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Artigo de periodico
Clinical implication of 14-3-3 epsilon expression in gastric cancer (2012)
Leal, Mariana Ferreira; Calcagno, Danielle Queiroz; Demachki, Sâmia; Assumpção, Paulo Pimentel; Chammas, Roger; Burbano, Rommel Rodríguez; Smith, Marília de Arruda Cardoso
Source: World Journal of Gastroenterology. Unidade: FM
Subjects: NEOPLASIAS GÁSTRICAS (GENÉTICA;IMUNOLOGIA), WESTERN BLOTTING, EXPRESSÃO GÊNICA, PROTEÍNAS PROTO-ONCOGÊNICAS
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ABNT
LEAL, Mariana Ferreira et al. Clinical implication of 14-3-3 epsilon expression in gastric cancer. World Journal of Gastroenterology, v. 18, n. 13, p. 1531-1537, 2012Tradução . . Disponível em: https://doi.org/10.3748/wjg.v18.i13.1531. Acesso em: 16 nov. 2024.
APA
Leal, M. F., Calcagno, D. Q., Demachki, S., Assumpção, P. P., Chammas, R., Burbano, R. R., & Smith, M. de A. C. (2012). Clinical implication of 14-3-3 epsilon expression in gastric cancer. World Journal of Gastroenterology, 18( 13), 1531-1537. doi:10.3748/wjg.v18.i13.1531
NLM
Leal MF, Calcagno DQ, Demachki S, Assumpção PP, Chammas R, Burbano RR, Smith M de AC. Clinical implication of 14-3-3 epsilon expression in gastric cancer [Internet]. World Journal of Gastroenterology. 2012 ; 18( 13): 1531-1537.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3748/wjg.v18.i13.1531
Vancouver
Leal MF, Calcagno DQ, Demachki S, Assumpção PP, Chammas R, Burbano RR, Smith M de AC. Clinical implication of 14-3-3 epsilon expression in gastric cancer [Internet]. World Journal of Gastroenterology. 2012 ; 18( 13): 1531-1537.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3748/wjg.v18.i13.1531
Artigo de periodico
The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice (2012)
Brand, Camila; Oliveira, Felipe L.; Takiya, Christina M.; Palumbo Jr, Antônio; Hsu, Daniel K.; Liu, Fu-Tong; Borojevic, Radovan; Chammas, Roger; El-Cheikh, Márcia C.
Source: Histology and Histopathology. Unidade: FM
Subjects: SCHISTOSOMA MANSONI (PATOLOGIA), ESQUISTOSSOMOSE MANSONI (PATOLOGIA), CAMUNDONGOS, DOENÇA CRÔNICA, DOENÇAS INFECCIOSAS (PARASITOLOGIA)
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ABNT
BRAND, Camila et al. The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice. Histology and Histopathology, v. 27, n. 8, p. 1109-1120, 2012Tradução . . Disponível em: http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf. Acesso em: 16 nov. 2024.
APA
Brand, C., Oliveira, F. L., Takiya, C. M., Palumbo Jr, A., Hsu, D. K., Liu, F. -T., et al. (2012). The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice. Histology and Histopathology, 27( 8), 1109-1120. Recuperado de http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf
NLM
Brand C, Oliveira FL, Takiya CM, Palumbo Jr A, Hsu DK, Liu F-T, Borojevic R, Chammas R, El-Cheikh MC. The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice [Internet]. Histology and Histopathology. 2012 ; 27( 8): 1109-1120.[citado 2024 nov. 16 ] Available from: http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf
Vancouver
Brand C, Oliveira FL, Takiya CM, Palumbo Jr A, Hsu DK, Liu F-T, Borojevic R, Chammas R, El-Cheikh MC. The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice [Internet]. Histology and Histopathology. 2012 ; 27( 8): 1109-1120.[citado 2024 nov. 16 ] Available from: http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf
Artigo de periodico
Fibronectin expression is decreased in metastatic renal cell carcinoma following endostatin gene therapy (2012)
Chaves, Karen Cristina Barbosa; Turaça, Lauro Thiago; Pesquero, João Bosco; Mennecier, Gregory; Dagli, Maria Lúcia Zaidan ; Chammas, Roger; Schor, Nestor; Bellini, Maria Helena
Source: Biomedicine & Pharmacotherapy. Unidades: FMVZ, FM
Subjects: NEOPLASIAS RENAIS, WESTERN BLOTTING, METÁSTASE NEOPLÁSICA, FIBRONECTINAS, FIBROBLASTOS, GLICOPROTEÍNAS, EXPRESSÃO GÊNICA, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
CHAVES, Karen Cristina Barbosa et al. Fibronectin expression is decreased in metastatic renal cell carcinoma following endostatin gene therapy. Biomedicine & Pharmacotherapy, v. 66, n. 6, p. 464-468, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.biopha.2012.04.003. Acesso em: 16 nov. 2024.
