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Artigo de periodico
An experimental DUAL model of advanced liver damage (2021)
Ubieto, Raquel Benedé ; Vázquez, Olga Estévez; Guo, Feifei; Chen, Chaobo ; Singh, Youvika ; Nakaya, Helder Takashi Imoto ; Moral, Manuel Gómez del ; Paz, Arantza Lamas; Morán, Laura; Alcántara, Nuria López; Reissing, Johanna; Bruns, Tony; Avila, Matías A ; Santamaría, Eva; Mazariegos, Marina S; Woitok, Marius Maximilian; Haas, Ute; Zheng, Kang; Juárez, Ignacio; Villa, José Manuel Martín; Asensio, Iris; Vaquero, Javier ; Peligros, Maria Isabel; Argemi, Josepmaria; Bataller, Ramón ; Ampuero, Javier; Gómez, Manuel Romero ; Trautwein, Christian; Liedtke, Christian ; Bañares, Rafael; Cubero, Francisco Javier ; Nevzorova, Yulia A
Source: Hepatology Communications. Unidade: FCF
Subjects: HEPATOPATIAS, ÁLCOOL, DIETA, FÍGADO
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ABNT
UBIETO, Raquel Benedé et al. An experimental DUAL model of advanced liver damage. Hepatology Communications, v. 5, n. 6, p. 1051-1068, 2021Tradução . . Disponível em: https://doi.org/10.1002/hep4.1698. Acesso em: 22 dez. 2024.
APA
Ubieto, R. B., Vázquez, O. E., Guo, F., Chen, C., Singh, Y., Nakaya, H. T. I., et al. (2021). An experimental DUAL model of advanced liver damage. Hepatology Communications, 5( 6), 1051-1068. doi:10.1002/hep4.1698
NLM
Ubieto RB, Vázquez OE, Guo F, Chen C, Singh Y, Nakaya HTI, Moral MG del, Paz AL, Morán L, Alcántara NL, Reissing J, Bruns T, Avila MA, Santamaría E, Mazariegos MS, Woitok MM, Haas U, Zheng K, Juárez I, Villa JMM, Asensio I, Vaquero J, Peligros MI, Argemi J, Bataller R, Ampuero J, Gómez MR, Trautwein C, Liedtke C, Bañares R, Cubero FJ, Nevzorova YA. An experimental DUAL model of advanced liver damage [Internet]. Hepatology Communications. 2021 ; 5( 6): 1051-1068.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1002/hep4.1698
Vancouver
Ubieto RB, Vázquez OE, Guo F, Chen C, Singh Y, Nakaya HTI, Moral MG del, Paz AL, Morán L, Alcántara NL, Reissing J, Bruns T, Avila MA, Santamaría E, Mazariegos MS, Woitok MM, Haas U, Zheng K, Juárez I, Villa JMM, Asensio I, Vaquero J, Peligros MI, Argemi J, Bataller R, Ampuero J, Gómez MR, Trautwein C, Liedtke C, Bañares R, Cubero FJ, Nevzorova YA. An experimental DUAL model of advanced liver damage [Internet]. Hepatology Communications. 2021 ; 5( 6): 1051-1068.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1002/hep4.1698
Artigo de periodico
Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis (2020)
Romualdo, Guilherme Ribeiro; Silva, Elizangela dos Anjos; Silva, Tereza Cristina da; Aloiav, Thiago Pinheiro Arrais ; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Vinken, Mathieu ; Barbisan, Luis Fernando ; Cogliati, Bruno
Source: Environmental Toxicology. Unidades: FCF, FMVZ
Subjects: HEPATOPATIAS, CARCINOGÊNESE ANIMAL, CHÁ, PLANTAS MEDICINAIS
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ABNT
ROMUALDO, Guilherme Ribeiro et al. Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis. Environmental Toxicology, v. 35, n. 4, p. 518-527, 2020Tradução . . Disponível em: https://doi.org/10.1002/tox.22887. Acesso em: 22 dez. 2024.
