Subjects: SEPSE (COMPLICAÇÕES), INFLAMAÇÃO, MACRÓFAGOS, CITOCINAS, ENDOTOXINAS, CAMUNDONGOS, MODELOS ANIMAIS
ABNT
GODOY, Luiz C. et al. Loss of CD40 endogenous s-nitrosylation during inflammatory response in endotoxemic mice and patients with sepsis. Shock, v. 33, n. 6, p. 626-633, 2010Tradução . . Disponível em: https://doi.org/10.1097/SHK.0b013e3181cb88e6. Acesso em: 24 ago. 2024.APA
Godoy, L. C., Moretti, A. I., Jurado, M. C., Oxer, D., Janiszewski, M., Ckless, K., et al. (2010). Loss of CD40 endogenous s-nitrosylation during inflammatory response in endotoxemic mice and patients with sepsis. Shock, 33( 6), 626-633. doi:10.1097/SHK.0b013e3181cb88e6NLM
Godoy LC, Moretti AI, Jurado MC, Oxer D, Janiszewski M, Ckless K, Velasco IT, Laurindo FRM, Souza HP. Loss of CD40 endogenous s-nitrosylation during inflammatory response in endotoxemic mice and patients with sepsis [Internet]. Shock. 2010 ; 33( 6): 626-633.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1097/SHK.0b013e3181cb88e6Vancouver
Godoy LC, Moretti AI, Jurado MC, Oxer D, Janiszewski M, Ckless K, Velasco IT, Laurindo FRM, Souza HP. Loss of CD40 endogenous s-nitrosylation during inflammatory response in endotoxemic mice and patients with sepsis [Internet]. Shock. 2010 ; 33( 6): 626-633.[citado 2024 ago. 24 ] Available from: https://doi.org/10.1097/SHK.0b013e3181cb88e6