APA
Chaves, K. C. B., Turaça, L. T., Pesquero, J. B., Mennecier, G., Dagli, M. L. Z., Chammas, R., et al. (2012). Fibronectin expression is decreased in metastatic renal cell carcinoma following endostatin gene therapy. Biomedicine & Pharmacotherapy, 66( 6), 464-468. doi:10.1016/j.biopha.2012.04.003
NLM
Chaves KCB, Turaça LT, Pesquero JB, Mennecier G, Dagli MLZ, Chammas R, Schor N, Bellini MH. Fibronectin expression is decreased in metastatic renal cell carcinoma following endostatin gene therapy [Internet]. Biomedicine & Pharmacotherapy. 2012 ; 66( 6): 464-468.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.biopha.2012.04.003
Vancouver
Chaves KCB, Turaça LT, Pesquero JB, Mennecier G, Dagli MLZ, Chammas R, Schor N, Bellini MH. Fibronectin expression is decreased in metastatic renal cell carcinoma following endostatin gene therapy [Internet]. Biomedicine & Pharmacotherapy. 2012 ; 66( 6): 464-468.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.biopha.2012.04.003
Artigo de periodico
Expression of PAFR as Part of a Prosurvival Response to Chemotherapy: A Novel Target for Combination Therapy in Melanoma (2012)
Onuchic, Ana Claudia; Machado, Camila M. L.; Saito, Renata F.; Rios, Francisco J.; Jancar, Sonia ; Chammas, Roger
Source: Mediators of Inflammation. Unidade: FM
Subjects: WESTERN BLOTTING, CITOMETRIA DE FLUXO, ESTRESSE OXIDATIVO, FATOR DE ATIVAÇÃO DE PLAQUETAS, MELANOMA (QUIMIOTERAPIA), PROGNÓSTICO, TERAPIA COMBINADA, EXPRESSÃO GÊNICA
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ABNT
ONUCHIC, Ana Claudia et al. Expression of PAFR as Part of a Prosurvival Response to Chemotherapy: A Novel Target for Combination Therapy in Melanoma. Mediators of Inflammation, v. 2012, p. 1-6, 2012Tradução . . Disponível em: https://doi.org/10.1155/2012/175408. Acesso em: 16 nov. 2024.
APA
Onuchic, A. C., Machado, C. M. L., Saito, R. F., Rios, F. J., Jancar, S., & Chammas, R. (2012). Expression of PAFR as Part of a Prosurvival Response to Chemotherapy: A Novel Target for Combination Therapy in Melanoma. Mediators of Inflammation, 2012, 1-6. doi:10.1155/2012/175408
NLM
Onuchic AC, Machado CML, Saito RF, Rios FJ, Jancar S, Chammas R. Expression of PAFR as Part of a Prosurvival Response to Chemotherapy: A Novel Target for Combination Therapy in Melanoma [Internet]. Mediators of Inflammation. 2012 ; 2012 1-6.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1155/2012/175408
Vancouver
Onuchic AC, Machado CML, Saito RF, Rios FJ, Jancar S, Chammas R. Expression of PAFR as Part of a Prosurvival Response to Chemotherapy: A Novel Target for Combination Therapy in Melanoma [Internet]. Mediators of Inflammation. 2012 ; 2012 1-6.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1155/2012/175408
Artigo de periodico
The inactive form of glycogen synthase kinase-3β is associated with the development of carcinomas in galectin-3 wild-type mice, but not in galectin-3-deficient mice (2012)
Mendonça, Daniella Fernandes; Chammas, Roger; Liu, Fu-Tong; Nonogaki, Suely; Cardoso, Sergio Vitorino; Loyola, Adriano Mota; Faria, Paulo Rogério de
Source: International Journal of Clinical and Experimental Medicine. Unidade: FM
Subjects: CARCINOMA (DESENVOLVIMENTO), CAMUNDONGOS, NEOPLASIAS BUCAIS (PATOLOGIA), CARCINOGÊNESE BUCAL, MODELOS ANIMAIS, MICROSCOPIA ELETRÔNICA, IMUNOHISTOQUÍMICA
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ABNT
MENDONÇA, Daniella Fernandes et al. The inactive form of glycogen synthase kinase-3β is associated with the development of carcinomas in galectin-3 wild-type mice, but not in galectin-3-deficient mice. International Journal of Clinical and Experimental Medicine, v. 5, n. 6, p. 547-554, 2012Tradução . . Disponível em: http://www.ijcep.com/files/IJCEP1203005.pdf. Acesso em: 16 nov. 2024.
