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Artigo de periodico
T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion (2015)
Souza, Paula Bargi de; Romano, Renata Marino; Silva, Francemilson Goulart da ; Brunetto, Erika Lia; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, GLICOPROTEINAS, GLÂNDULA PITUITÁRIA, RATOS, SECREÇÃO ANIMAL
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ABNT
SOUZA, Paula Bargi de et al. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, v. 409, p. 73-81, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.04.002. Acesso em: 16 set. 2024.
APA
Souza, P. B. de, Romano, R. M., Silva, F. G. da, Brunetto, E. L., & Nunes, M. T. (2015). T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, 409, 73-81. doi:10.1016/j.mce.2015.04.002
NLM
Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
Vancouver
Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
Artigo de periodico
Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders (2015)
Lino, Caroline Antunes; Silva, Ivson Bezerra da; Shibata, Caroline Emy Rodrigues; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, RATOS WISTAR, DOENÇAS CARDIOVASCULARES EM ANIMAL, SISTEMA RENINA-ANGIOTENSINA
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ABNT
LINO, Caroline Antunes et al. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, v. 416, p. 1-8, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.08.015. Acesso em: 16 set. 2024.
APA
Lino, C. A., Silva, I. B. da, Shibata, C. E. R., & Barreto-Chaves, M. L. M. (2015). Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders. Molecular and Cellular Endocrinology, 416, 1-8. doi:10.1016/j.mce.2015.08.015
NLM
Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
Vancouver
Lino CA, Silva IB da, Shibata CER, Barreto-Chaves MLM. Maternal hyperthyroidism increases the susceptibility of rat adult offspring to cardiovascular disorders [Internet]. Molecular and Cellular Endocrinology. 2015 ; 416 1-8.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2015.08.015
Artigo de periodico
N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex (2013)
Mendonça, Pedro Omori Ribeiro de; Liria, Cleber Wanderlei; Machini, Maria Teresa ; Lotfi, Claudimara Ferini Pacicco
Source: Molecular and Cellular Endocrinology. Unidades: IQ, ICB
Subjects: CÓRTEX ADRENAL, HORMÔNIO ADRENOCORTICOTRÓFICO
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ABNT
MENDONÇA, Pedro Omori Ribeiro de et al. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex. Molecular and Cellular Endocrinology, v. 37, n. 1-2, p. 166-173, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2012.11.017. Acesso em: 16 set. 2024.
APA
Mendonça, P. O. R. de, Liria, C. W., Machini, M. T., & Lotfi, C. F. P. (2013). N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex. Molecular and Cellular Endocrinology, 37( 1-2), 166-173. doi:10.1016/j.mce.2012.11.017
NLM
Mendonça POR de, Liria CW, Machini MT, Lotfi CFP. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex [Internet]. Molecular and Cellular Endocrinology. 2013 ; 37( 1-2): 166-173.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2012.11.017
Vancouver
Mendonça POR de, Liria CW, Machini MT, Lotfi CFP. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex [Internet]. Molecular and Cellular Endocrinology. 2013 ; 37( 1-2): 166-173.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2012.11.017
Artigo de periodico
AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth (2013)
Takano, Ana Paula Cremasco ; Diniz, Gabriela Placoná ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
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ABNT
TAKANO, Ana Paula Cremasco e DINIZ, Gabriela Placoná e BARRETO-CHAVES, Maria Luiza Morais. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, v. 376, n. 1-2, p. 43-50, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.05.024. Acesso em: 16 set. 2024.
APA
Takano, A. P. C., Diniz, G. P., & Barreto-Chaves, M. L. M. (2013). AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, 376( 1-2), 43-50. doi:10.1016/j.mce.2013.05.024
NLM
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Vancouver
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Artigo de periodico
Identification of nuclear factor-κB sites in the Slc2a4 gene promoter (2013)
Furuya, Daniela Tomie; Neri, Elida Adalgisa; Poletto, Ana Claudia; Anhê, G. F.; Freitas, H. S.; Campello, Raquel Saldanha; Rebouças, Nancy Amaral; Machado, Ubiratan Fabres
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FURUYA, Daniela Tomie et al. Identification of nuclear factor-κB sites in the Slc2a4 gene promoter. Molecular and Cellular Endocrinology, v. 370, n. 1-2, p. 87-95, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.01.019. Acesso em: 16 set. 2024.
APA
Furuya, D. T., Neri, E. A., Poletto, A. C., Anhê, G. F., Freitas, H. S., Campello, R. S., et al. (2013). Identification of nuclear factor-κB sites in the Slc2a4 gene promoter. Molecular and Cellular Endocrinology, 370( 1-2), 87-95. doi:10.1016/j.mce.2013.01.019
NLM
Furuya DT, Neri EA, Poletto AC, Anhê GF, Freitas HS, Campello RS, Rebouças NA, Machado UF. Identification of nuclear factor-κB sites in the Slc2a4 gene promoter [Internet]. Molecular and Cellular Endocrinology. 2013 ; 370( 1-2): 87-95.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2013.01.019
Vancouver
Furuya DT, Neri EA, Poletto AC, Anhê GF, Freitas HS, Campello RS, Rebouças NA, Machado UF. Identification of nuclear factor-κB sites in the Slc2a4 gene promoter [Internet]. Molecular and Cellular Endocrinology. 2013 ; 370( 1-2): 87-95.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2013.01.019
Artigo de periodico
miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation (2013)
Diniz, Gabriela Placoná ; Takano, Ana Paula Cremasco ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná e TAKANO, Ana Paula Cremasco e BARRETO-CHAVES, Maria Luiza Morais. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, v. 374, n. 1-2, p. 117-124, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.04.010. Acesso em: 16 set. 2024.
APA
Diniz, G. P., Takano, A. P. C., & Barreto-Chaves, M. L. M. (2013). miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, 374( 1-2), 117-124. doi:10.1016/j.mce.2013.04.010
NLM
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
Vancouver
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
Artigo de periodico
Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms (2012)
Calil-Silveira, Jamile; Serrano-Nascimento, Caroline; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CALIL-SILVEIRA, Jamile e SERRANO-NASCIMENTO, Caroline e NUNES, Maria Tereza. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms. Molecular and Cellular Endocrinology, v. 350, n. 1, p. 118-124, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2011.12.002. Acesso em: 16 set. 2024.
APA
Calil-Silveira, J., Serrano-Nascimento, C., & Nunes, M. T. (2012). Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms. Molecular and Cellular Endocrinology, 350( 1), 118-124. doi:10.1016/j.mce.2011.12.002
NLM
Calil-Silveira J, Serrano-Nascimento C, Nunes MT. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms [Internet]. Molecular and Cellular Endocrinology. 2012 ; 350( 1): 118-124.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2011.12.002
Vancouver
Calil-Silveira J, Serrano-Nascimento C, Nunes MT. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms [Internet]. Molecular and Cellular Endocrinology. 2012 ; 350( 1): 118-124.[citado 2024 set. 16 ] Available from: https://doi.org/10.1016/j.mce.2011.12.002

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