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Artigo de periodico
DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide (2022)
Latancia, Marcela Teatin ; Moreno, Natália Cestari ; Leandro, Giovana da Silva ; Ribeiro, Victoria Chaves ; Souza, Izadora de; Vieira, William Kleber Martins; Bastos, André Uchimura ; Hoch, Nicolas Carlos ; Rocha, Clarissa Ribeiro Reily ; Menck, Carlos Frederico Martins
Source: Mutation Research - Genetic Toxicology and Environmental Mutagenesis. Unidades: IQ, ICB, BIOTECNOLOGIA
Subjects: MICROBIOLOGIA, GLIOMA, NEOPLASIAS CEREBRAIS, DNA POLIMERASES, DANO AO DNA, ANTINEOPLÁSICOS, CÉLULAS CULTIVADAS DE TUMOR, MUTAGÊNESE, MELANOMA
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ABNT
LATANCIA, Marcela Teatin et al. DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, v. 878, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.mrgentox.2022.503498. Acesso em: 03 nov. 2024.
APA
Latancia, M. T., Moreno, N. C., Leandro, G. da S., Ribeiro, V. C., Souza, I. de, Vieira, W. K. M., et al. (2022). DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide. Mutation Research - Genetic Toxicology and Environmental Mutagenesis, 878, 1-11. doi:10.1016/j.mrgentox.2022.503498
NLM
Latancia MT, Moreno NC, Leandro G da S, Ribeiro VC, Souza I de, Vieira WKM, Bastos AU, Hoch NC, Rocha CRR, Menck CFM. DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2022 ; 878 1-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.mrgentox.2022.503498
Vancouver
Latancia MT, Moreno NC, Leandro G da S, Ribeiro VC, Souza I de, Vieira WKM, Bastos AU, Hoch NC, Rocha CRR, Menck CFM. DNA polymerase eta protects human cells against DNA damage induced by the tumor chemotherapeutic temozolomide [Internet]. Mutation Research - Genetic Toxicology and Environmental Mutagenesis. 2022 ; 878 1-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.mrgentox.2022.503498
Artigo de periodico
Exoenzyme C3 transferase lowers actin cytoskeleton dynamics, genomic stability and survival of malignant melanoma cells under UV-light stress (2020)
Magalhães, Yuli Thamires; Cardella, Giovanna Duo; Forti, Fabio Luis
Source: Journal of Photochemistry and Photobiology B: Biology. Unidades: IQ, FCF
Subjects: CITOESQUELETO, MELANOMA, RADIAÇÃO ULTRAVIOLETA, DANO AO DNA
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ABNT
MAGALHÃES, Yuli Thamires e CARDELLA, Giovanna Duo e FORTI, Fabio Luis. Exoenzyme C3 transferase lowers actin cytoskeleton dynamics, genomic stability and survival of malignant melanoma cells under UV-light stress. Journal of Photochemistry and Photobiology B: Biology, v. 209, p. 1-9 art. 111947, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.jphotobiol.2020.111947. Acesso em: 03 nov. 2024.
APA
Magalhães, Y. T., Cardella, G. D., & Forti, F. L. (2020). Exoenzyme C3 transferase lowers actin cytoskeleton dynamics, genomic stability and survival of malignant melanoma cells under UV-light stress. Journal of Photochemistry and Photobiology B: Biology, 209, 1-9 art. 111947. doi:10.1016/j.jphotobiol.2020.111947
NLM
Magalhães YT, Cardella GD, Forti FL. Exoenzyme C3 transferase lowers actin cytoskeleton dynamics, genomic stability and survival of malignant melanoma cells under UV-light stress [Internet]. Journal of Photochemistry and Photobiology B: Biology. 2020 ; 209 1-9 art. 111947.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.jphotobiol.2020.111947
Vancouver
Magalhães YT, Cardella GD, Forti FL. Exoenzyme C3 transferase lowers actin cytoskeleton dynamics, genomic stability and survival of malignant melanoma cells under UV-light stress [Internet]. Journal of Photochemistry and Photobiology B: Biology. 2020 ; 209 1-9 art. 111947.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.jphotobiol.2020.111947
Trabalho de evento-resumo periodico
The RhoA GTPase mediate the repair pathways triggered by UV-promoted DNA lesions in mewo cells (2017)
Magalhães, Yuli Thamires; Forti, Fabio Luis
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Workshop on Melanoma Models. Unidade: IQ
Subjects: DANO AO DNA, MELANOMA
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ABNT
MAGALHÃES, Yuli Thamires e FORTI, Fabio Luis. The RhoA GTPase mediate the repair pathways triggered by UV-promoted DNA lesions in mewo cells. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Instituto de Química, Universidade de São Paulo. . Acesso em: 03 nov. 2024. , 2017
APA
Magalhães, Y. T., & Forti, F. L. (2017). The RhoA GTPase mediate the repair pathways triggered by UV-promoted DNA lesions in mewo cells. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Instituto de Química, Universidade de São Paulo.