APA
Mendonça, D. F., Chammas, R., Liu, F. -T., Nonogaki, S., Cardoso, S. V., Loyola, A. M., & Faria, P. R. de. (2012). The inactive form of glycogen synthase kinase-3β is associated with the development of carcinomas in galectin-3 wild-type mice, but not in galectin-3-deficient mice. International Journal of Clinical and Experimental Medicine, 5( 6), 547-554. Recuperado de http://www.ijcep.com/files/IJCEP1203005.pdf
NLM
Mendonça DF, Chammas R, Liu F-T, Nonogaki S, Cardoso SV, Loyola AM, Faria PR de. The inactive form of glycogen synthase kinase-3β is associated with the development of carcinomas in galectin-3 wild-type mice, but not in galectin-3-deficient mice [Internet]. International Journal of Clinical and Experimental Medicine. 2012 ; 5( 6): 547-554.[citado 2024 nov. 16 ] Available from: http://www.ijcep.com/files/IJCEP1203005.pdf
Vancouver
Mendonça DF, Chammas R, Liu F-T, Nonogaki S, Cardoso SV, Loyola AM, Faria PR de. The inactive form of glycogen synthase kinase-3β is associated with the development of carcinomas in galectin-3 wild-type mice, but not in galectin-3-deficient mice [Internet]. International Journal of Clinical and Experimental Medicine. 2012 ; 5( 6): 547-554.[citado 2024 nov. 16 ] Available from: http://www.ijcep.com/files/IJCEP1203005.pdf
Artigo de periodico
Endostatin neoadjuvant gene therapy extends survival in an orthotopic metastatic mouse model of renal cell carcinoma (2012)
Braga, Marina de Souza; Chaves, Karen Barbosa; Chammas, Roger; Schor, Nestor; Bellini, Maria Helena
Source: Biomedicine & Pharmacotherapy. Unidade: FM
Subjects: NEOPLASIAS RENAIS (CIRURGIA), CARCINOMA (QUIMIOTERAPIA), METÁSTASE NEOPLÁSICA, MODELOS ANIMAIS, PROLIFERAÇÃO CELULAR, CAMUNDONGOS, NEDRECTOMIA, FATORES DE RISCO
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ABNT
BRAGA, Marina de Souza et al. Endostatin neoadjuvant gene therapy extends survival in an orthotopic metastatic mouse model of renal cell carcinoma. Biomedicine & Pharmacotherapy, v. 66, n. 4, p. 237-241, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.biopha.2011.11.002. Acesso em: 16 nov. 2024.
APA
Braga, M. de S., Chaves, K. B., Chammas, R., Schor, N., & Bellini, M. H. (2012). Endostatin neoadjuvant gene therapy extends survival in an orthotopic metastatic mouse model of renal cell carcinoma. Biomedicine & Pharmacotherapy, 66( 4), 237-241. doi:10.1016/j.biopha.2011.11.002
NLM
Braga M de S, Chaves KB, Chammas R, Schor N, Bellini MH. Endostatin neoadjuvant gene therapy extends survival in an orthotopic metastatic mouse model of renal cell carcinoma [Internet]. Biomedicine & Pharmacotherapy. 2012 ; 66( 4): 237-241.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.biopha.2011.11.002
Vancouver
Braga M de S, Chaves KB, Chammas R, Schor N, Bellini MH. Endostatin neoadjuvant gene therapy extends survival in an orthotopic metastatic mouse model of renal cell carcinoma [Internet]. Biomedicine & Pharmacotherapy. 2012 ; 66( 4): 237-241.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.biopha.2011.11.002
Trabalho de evento-resumo
Galectin-3 influences the oxidative activity of 'CU(ISAEPY) IND. 2' in melanoma cells (2012)
Borges, B. E; Da Silveira, V. C; Zattoni, I. F; Appel, M. H; Borges, M. E; Murasse, M. T; Steclan, C. A; Da Silveira, R. B; Ferreira, Ana Maria da Costa ; Chammas, Roger; Nakao, Lia Sumie
Source: Program and Index. Conference titles: Annual Meeting of the Brazilian Biochemistry and Molecular Biology Society (SBBq). Unidades: IQ, FM
Subjects: MELANOMA, ANTINEOPLÁSICOS
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ABNT
BORGES, B. E et al. Galectin-3 influences the oxidative activity of 'CU(ISAEPY) IND. 2' in melanoma cells. 2012, Anais.. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq), 2012. . Acesso em: 16 nov. 2024.
APA
Borges, B. E., Da Silveira, V. C., Zattoni, I. F., Appel, M. H., Borges, M. E., Murasse, M. T., et al. (2012). Galectin-3 influences the oxidative activity of 'CU(ISAEPY) IND. 2' in melanoma cells. In Program and Index. São Paulo: Sociedade Brasileira de Bioquímica e Biologia Molecular (SBBq).