APA
Romualdo, G. R., Silva, E. dos A., Silva, T. C. da, Aloiav, T. P. A., Nogueira, M. S., Castro, I. A. de, et al. (2020). Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis. Environmental Toxicology, 35( 4), 518-527. doi:10.1002/tox.22887
NLM
Romualdo GR, Silva E dos A, Silva TC da, Aloiav TPA, Nogueira MS, Castro IA de, Vinken M, Barbisan LF, Cogliati B. Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis [Internet]. Environmental Toxicology. 2020 ; 35( 4): 518-527.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1002/tox.22887
Vancouver
Romualdo GR, Silva E dos A, Silva TC da, Aloiav TPA, Nogueira MS, Castro IA de, Vinken M, Barbisan LF, Cogliati B. Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis [Internet]. Environmental Toxicology. 2020 ; 35( 4): 518-527.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1002/tox.22887
Artigo de periodico
Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model (2018)
Yanguas, Sara Crespo; Silva, Tereza Cristina da; Pereira, Isabel Veloso Alves; Maes, Michaël; Willebrords, Joost; Shestopalov, Valery I; Goes, Bruna Marcela; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Romualdo, Guilherme Ribeiro; Barbisan, Luis Fernando; Gijbels, Eva; Vinken, Mathieu; Cogliati, Bruno
Source: Archives of Toxicology. Unidades: FCF, FMVZ
Subjects: HEPATOPATIAS, BIOMARCADORES, PROTEÍNAS DA MEMBRANA, MODELOS ANIMAIS DE DOENÇAS
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ABNT
YANGUAS, Sara Crespo et al. Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model. Archives of Toxicology, v. 92, n. 8, p. 2607-2627, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00204-018-2255-3. Acesso em: 22 dez. 2024.
APA
Yanguas, S. C., Silva, T. C. da, Pereira, I. V. A., Maes, M., Willebrords, J., Shestopalov, V. I., et al. (2018). Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model. Archives of Toxicology, 92( 8), 2607-2627. doi:10.1007/s00204-018-2255-3
NLM
Yanguas SC, Silva TC da, Pereira IVA, Maes M, Willebrords J, Shestopalov VI, Goes BM, Nogueira MS, Castro IA de, Romualdo GR, Barbisan LF, Gijbels E, Vinken M, Cogliati B. Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model [Internet]. Archives of Toxicology. 2018 ; 92( 8): 2607-2627.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1007/s00204-018-2255-3
Vancouver
Yanguas SC, Silva TC da, Pereira IVA, Maes M, Willebrords J, Shestopalov VI, Goes BM, Nogueira MS, Castro IA de, Romualdo GR, Barbisan LF, Gijbels E, Vinken M, Cogliati B. Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model [Internet]. Archives of Toxicology. 2018 ; 92( 8): 2607-2627.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1007/s00204-018-2255-3
Artigo de periodico
Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease (2018)
Willebrords, Joost; Maes, Michaël; Pereira, Isabel Veloso Alves; Silva, Tereza Cristina da; Govoni, Verônica Mollica; Lopes, Valéria Veras; Yanguas, Sara Crespo; Shestopalov, Valery I; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Farhood, Anwar; Mannaerts, Inge; Van Grunsven, Leo; Akakpo, Jephte; Lebofsky, Margitta; Jaeschke, Hartmut; Cogliati, Bruno ; Vinken, Mathieu
Source: Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. Unidades: FCF, FMVZ
Subjects: HEPATOPATIAS, PROTEÍNAS DA MEMBRANA, PATOGENIA ANIMAL
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ABNT
WILLEBRORDS, Joost et al. Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, v. 1864, n. 3, p. 819-830, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2017.12.013. Acesso em: 22 dez. 2024.
APA
Willebrords, J., Maes, M., Pereira, I. V. A., Silva, T. C. da, Govoni, V. M., Lopes, V. V., et al. (2018). Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1864( 3), 819-830. doi:10.1016/j.bbadis.2017.12.013
NLM
Willebrords J, Maes M, Pereira IVA, Silva TC da, Govoni VM, Lopes VV, Yanguas SC, Shestopalov VI, Nogueira MS, Castro IA de, Farhood A, Mannaerts I, Van Grunsven L, Akakpo J, Lebofsky M, Jaeschke H, Cogliati B, Vinken M. Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2018 ; 1864( 3): 819-830.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.bbadis.2017.12.013
Vancouver
Willebrords J, Maes M, Pereira IVA, Silva TC da, Govoni VM, Lopes VV, Yanguas SC, Shestopalov VI, Nogueira MS, Castro IA de, Farhood A, Mannaerts I, Van Grunsven L, Akakpo J, Lebofsky M, Jaeschke H, Cogliati B, Vinken M. Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2018 ; 1864( 3): 819-830.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.bbadis.2017.12.013
Artigo de periodico
TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice (2018)
Yanguas, Sara Crespo; Silva, Tereza Cristina da; Pereira, Isabel Veloso Alves; Willebrords, Joost; Maes, Michaël; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Leclercq, Isabelle; Romualdo, Guilherme Ribeiro; Barbisan, Luis Fernando; Leybaert, Luc; Cogliati, Bruno ; Vinken, Mathieu
Source: International Journal of Molecular Sciences. Unidades: FMVZ, FCF
Subjects: HEPATOPATIAS, CONEXINAS, FIBROSE CÍSTICA
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ABNT
YANGUAS, Sara Crespo et al. TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice. International Journal of Molecular Sciences, v. 19, n. 3, p. 1-17, 2018Tradução . . Disponível em: https://doi.org/10.3390/ijms19030817. Acesso em: 22 dez. 2024.
