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Artigo de periodico
Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress (2022)
Costa, Rafael Menezes da ; Alves-Lopes, Rhéure ; Alves, Juliano Vilela ; Servian, Carolina P.; Mestriner, Fabiola Leslie Antunes Cardoso; Carneiro, Fernando Silva ; Lobato, Núbia de S.; Tostes, Rita de Cássia Aleixo
Source: Frontiers in Physiology. Unidade: FMRP
Subjects: OBESIDADE, ESTRESSE OXIDATIVO, TESTOSTERONA, DOENÇAS VASCULARES
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ABNT
COSTA, Rafael Menezes da et al. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, v. 13, p. 1-14, 2022Tradução . . Disponível em: https://doi.org/10.3389/fphys.2022.837603. Acesso em: 06 set. 2024.
APA
Costa, R. M. da, Alves-Lopes, R., Alves, J. V., Servian, C. P., Mestriner, F. L. A. C., Carneiro, F. S., et al. (2022). Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress. Frontiers in Physiology, 13, 1-14. doi:10.3389/fphys.2022.837603
NLM
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 set. 06 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Vancouver
Costa RM da, Alves-Lopes R, Alves JV, Servian CP, Mestriner FLAC, Carneiro FS, Lobato N de S, Tostes R de CA. Testosterone contributes to vascular dysfunction in young mice fed a high fat Diet by promoting nuclear factor E2–related factor 2 downregulation and oxidative stress [Internet]. Frontiers in Physiology. 2022 ; 13 1-14.[citado 2024 set. 06 ] Available from: https://doi.org/10.3389/fphys.2022.837603
Artigo de periodico
Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species (2017)
Leite, Letícia N.; Vale, Gabriel T. do; Simplicio, Janaína A.; De Martinis, Bruno Spinosa; Carneiro, Fernando Silva; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: EERP, FFCLRP, FMRP
Subjects: ETANOL, ESTRESSE OXIDATIVO, DISFUNÇÃO ERÉTIL, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEITE, Letícia N. et al. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species. European Journal of Pharmacology, v. 804, p. 82-93, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2017.03.024. Acesso em: 06 set. 2024.
APA
Leite, L. N., Vale, G. T. do, Simplicio, J. A., De Martinis, B. S., Carneiro, F. S., & Tirapelli, C. R. (2017). Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species. European Journal of Pharmacology, 804, 82-93. doi:10.1016/j.ejphar.2017.03.024
NLM
Leite LN, Vale GT do, Simplicio JA, De Martinis BS, Carneiro FS, Tirapelli CR. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species [Internet]. European Journal of Pharmacology. 2017 ; 804 82-93.[citado 2024 set. 06 ] Available from: https://doi.org/10.1016/j.ejphar.2017.03.024
Vancouver
Leite LN, Vale GT do, Simplicio JA, De Martinis BS, Carneiro FS, Tirapelli CR. Ethanol-induced erectile dysfunction and increased expression of pro-inflammatory proteins in the rat cavernosal smooth muscle are mediated by NADPH oxidase-derived reactive oxygen species [Internet]. European Journal of Pharmacology. 2017 ; 804 82-93.[citado 2024 set. 06 ] Available from: https://doi.org/10.1016/j.ejphar.2017.03.024
Artigo de periodico
Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms (2016)
Lopes, Rhéure Alves Moreira; Neves, Karla B.; Montezano, Augusto C.; Harvey, Adam; Carneiro, Fernando Silva; Touyz, Rhian M.; Tostes, Rita de Cássia Aleixo
Source: Hypertension. Unidade: FMRP
Subjects: DIABETES MELLITUS, PROTEÍNAS QUINASES, DISFUNÇÃO ERÉTIL, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Rhéure Alves Moreira et al. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, v. 68, n. 4, p. 1056-1064, 2016Tradução . . Disponível em: https://doi.org/10.1161/hypertensionaha.116.07518. Acesso em: 06 set. 2024.
APA
Lopes, R. A. M., Neves, K. B., Montezano, A. C., Harvey, A., Carneiro, F. S., Touyz, R. M., & Tostes, R. de C. A. (2016). Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms. Hypertension, 68( 4), 1056-1064. doi:10.1161/hypertensionaha.116.07518
NLM
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 set. 06 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518
Vancouver
Lopes RAM, Neves KB, Montezano AC, Harvey A, Carneiro FS, Touyz RM, Tostes R de CA. Internal pudental artery dysfunction in diabetes mellitus is mediated by NOX1-derived ROS-, Nrf2-, and Rho kinase–dependent mechanisms [Internet]. Hypertension. 2016 ; 68( 4): 1056-1064.[citado 2024 set. 06 ] Available from: https://doi.org/10.1161/hypertensionaha.116.07518

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