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Artigo de periodico
Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats (2016)
Moreira, Josimar D; Pernomian, Larissa; Gomes, Mayara Santos; Moreira, Rafael P; Prado, Alejandro F. do; Silva, Carlos Henrique Tomich de Paula da; Oliveira, Ana Maria de
Source: European Journal of Pharmacology. Unidade: FCFRP
Subjects: SISTEMA RENINA-ANGIOTENSINA, ÓXIDO NÍTRICO, DIABETES MELLITUS, ESTRESSE OXIDATIVO
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ABNT
MOREIRA, Josimar D et al. Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats. European Journal of Pharmacology, v. 783, p. 11-22, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2016.04.050. Acesso em: 09 nov. 2024.
APA
Moreira, J. D., Pernomian, L., Gomes, M. S., Moreira, R. P., Prado, A. F. do, Silva, C. H. T. de P. da, & Oliveira, A. M. de. (2016). Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats. European Journal of Pharmacology, 783, 11-22. doi:10.1016/j.ejphar.2016.04.050
NLM
Moreira JD, Pernomian L, Gomes MS, Moreira RP, Prado AF do, Silva CHT de P da, Oliveira AM de. Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats [Internet]. European Journal of Pharmacology. 2016 ; 783 11-22.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2016.04.050
Vancouver
Moreira JD, Pernomian L, Gomes MS, Moreira RP, Prado AF do, Silva CHT de P da, Oliveira AM de. Enhanced nitric oxide generation from nitric oxide synthases as the cause of increased peroxynitrite formation during acute restraint stress: Effects on carotid responsiveness to angiotensinergic stimuli in type-1 diabetic rats [Internet]. European Journal of Pharmacology. 2016 ; 783 11-22.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2016.04.050
Artigo de periodico
Diabetes confers a vasoprotective role to the neurocompensatory response elicited by carotid balloon injury: consequences on contralateral carotid tone and blood flow (2013)
Pernomian, Larissa; Gomes, Mayara Santos; Corrêa, Fernando Morgan de Aguiar; Restini, Carolina Baraldi Araujo; Ramalho, Leandra Naíra Zambelli; Oliveira, Ana Maria de
Source: European Journal of Pharmacology. Unidades: FCFRP, FMRP
Subjects: DIABETES MELLITUS, ANGIOPATIAS DIABÉTICAS, OXIGÊNIO (ESPECIFICAÇÃO)
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ABNT
PERNOMIAN, Larissa et al. Diabetes confers a vasoprotective role to the neurocompensatory response elicited by carotid balloon injury: consequences on contralateral carotid tone and blood flow. European Journal of Pharmacology, v. 708, n. 1-3, p. 124-138, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2013.02.053. Acesso em: 09 nov. 2024.
APA
Pernomian, L., Gomes, M. S., Corrêa, F. M. de A., Restini, C. B. A., Ramalho, L. N. Z., & Oliveira, A. M. de. (2013). Diabetes confers a vasoprotective role to the neurocompensatory response elicited by carotid balloon injury: consequences on contralateral carotid tone and blood flow. European Journal of Pharmacology, 708( 1-3), 124-138. doi:10.1016/j.ejphar.2013.02.053
NLM
Pernomian L, Gomes MS, Corrêa FM de A, Restini CBA, Ramalho LNZ, Oliveira AM de. Diabetes confers a vasoprotective role to the neurocompensatory response elicited by carotid balloon injury: consequences on contralateral carotid tone and blood flow [Internet]. European Journal of Pharmacology. 2013 ; 708( 1-3): 124-138.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2013.02.053
Vancouver
Pernomian L, Gomes MS, Corrêa FM de A, Restini CBA, Ramalho LNZ, Oliveira AM de. Diabetes confers a vasoprotective role to the neurocompensatory response elicited by carotid balloon injury: consequences on contralateral carotid tone and blood flow [Internet]. European Journal of Pharmacology. 2013 ; 708( 1-3): 124-138.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2013.02.053
Artigo de periodico
The role of reactive oxygen species in the modulation of the contraction induced by angiotensin II in carotid artery from diabetic rat (2012)
Pernomian, Larissa; Gomes, Mayara Santos; Restini, Carolina Baraldi Araujo; Ramalho, Leandra Naíra Zambelli; Tirapelli, Carlos Renato ; Oliveira, Ana Maria de
Source: European Journal of Pharmacology. Unidades: FCFRP, FMRP, EERP
Subjects: DIABETES MELLITUS, ARTÉRIAS CARÓTIDAS, ANGIOTENSINA II
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ABNT
PERNOMIAN, Larissa et al. The role of reactive oxygen species in the modulation of the contraction induced by angiotensin II in carotid artery from diabetic rat. European Journal of Pharmacology, v. 678, n. 1-3, p. 15-25, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2011.12.036. Acesso em: 09 nov. 2024.
APA
Pernomian, L., Gomes, M. S., Restini, C. B. A., Ramalho, L. N. Z., Tirapelli, C. R., & Oliveira, A. M. de. (2012). The role of reactive oxygen species in the modulation of the contraction induced by angiotensin II in carotid artery from diabetic rat. European Journal of Pharmacology, 678( 1-3), 15-25. doi:10.1016/j.ejphar.2011.12.036
NLM
Pernomian L, Gomes MS, Restini CBA, Ramalho LNZ, Tirapelli CR, Oliveira AM de. The role of reactive oxygen species in the modulation of the contraction induced by angiotensin II in carotid artery from diabetic rat [Internet]. European Journal of Pharmacology. 2012 ; 678( 1-3): 15-25.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2011.12.036
Vancouver
Pernomian L, Gomes MS, Restini CBA, Ramalho LNZ, Tirapelli CR, Oliveira AM de. The role of reactive oxygen species in the modulation of the contraction induced by angiotensin II in carotid artery from diabetic rat [Internet]. European Journal of Pharmacology. 2012 ; 678( 1-3): 15-25.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2011.12.036
Artigo de periodico
Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats (2008)
Martinez, Marcio L. L.; Rizzi, Elen; Castro, Michele M.; Fernandes, Karla; Bendhack, Lusiane Maria; Gerlach, Raquel Fernanda; Tanus-Santos, José Eduardo
Source: European Journal of Pharmacology. Unidades: FMRP, FCFRP, FORP
Subjects: DIABETES MELLITUS, METALOPROTEINASES, DOENÇAS CARDIOVASCULARES, ESTRESSE OXIDATIVO
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ABNT
MARTINEZ, Marcio L. L. et al. Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats. European Journal of Pharmacology, v. 599, n. 1-3, p. 110-116, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2008.10.007. Acesso em: 09 nov. 2024.
APA
Martinez, M. L. L., Rizzi, E., Castro, M. M., Fernandes, K., Bendhack, L. M., Gerlach, R. F., & Tanus-Santos, J. E. (2008). Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats. European Journal of Pharmacology, 599( 1-3), 110-116. doi:10.1016/j.ejphar.2008.10.007
NLM
Martinez MLL, Rizzi E, Castro MM, Fernandes K, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats [Internet]. European Journal of Pharmacology. 2008 ; 599( 1-3): 110-116.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2008.10.007
Vancouver
Martinez MLL, Rizzi E, Castro MM, Fernandes K, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats [Internet]. European Journal of Pharmacology. 2008 ; 599( 1-3): 110-116.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.ejphar.2008.10.007

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