APA
Yanguas, S. C., Silva, T. C. da, Pereira, I. V. A., Willebrords, J., Maes, M., Nogueira, M. S., et al. (2018). TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice. International Journal of Molecular Sciences, 19( 3), 1-17. doi:10.3390/ijms19030817
NLM
Yanguas SC, Silva TC da, Pereira IVA, Willebrords J, Maes M, Nogueira MS, Castro IA de, Leclercq I, Romualdo GR, Barbisan LF, Leybaert L, Cogliati B, Vinken M. TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 3): 1-17.[citado 2024 dez. 22 ] Available from: https://doi.org/10.3390/ijms19030817
Vancouver
Yanguas SC, Silva TC da, Pereira IVA, Willebrords J, Maes M, Nogueira MS, Castro IA de, Leclercq I, Romualdo GR, Barbisan LF, Leybaert L, Cogliati B, Vinken M. TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 3): 1-17.[citado 2024 dez. 22 ] Available from: https://doi.org/10.3390/ijms19030817
Artigo de periodico
Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis (2017)
Tiburcio, Taynã Cristina; Willebrords, Joost; Silva, Tereza Cristina da; Pereira, Isabel Veloso Alves; Nogueira, Marina Sayuri; Yanguas, Sara Crespo; Maes, Michaël; Silva, Elisangela dos Anjos; Dagli, Maria Lúcia Zaidan ; Castro, Inar Alves de ; Oliveira, Cláudia Pinto Marques Souza de; Vinken, Mathieu; Cogliati, Bruno
Source: Clinical and Experimental Pharmacology and Physiology. Unidades: FMVZ, FCF, FM
Subjects: CONEXINAS, HEPATOPATIAS, ESTRESSE OXIDATIVO
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ABNT
TIBURCIO, Taynã Cristina et al. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis. Clinical and Experimental Pharmacology and Physiology, v. 44, n. 2, p. 197-206, 2017Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.12701. Acesso em: 22 dez. 2024.
APA
Tiburcio, T. C., Willebrords, J., Silva, T. C. da, Pereira, I. V. A., Nogueira, M. S., Yanguas, S. C., et al. (2017). Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis. Clinical and Experimental Pharmacology and Physiology, 44( 2), 197-206. doi:10.1111/1440-1681.12701
NLM
Tiburcio TC, Willebrords J, Silva TC da, Pereira IVA, Nogueira MS, Yanguas SC, Maes M, Silva E dos A, Dagli MLZ, Castro IA de, Oliveira CPMS de, Vinken M, Cogliati B. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis [Internet]. Clinical and Experimental Pharmacology and Physiology. 2017 ; 44( 2): 197-206.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1111/1440-1681.12701
Vancouver
Tiburcio TC, Willebrords J, Silva TC da, Pereira IVA, Nogueira MS, Yanguas SC, Maes M, Silva E dos A, Dagli MLZ, Castro IA de, Oliveira CPMS de, Vinken M, Cogliati B. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis [Internet]. Clinical and Experimental Pharmacology and Physiology. 2017 ; 44( 2): 197-206.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1111/1440-1681.12701
Artigo de periodico
Pequi (Caryocar brasiliense Camb.) almond oil attenuates carbon tetrachloride-induced acute hepatic injury in rats: Antioxidant and anti-inflammatory effects (2016)
Tôrres, Lucillia Rabelo de Oliveira; Santana, Fernanda Carvalho de; Torres-Leal, Francisco Leonardo; Melo, Illana Louise Pereira de; Yoshime, Luciana Tedesco; Matos Neto, Emidio Marques de; Seelaender, Marília Cerqueira Leite; Araújo, Cintia Maria Monteiro de; Cogliati, Bruno ; Mancini-Filho, Jorge
Source: Food and Chemical Toxicology. Unidades: ICB, FMVZ, FCF
Subjects: ÓLEOS E GORDURAS VEGETAIS COMESTÍVEIS, ANTIOXIDANTES, ANTI-INFLAMATÓRIOS, ÁCIDOS GRAXOS NÃO SATURADOS, ENTEROPATIAS EM ANIMAL, HEPATOPATIAS
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ABNT
TÔRRES, Lucillia Rabelo de Oliveira et al. Pequi (Caryocar brasiliense Camb.) almond oil attenuates carbon tetrachloride-induced acute hepatic injury in rats: Antioxidant and anti-inflammatory effects. Food and Chemical Toxicology, v. 97, p. 205-216, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.fct.2016.09.009. Acesso em: 22 dez. 2024.
