna, V. G., Chiavegatto, S., Irigoyen, M. C., & Oliveira, E. M. (2011). The renin–angiotensin system is modulated by swimming training depending on the age of spontaneously hypertensive rats. Life Sciences, 89( 3-4), 93-99. doi:10.1016/j.lfs.2011.05.004
    • NLM

      Zamo FS, Barauna VG, Chiavegatto S, Irigoyen MC, Oliveira EM. The renin–angiotensin system is modulated by swimming training depending on the age of spontaneously hypertensive rats [Internet]. Life Sciences. 2011 ; 89( 3-4): 93-99.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2011.05.004
    • Vancouver

      Zamo FS, Barauna VG, Chiavegatto S, Irigoyen MC, Oliveira EM. The renin–angiotensin system is modulated by swimming training depending on the age of spontaneously hypertensive rats [Internet]. Life Sciences. 2011 ; 89( 3-4): 93-99.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2011.05.004
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

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      GALLO, Luana C. et al. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, v. 87, n. 9-10 p. 302-308, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2010.07.005. Acesso em: 04 out. 2024.
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      Gallo, L. C., Davel, A. P. C., Xavier, F. E., & Rossoni, L. V. (2010). Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, 87( 9-10 p. 302-308). doi:10.1016/j.lfs.2010.07.005
    • NLM

      Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
    • Vancouver

      Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
  • Source: Life Sciences. Unidade: FCF

    Subjects: SUPLEMENTAÇÃO ALIMENTAR, AMINOÁCIDOS, EXERCÍCIO FÍSICO

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      ARAÚJO JÚNIOR, Jonas Alves de et al. Effect of chronic supplementation with branched-chain amino acids on the performance and hepatic and muscle glycogen content in trained rats. Life Sciences, v. 79, n. 14, p. 1343-1348, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2006.03.045. Acesso em: 04 out. 2024.
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      Araújo Júnior, J. A. de, Falavigna, G., Rogero, M. M., Pires, I. S. de O., Pedrosa, R. G., Castro, I. A. de, et al. (2006). Effect of chronic supplementation with branched-chain amino acids on the performance and hepatic and muscle glycogen content in trained rats. Life Sciences, 79( 14), 1343-1348. doi:10.1016/j.lfs.2006.03.045
    • NLM

      Araújo Júnior JA de, Falavigna G, Rogero MM, Pires IS de O, Pedrosa RG, Castro IA de, Donato Junior J, Tirapegui J. Effect of chronic supplementation with branched-chain amino acids on the performance and hepatic and muscle glycogen content in trained rats [Internet]. Life Sciences. 2006 ; 79( 14): 1343-1348.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2006.03.045
    • Vancouver

      Araújo Júnior JA de, Falavigna G, Rogero MM, Pires IS de O, Pedrosa RG, Castro IA de, Donato Junior J, Tirapegui J. Effect of chronic supplementation with branched-chain amino acids on the performance and hepatic and muscle glycogen content in trained rats [Internet]. Life Sciences. 2006 ; 79( 14): 1343-1348.[citado 2024 out. 04 ] Available from: https://doi.org/10.1016/j.lfs.2006.03.045

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