Source: Frontiers in Neuroanatomy. Unidade: ICB
Subjects: FISIOLOGIA, DOENÇA DE PARKINSON, ATROFIA DE MÚLTIPLOS SISTEMAS, CITOCINAS, DOENÇAS NEURODEGENERATIVAS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, IMUNOHISTOQUÍMICA, ELISA EM ANIMAL
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BIANCHETTI, Maria Eduada Goulart e FERREIRA, Ana Flávia Fernandes e BRITTO, Luiz Roberto Giorgetti de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model. Frontiers in Neuroanatomy, v. 18, p. 12 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fnana.2024.1511951. Acesso em: 08 out. 2025.APA
Bianchetti, M. E. G., Ferreira, A. F. F., & Britto, L. R. G. de. (2024). Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model. Frontiers in Neuroanatomy, 18, 12 . doi:10.3389/fnana.2024.1511951NLM
Bianchetti MEG, Ferreira AFF, Britto LRG de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model [Internet]. Frontiers in Neuroanatomy. 2024 ; 18 12 .[citado 2025 out. 08 ] Available from: https://doi.org/10.3389/fnana.2024.1511951Vancouver
Bianchetti MEG, Ferreira AFF, Britto LRG de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model [Internet]. Frontiers in Neuroanatomy. 2024 ; 18 12 .[citado 2025 out. 08 ] Available from: https://doi.org/10.3389/fnana.2024.1511951