APA
Tôrres, L. R. de O., Santana, F. C. de, Torres-Leal, F. L., Melo, I. L. P. de, Yoshime, L. T., Matos Neto, E. M. de, et al. (2016). Pequi (Caryocar brasiliense Camb.) almond oil attenuates carbon tetrachloride-induced acute hepatic injury in rats: Antioxidant and anti-inflammatory effects. Food and Chemical Toxicology, 97, 205-216. doi:10.1016/j.fct.2016.09.009
NLM
Tôrres LR de O, Santana FC de, Torres-Leal FL, Melo ILP de, Yoshime LT, Matos Neto EM de, Seelaender MCL, Araújo CMM de, Cogliati B, Mancini-Filho J. Pequi (Caryocar brasiliense Camb.) almond oil attenuates carbon tetrachloride-induced acute hepatic injury in rats: Antioxidant and anti-inflammatory effects [Internet]. Food and Chemical Toxicology. 2016 ; 97 205-216.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.fct.2016.09.009
Vancouver
Tôrres LR de O, Santana FC de, Torres-Leal FL, Melo ILP de, Yoshime LT, Matos Neto EM de, Seelaender MCL, Araújo CMM de, Cogliati B, Mancini-Filho J. Pequi (Caryocar brasiliense Camb.) almond oil attenuates carbon tetrachloride-induced acute hepatic injury in rats: Antioxidant and anti-inflammatory effects [Internet]. Food and Chemical Toxicology. 2016 ; 97 205-216.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.fct.2016.09.009
Artigo de periodico
Suppressing activity of tributyrin on hepatocarcinogenesis is associated with inhibiting the p53-CRM1 interaction and changing the cellular compartmentalization of p53 protein (2016)
Ortega, Juliana Festa; Conti, Aline de; Tryndyak, Volodymyr; Furtado, kelly Silva; Heidor, Renato ; Horst, Maria Aderuza; Fernandes, Laura Helena Gasparini; Tavares, Paulo Eduardo Latorre Martins; Pogribna, Marta; Shpyleva, Svitlana; Beland, Frederick A; Pogribny, Igor P; Moreno, Fernando Salvador
Source: Oncotarget. Unidade: FCF
Subjects: HEPATOPATIAS, CARCINOMA HEPATOCELULAR
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ABNT
ORTEGA, Juliana Festa et al. Suppressing activity of tributyrin on hepatocarcinogenesis is associated with inhibiting the p53-CRM1 interaction and changing the cellular compartmentalization of p53 protein. Oncotarget, v. 7, n. 17, p. 24339-24347, 2016Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.8248. Acesso em: 22 dez. 2024.