NLM
Magalhães YT, Forti FL. The RhoA GTPase mediate the repair pathways triggered by UV-promoted DNA lesions in mewo cells. Brazilian Journal of Pharmaceutical Sciences. 2017 ; 53 11 res. WM-037.[citado 2024 nov. 03 ]
Vancouver
Magalhães YT, Forti FL. The RhoA GTPase mediate the repair pathways triggered by UV-promoted DNA lesions in mewo cells. Brazilian Journal of Pharmaceutical Sciences. 2017 ; 53 11 res. WM-037.[citado 2024 nov. 03 ]
Trabalho de evento-resumo
The RhoA and RhoB small GTPases mediate the repair signaling pathways triggered by UV-promoted DNA lesions (2017)
Magalhães, Yuli Thamires; Espinha, Gisele; Forti, Fabio Luis
Source: Abstracts. Conference titles: Brazilian International Congress of Genetics. Unidade: IQ
Subjects: DANO AO DNA, MELANOMA
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ABNT
MAGALHÃES, Yuli Thamires e ESPINHA, Gisele e FORTI, Fabio Luis. The RhoA and RhoB small GTPases mediate the repair signaling pathways triggered by UV-promoted DNA lesions. 2017, Anais.. Ribeirão Preto: Sociedade Brasileira de Genética, 2017. . Acesso em: 03 nov. 2024.
APA
Magalhães, Y. T., Espinha, G., & Forti, F. L. (2017). The RhoA and RhoB small GTPases mediate the repair signaling pathways triggered by UV-promoted DNA lesions. In Abstracts. Ribeirão Preto: Sociedade Brasileira de Genética.
NLM
Magalhães YT, Espinha G, Forti FL. The RhoA and RhoB small GTPases mediate the repair signaling pathways triggered by UV-promoted DNA lesions. Abstracts. 2017 ;[citado 2024 nov. 03 ]
Vancouver
Magalhães YT, Espinha G, Forti FL. The RhoA and RhoB small GTPases mediate the repair signaling pathways triggered by UV-promoted DNA lesions. Abstracts. 2017 ;[citado 2024 nov. 03 ]
Trabalho de evento-resumo
4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model (2016)
Fernandes, Débora Kristina Alves; Faião-Flores, Fernanda; Sandri, Silvana; Oliveira, Erica Aparecida de; Turato, Walter Miguel; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Resumos. Conference titles: Congresso da Sociedade Brasileira de Biologia Celular. Unidade: FCF
Subjects: MELANOMA, DANO AO DNA
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ABNT
FERNANDES, Débora Kristina Alves et al. 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. 2016, Anais.. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC), 2016. . Acesso em: 03 nov. 2024.
APA
Fernandes, D. K. A., Faião-Flores, F., Sandri, S., Oliveira, E. A. de, Turato, W. M., Barros, S. B. de M., & Maria-Engler, S. S. (2016). 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. In Resumos. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC).
NLM
Fernandes DKA, Faião-Flores F, Sandri S, Oliveira EA de, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. Resumos. 2016 ;[citado 2024 nov. 03 ]
Vancouver
Fernandes DKA, Faião-Flores F, Sandri S, Oliveira EA de, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. Resumos. 2016 ;[citado 2024 nov. 03 ]
Trabalho de evento-resumo periodico
4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma (2015)
Alves, Débora Kristina Magalhães; Faião-Flores, Fernanda; Turato, Walter Miguel; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Pharmaceutical Science and Technology Meeting of the Faculty of Pharmaceutical Sciences, University of São Paulo. Unidade: FCF
Subjects: MELANOMA, DANO AO DNA
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ABNT
ALVES, Débora Kristina Magalhães et al. 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo. . Acesso em: 03 nov. 2024. , 2015
APA
Alves, D. K. M., Faião-Flores, F., Turato, W. M., Barros, S. B. de M., & Maria-Engler, S. S. (2015). 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo.