NLM
Borges BE, Da Silveira VC, Zattoni IF, Appel MH, Borges ME, Murasse MT, Steclan CA, Da Silveira RB, Ferreira AM da C, Chammas R, Nakao LS. Galectin-3 influences the oxidative activity of 'CU(ISAEPY) IND. 2' in melanoma cells. Program and Index. 2012 ;[citado 2024 nov. 16 ]
Vancouver
Borges BE, Da Silveira VC, Zattoni IF, Appel MH, Borges ME, Murasse MT, Steclan CA, Da Silveira RB, Ferreira AM da C, Chammas R, Nakao LS. Galectin-3 influences the oxidative activity of 'CU(ISAEPY) IND. 2' in melanoma cells. Program and Index. 2012 ;[citado 2024 nov. 16 ]
Artigo de periodico
Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA (2012)
Gimenez, Marcela; Marie, Suely Kazue Nagahashi; Oba-Shinjo, Sueli Mieko; Uno, Miyuki; Silva, Roseli da; Laure, Helen Julie; Izumi, Clarice; Otake, Andreia; Chammas, Roger; Rosa, José César
Source: Proteomics. Unidades: FM, FMRP
Subjects: FOSFOPROTEÍNAS, APOPTOSE, NEOPLASIAS CEREBRAIS, BIOQUÍMICA CELULAR
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ABNT
GIMENEZ, Marcela et al. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA. Proteomics, v. 12, n. 17, p. 2632-2640, 2012Tradução . . Disponível em: https://doi.org/10.1002/pmic.201200034. Acesso em: 16 nov. 2024.
APA
Gimenez, M., Marie, S. K. N., Oba-Shinjo, S. M., Uno, M., Silva, R. da, Laure, H. J., et al. (2012). Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA. Proteomics, 12( 17), 2632-2640. doi:10.1002/pmic.201200034
NLM
Gimenez M, Marie SKN, Oba-Shinjo SM, Uno M, Silva R da, Laure HJ, Izumi C, Otake A, Chammas R, Rosa JC. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA [Internet]. Proteomics. 2012 ; 12( 17): 2632-2640.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/pmic.201200034
Vancouver
Gimenez M, Marie SKN, Oba-Shinjo SM, Uno M, Silva R da, Laure HJ, Izumi C, Otake A, Chammas R, Rosa JC. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA [Internet]. Proteomics. 2012 ; 12( 17): 2632-2640.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/pmic.201200034
Artigo de periodico
Endostatin gene therapy stimulates upregulation of ICAM-1 and VCAM-1 in a metastatic renal cell carcinoma model (2012)
Chaves, K. C. B.; Peron, Jean Pierre Schatzmann ; Chammas, R.; Turaça, L. T.; Pesquero, J. B.; Braga, M. S.; Foguer, K.; Schor, N.; Bellini, M. H.
Source: Cancer Gene Therapy. Unidade: FM
Subjects: IMUNOTERAPIA, DNA, CARCINOMA
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ABNT
CHAVES, K. C. B. et al. Endostatin gene therapy stimulates upregulation of ICAM-1 and VCAM-1 in a metastatic renal cell carcinoma model. Cancer Gene Therapy, v. 19, n. 8, p. 558-565, 2012Tradução . . Disponível em: https://doi.org/10.1038/cgt.2012.32. Acesso em: 16 nov. 2024.
APA
Chaves, K. C. B., Peron, J. P. S., Chammas, R., Turaça, L. T., Pesquero, J. B., Braga, M. S., et al. (2012). Endostatin gene therapy stimulates upregulation of ICAM-1 and VCAM-1 in a metastatic renal cell carcinoma model. Cancer Gene Therapy, 19( 8), 558-565. doi:10.1038/cgt.2012.32
NLM
Chaves KCB, Peron JPS, Chammas R, Turaça LT, Pesquero JB, Braga MS, Foguer K, Schor N, Bellini MH. Endostatin gene therapy stimulates upregulation of ICAM-1 and VCAM-1 in a metastatic renal cell carcinoma model [Internet]. Cancer Gene Therapy. 2012 ; 19( 8): 558-565.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/cgt.2012.32
Vancouver
Chaves KCB, Peron JPS, Chammas R, Turaça LT, Pesquero JB, Braga MS, Foguer K, Schor N, Bellini MH. Endostatin gene therapy stimulates upregulation of ICAM-1 and VCAM-1 in a metastatic renal cell carcinoma model [Internet]. Cancer Gene Therapy. 2012 ; 19( 8): 558-565.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/cgt.2012.32

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