APA
Ortega, J. F., Conti, A. de, Tryndyak, V., Furtado, kelly S., Heidor, R., Horst, M. A., et al. (2016). Suppressing activity of tributyrin on hepatocarcinogenesis is associated with inhibiting the p53-CRM1 interaction and changing the cellular compartmentalization of p53 protein. Oncotarget, 7( 17), 24339-24347. doi:10.18632/oncotarget.8248
NLM
Ortega JF, Conti A de, Tryndyak V, Furtado kelly S, Heidor R, Horst MA, Fernandes LHG, Tavares PELM, Pogribna M, Shpyleva S, Beland FA, Pogribny IP, Moreno FS. Suppressing activity of tributyrin on hepatocarcinogenesis is associated with inhibiting the p53-CRM1 interaction and changing the cellular compartmentalization of p53 protein [Internet]. Oncotarget. 2016 ; 7( 17): 24339-24347.[citado 2024 dez. 22 ] Available from: https://doi.org/10.18632/oncotarget.8248
Vancouver
Ortega JF, Conti A de, Tryndyak V, Furtado kelly S, Heidor R, Horst MA, Fernandes LHG, Tavares PELM, Pogribna M, Shpyleva S, Beland FA, Pogribny IP, Moreno FS. Suppressing activity of tributyrin on hepatocarcinogenesis is associated with inhibiting the p53-CRM1 interaction and changing the cellular compartmentalization of p53 protein [Internet]. Oncotarget. 2016 ; 7( 17): 24339-24347.[citado 2024 dez. 22 ] Available from: https://doi.org/10.18632/oncotarget.8248
Artigo de periodico
Chemopreventive effects of 'beta'-ionone and geraniol during rat hepatocarcinogenesis promotion: distinct actions on cell proliferation, apoptosis, HMGCoA reductase, and RhoA (2011)
Cardozo, Mônica Testoni; Conti, Aline de; Ong, Thomas Prates ; Scolastici, Clarissa; Purgatto, Eduardo ; Horst, Maria Aderuza; Bassoli, Bruna Kempfer; Moreno, Fernando Salvador
Source: Journal of Nutritional Biochemistry. Unidade: FCF
Subjects: HEPATOPATIAS, CARCINOGÊNESE ANIMAL, PROLIFERAÇÃO CELULAR, APOPTOSE
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ABNT
CARDOZO, Mônica Testoni et al. Chemopreventive effects of 'beta'-ionone and geraniol during rat hepatocarcinogenesis promotion: distinct actions on cell proliferation, apoptosis, HMGCoA reductase, and RhoA. Journal of Nutritional Biochemistry, v. 22, n. 2, p. 130-135, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.nutbio.2009.12.007. Acesso em: 22 dez. 2024.
APA
Cardozo, M. T., Conti, A. de, Ong, T. P., Scolastici, C., Purgatto, E., Horst, M. A., et al. (2011). Chemopreventive effects of 'beta'-ionone and geraniol during rat hepatocarcinogenesis promotion: distinct actions on cell proliferation, apoptosis, HMGCoA reductase, and RhoA. Journal of Nutritional Biochemistry, 22( 2), 130-135. doi:10.1016/j.nutbio.2009.12.007
NLM
Cardozo MT, Conti A de, Ong TP, Scolastici C, Purgatto E, Horst MA, Bassoli BK, Moreno FS. Chemopreventive effects of 'beta'-ionone and geraniol during rat hepatocarcinogenesis promotion: distinct actions on cell proliferation, apoptosis, HMGCoA reductase, and RhoA [Internet]. Journal of Nutritional Biochemistry. 2011 ; 22( 2): 130-135.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.nutbio.2009.12.007
Vancouver
Cardozo MT, Conti A de, Ong TP, Scolastici C, Purgatto E, Horst MA, Bassoli BK, Moreno FS. Chemopreventive effects of 'beta'-ionone and geraniol during rat hepatocarcinogenesis promotion: distinct actions on cell proliferation, apoptosis, HMGCoA reductase, and RhoA [Internet]. Journal of Nutritional Biochemistry. 2011 ; 22( 2): 130-135.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1016/j.nutbio.2009.12.007
Artigo de periodico
Use of AST platelet ratio index (APRI Score) as an alternative to liver biopsy for treatment indication in chronic hepatitis C (2009)
Viana, Mônica Salum Valverde Borsoi; Takei, Kioko; Yamaguti, Diva Carvalho Collarile; Guz, Betty; Strauss, Edna
Source: Annals of Hepatology. Unidade: FCF
Subjects: HEPATITE C, HEPATITE CRÔNICA, HEPATOPATIAS
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ABNT
VIANA, Mônica Salum Valverde Borsoi et al. Use of AST platelet ratio index (APRI Score) as an alternative to liver biopsy for treatment indication in chronic hepatitis C. Annals of Hepatology, v. 8, n. 1, p. 26-31, 2009Tradução . . Disponível em: http://www.medigraphic.com/pdfs/hepato/ah-2009/ah091g.pdf. Acesso em: 22 dez. 2024.