NLM
Alves DKM, Faião-Flores F, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences. 2015 ; 51 44 res. FCF083.[citado 2024 nov. 03 ]
Vancouver
Alves DKM, Faião-Flores F, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences. 2015 ; 51 44 res. FCF083.[citado 2024 nov. 03 ]
Artigo de periodico
DNA damage and repair in leukocytes of melanoma patients exposed in vitro to cisplatin (2011)
Shimabukuro, Fernanda; Neto, Cyro F.; Sanches Jr., José A.; Gattás, Gilka J. F.
Source: Melanoma research. Unidade: FM
Subjects: DANO AO DNA, LEUCÓCITOS, MELANOMA, REPARAÇÃO DO DNA, NEOPLASIAS CUTÂNEAS (INCIDÊNCIA;QUIMIOTERAPIA), QUIMIOTERÁPICOS (USO TERAPÊUTICO)
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ABNT
SHIMABUKURO, Fernanda et al. DNA damage and repair in leukocytes of melanoma patients exposed in vitro to cisplatin. Melanoma research, v. 21, n. 2, p. 99-105, 2011Tradução . . Disponível em: https://doi.org/10.1097/CMR.0b013e3283426839. Acesso em: 03 nov. 2024.
APA
Shimabukuro, F., Neto, C. F., Sanches Jr., J. A., & Gattás, G. J. F. (2011). DNA damage and repair in leukocytes of melanoma patients exposed in vitro to cisplatin. Melanoma research, 21( 2), 99-105. doi:10.1097/CMR.0b013e3283426839
NLM
Shimabukuro F, Neto CF, Sanches Jr. JA, Gattás GJF. DNA damage and repair in leukocytes of melanoma patients exposed in vitro to cisplatin [Internet]. Melanoma research. 2011 ; 21( 2): 99-105.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1097/CMR.0b013e3283426839
Vancouver
Shimabukuro F, Neto CF, Sanches Jr. JA, Gattás GJF. DNA damage and repair in leukocytes of melanoma patients exposed in vitro to cisplatin [Internet]. Melanoma research. 2011 ; 21( 2): 99-105.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1097/CMR.0b013e3283426839
Trabalho de evento-resumo periodico
Polymorphisms in nucleotide excision repair genes modify the risk of cutaneous melanoma (2007)
Francisco, G.; Souza, S. R. P. de; Eluf Neto, José; Chammas, Roger
Source: Clinics. Conference titles: Simpósio avanços em pesquisas médicas. Unidade: FM
Subjects: POLIMORFISMO, NEOPLASIAS CUTÂNEAS (GENÉTICA), MELANOMA, NUCLEOTÍDEOS, DANO AO DNA, RESUMOS (SIMPÓSIOS)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FRANCISCO, G. et al. Polymorphisms in nucleotide excision repair genes modify the risk of cutaneous melanoma. Clinics. São Paulo: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 03 nov. 2024. , 2007
APA
Francisco, G., Souza, S. R. P. de, Eluf Neto, J., & Chammas, R. (2007). Polymorphisms in nucleotide excision repair genes modify the risk of cutaneous melanoma. Clinics. São Paulo: Faculdade de Medicina, Universidade de São Paulo.
NLM
Francisco G, Souza SRP de, Eluf Neto J, Chammas R. Polymorphisms in nucleotide excision repair genes modify the risk of cutaneous melanoma. Clinics. 2007 ; 62 S83.[citado 2024 nov. 03 ]
Vancouver
Francisco G, Souza SRP de, Eluf Neto J, Chammas R. Polymorphisms in nucleotide excision repair genes modify the risk of cutaneous melanoma. Clinics. 2007 ; 62 S83.[citado 2024 nov. 03 ]

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