Fonte: Hypertension. Unidade: FMRP
Assuntos: DIABETES MELLITUS, PROTEÍNAS QUINASES, DISFUNÇÃO ERÉTIL, ESTRESSE OXIDATIVO
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ABNT
LOPES, Rhéure Alves Moreira et al. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, v. 68, n. 4, p. 1056-1064, 2016Tradução . . Disponível em: https://doi.org/10.1161/hypertensionaha.116.07518. Acesso em: 06 set. 2024.APA
Lopes, R. A. M., Neves, K. B., Montezano, A. C., Harvey, A., Carneiro, F. S., Touyz, R. M., & Tostes, R. de C. A. (2016). Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, 68( 4), 1056-1064. doi:10.1161/hypertensionaha.116.07518NLM
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 set. 06 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518Vancouver
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 set. 06 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518