APA
Viana, M. S. V. B., Takei, K., Yamaguti, D. C. C., Guz, B., & Strauss, E. (2009). Use of AST platelet ratio index (APRI Score) as an alternative to liver biopsy for treatment indication in chronic hepatitis C. Annals of Hepatology, 8( 1), 26-31. Recuperado de http://www.medigraphic.com/pdfs/hepato/ah-2009/ah091g.pdf
NLM
Viana MSVB, Takei K, Yamaguti DCC, Guz B, Strauss E. Use of AST platelet ratio index (APRI Score) as an alternative to liver biopsy for treatment indication in chronic hepatitis C [Internet]. Annals of Hepatology. 2009 ; 8( 1): 26-31.[citado 2024 dez. 22 ] Available from: http://www.medigraphic.com/pdfs/hepato/ah-2009/ah091g.pdf
Vancouver
Viana MSVB, Takei K, Yamaguti DCC, Guz B, Strauss E. Use of AST platelet ratio index (APRI Score) as an alternative to liver biopsy for treatment indication in chronic hepatitis C [Internet]. Annals of Hepatology. 2009 ; 8( 1): 26-31.[citado 2024 dez. 22 ] Available from: http://www.medigraphic.com/pdfs/hepato/ah-2009/ah091g.pdf
Artigo de periodico
Mass Spectrometric Analysis of a Cyclic 7,8-Butanoguanine Adduct of N-Nitrosopyrrolidine: Comparison to Other N-Nitrosopyrrolidine Adducts in Rat Hepatic DNA (2009)
Loureiro, Ana Paula de Melo ; Zhang, Wenbing; Kassie, Fekadu; Zhang, Siyi; Villalta, Peter W; Wang, Mingyao; Hecht, Stephen S
Source: Chemical Research in Toxicology. Unidade: FCF
Subjects: ESPECTROMETRIA DE MASSAS (ANÁLISE), HEPATOPATIAS
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ABNT
LOUREIRO, Ana Paula de Melo et al. Mass Spectrometric Analysis of a Cyclic 7,8-Butanoguanine Adduct of N-Nitrosopyrrolidine: Comparison to Other N-Nitrosopyrrolidine Adducts in Rat Hepatic DNA. Chemical Research in Toxicology, v. 22, n. 10, p. 1728-1735, 2009Tradução . . Disponível em: https://doi.org/10.1021/tx900238t. Acesso em: 22 dez. 2024.
APA
Loureiro, A. P. de M., Zhang, W., Kassie, F., Zhang, S., Villalta, P. W., Wang, M., & Hecht, S. S. (2009). Mass Spectrometric Analysis of a Cyclic 7,8-Butanoguanine Adduct of N-Nitrosopyrrolidine: Comparison to Other N-Nitrosopyrrolidine Adducts in Rat Hepatic DNA. Chemical Research in Toxicology, 22( 10), 1728-1735. doi:10.1021/tx900238t
NLM
Loureiro AP de M, Zhang W, Kassie F, Zhang S, Villalta PW, Wang M, Hecht SS. Mass Spectrometric Analysis of a Cyclic 7,8-Butanoguanine Adduct of N-Nitrosopyrrolidine: Comparison to Other N-Nitrosopyrrolidine Adducts in Rat Hepatic DNA [Internet]. Chemical Research in Toxicology. 2009 ; 22( 10): 1728-1735.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1021/tx900238t
Vancouver
Loureiro AP de M, Zhang W, Kassie F, Zhang S, Villalta PW, Wang M, Hecht SS. Mass Spectrometric Analysis of a Cyclic 7,8-Butanoguanine Adduct of N-Nitrosopyrrolidine: Comparison to Other N-Nitrosopyrrolidine Adducts in Rat Hepatic DNA [Internet]. Chemical Research in Toxicology. 2009 ; 22( 10): 1728-1735.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1021/tx900238t
Trabalho de evento-resumo
Mass spectrometric analysis of a cyclic 7,8-guanine adduct of the carcinogen N-nitrosopyrrolidine (2009)
Loureiro, Ana Paula de Melo ; Zhang, Wenbing; Villalta, Peter W; Wang, Mingyao; Hecht, Stephen S
Source: Abstracts. Conference titles: Annual Meeting of the American Association for Cancer Research (AACR). Unidade: FCF
Subjects: ESPECTROMETRIA DE MASSAS (ANÁLISE), HEPATOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOUREIRO, Ana Paula de Melo et al. Mass spectrometric analysis of a cyclic 7,8-guanine adduct of the carcinogen N-nitrosopyrrolidine. 2009, Anais.. Philadelphia: American Association for Cancer Research, 2009. . Acesso em: 22 dez. 2024.
APA
Loureiro, A. P. de M., Zhang, W., Villalta, P. W., Wang, M., & Hecht, S. S. (2009). Mass spectrometric analysis of a cyclic 7,8-guanine adduct of the carcinogen N-nitrosopyrrolidine. In Abstracts. Philadelphia: American Association for Cancer Research.
NLM
Loureiro AP de M, Zhang W, Villalta PW, Wang M, Hecht SS. Mass spectrometric analysis of a cyclic 7,8-guanine adduct of the carcinogen N-nitrosopyrrolidine. Abstracts. 2009 ;[citado 2024 dez. 22 ]
Vancouver
Loureiro AP de M, Zhang W, Villalta PW, Wang M, Hecht SS. Mass spectrometric analysis of a cyclic 7,8-guanine adduct of the carcinogen N-nitrosopyrrolidine. Abstracts. 2009 ;[citado 2024 dez. 22 ]
Trabalho de evento-resumo periodico
5-Azaditidina (5AzC) tem ação iniciante da hepatocarcinogênese em ratos (2006)
Conti, Aline de; Kuroiwa-Trzmielina, Joice; Ong, Thomas Prates ; Heidor, Renato ; Pereira, Douglas; Moreno, Fernando Salvador
Source: Revista Brasileira de Ciências Farmacêuticas. Conference titles: Semana Farmacêutica de Ciência e Tecnologia da FCF-USP. Unidade: FCF
Subjects: HEPATOPATIAS, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CONTI, Aline de et al. 5-Azaditidina (5AzC) tem ação iniciante da hepatocarcinogênese em ratos. Revista Brasileira de Ciências Farmacêuticas. São Paulo: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo. . Acesso em: 22 dez. 2024. , 2006
APA
Conti, A. de, Kuroiwa-Trzmielina, J., Ong, T. P., Heidor, R., Pereira, D., & Moreno, F. S. (2006). 5-Azaditidina (5AzC) tem ação iniciante da hepatocarcinogênese em ratos. Revista Brasileira de Ciências Farmacêuticas. São Paulo: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo.
NLM
Conti A de, Kuroiwa-Trzmielina J, Ong TP, Heidor R, Pereira D, Moreno FS. 5-Azaditidina (5AzC) tem ação iniciante da hepatocarcinogênese em ratos. Revista Brasileira de Ciências Farmacêuticas. 2006 ; 42 75 res.FCF145.[citado 2024 dez. 22 ]
Vancouver
Conti A de, Kuroiwa-Trzmielina J, Ong TP, Heidor R, Pereira D, Moreno FS. 5-Azaditidina (5AzC) tem ação iniciante da hepatocarcinogênese em ratos. Revista Brasileira de Ciências Farmacêuticas. 2006 ; 42 75 res.FCF145.[citado 2024 dez. 22 ]
Texto na web
Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase (2005)
Ong, Thomas Prates ; Heidor, Renato ; Conti, Aline de; Dagli, Maria Lúcia Zaidan ; Moreno, Fernando Salvador
Source: Carcinogenesis. Unidades: FMVZ, FCF
Subjects: CARCINOGÊNESE ANIMAL, HEPATOPATIAS, QUÍMICA ORGÂNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ONG, Thomas Prates et al. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. Oxford: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo. . Acesso em: 22 dez. 2024. , 2005
APA
Ong, T. P., Heidor, R., Conti, A. de, Dagli, M. L. Z., & Moreno, F. S. (2005). Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. Oxford: Faculdade de Medicina Veterinária e Zootecnia, Universidade de São Paulo.
NLM
Ong TP, Heidor R, Conti A de, Dagli MLZ, Moreno FS. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. 2005 ; 1-43.[citado 2024 dez. 22 ]
Vancouver
Ong TP, Heidor R, Conti A de, Dagli MLZ, Moreno FS. Farnesol and geraniol chemopreventive activities during the initial phase of hepatocarcinogenesis involve similar actions on cell profliferation and DNA damage, but distinct actions on apoptosis, plasma cholesterol and HMGCoA reductase. Carcinogenesis. 2005 ; 1-43.[citado 2024 dez. 22 ]
Trabalho de evento-resumo periodico
Estresse oxidativo na doença gordurosa não alcoólica do fígado (NAFLD) em pacientes portadores de obesidade mórbida (2003)
Oliveira, C. P. M. S.; Rascovski, A.; Furuya Junior, C. K.; Bastos, M. S.; Della Nina, B. I.; Yuahasi, K.; Faintuch, Joel; Abdalla, Dulcinéia Saes Parra ; Matsuda, M.; Zilberstein, Bruno; Gayotto, Luiz Carlos da Costa; Gama Rodrigues, Joaquim José; Halpern, Alfredo; Carrilho, Flair Jose
Source: GED: Gastroenterologia Endoscopia Digestiva. Unidades: FM, FCF
Subjects: FÍGADO GORDUROSO, OBESIDADE MORBIDA (PATOLOGIA), HEPATOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, C. P. M. S. et al. Estresse oxidativo na doença gordurosa não alcoólica do fígado (NAFLD) em pacientes portadores de obesidade mórbida. GED: Gastroenterologia Endoscopia Digestiva. São Paulo: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 22 dez. 2024. , 2003
APA
Oliveira, C. P. M. S., Rascovski, A., Furuya Junior, C. K., Bastos, M. S., Della Nina, B. I., Yuahasi, K., et al. (2003). Estresse oxidativo na doença gordurosa não alcoólica do fígado (NAFLD) em pacientes portadores de obesidade mórbida. GED: Gastroenterologia Endoscopia Digestiva. São Paulo: Faculdade de Medicina, Universidade de São Paulo.
NLM
Oliveira CPMS, Rascovski A, Furuya Junior CK, Bastos MS, Della Nina BI, Yuahasi K, Faintuch J, Abdalla DSP, Matsuda M, Zilberstein B, Gayotto LC da C, Gama Rodrigues JJ, Halpern A, Carrilho FJ. Estresse oxidativo na doença gordurosa não alcoólica do fígado (NAFLD) em pacientes portadores de obesidade mórbida. GED: Gastroenterologia Endoscopia Digestiva. 2003 ; 22 S16.[citado 2024 dez. 22 ]
Vancouver
Oliveira CPMS, Rascovski A, Furuya Junior CK, Bastos MS, Della Nina BI, Yuahasi K, Faintuch J, Abdalla DSP, Matsuda M, Zilberstein B, Gayotto LC da C, Gama Rodrigues JJ, Halpern A, Carrilho FJ. Estresse oxidativo na doença gordurosa não alcoólica do fígado (NAFLD) em pacientes portadores de obesidade mórbida. GED: Gastroenterologia Endoscopia Digestiva. 2003 ; 22 S16.[citado 2024 dez. 22 ]
Artigo de periodico
Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis (1998)
Dagli, Maria Lúcia Zaidan ; Guerra, José Luiz; Sinhorini, Idercio Luiz; Wu, Ton-Sen; Rizzi, Maria Beatriz de Souza Lima; Penteado, Marilene de Vuono Camargo; Moreno, Fernando Salvador
Source: Pathology. Unidades: FMVZ, FCF
Subjects: CARCINOGÊNESE ANIMAL, CAROTENOIDES, HEPATOPATIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAGLI, Maria Lúcia Zaidan et al. Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis. Pathology, v. 30, n. 3, p. 259-266, 1998Tradução . . Disponível em: https://doi.org/10.1080/00313029800169416. Acesso em: 22 dez. 2024.
APA
Dagli, M. L. Z., Guerra, J. L., Sinhorini, I. L., Wu, T. -S., Rizzi, M. B. de S. L., Penteado, M. de V. C., & Moreno, F. S. (1998). Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis. Pathology, 30( 3), 259-266. doi:10.1080/00313029800169416
NLM
Dagli MLZ, Guerra JL, Sinhorini IL, Wu T-S, Rizzi MB de SL, Penteado M de VC, Moreno FS. Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis [Internet]. Pathology. 1998 ; 30( 3): 259-266.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1080/00313029800169416
Vancouver
Dagli MLZ, Guerra JL, Sinhorini IL, Wu T-S, Rizzi MB de SL, Penteado M de VC, Moreno FS. Beta-carotene reduces the ductular cell reaction in the liver of Wistar rats submitted to the resistant hepatocyte model of carcinogenesis [Internet]. Pathology. 1998 ; 30( 3): 259-266.[citado 2024 dez. 22 ] Available from: https://doi.org/10.1080/00313029800169416

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