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Artigo de periodico
Vascular activation of K+ channels and Na+-K+ ATPase activity of estrogen-deficient female rats (2017)
Ribeiro Junior, Rogério Faustino; Fiorim, Jonaina; Marques, Vinicius Bermond; Ronconi, Karoline de Sousa; Botelho, Tatiani; Grando, Marcella Daruge; Bendhack, Lusiane Maria; Vassallo, Dalton Valentim; Stefanon, Ivanita
Source: Vascular Pharmacology. Unidade: FCFRP
Subjects: ESTRÓGENOS, ÓXIDO NÍTRICO, ESTRESSE OXIDATIVO, CANAIS DE POTÁSSIO
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ABNT
RIBEIRO JUNIOR, Rogério Faustino et al. Vascular activation of K+ channels and Na+-K+ ATPase activity of estrogen-deficient female rats. Vascular Pharmacology, v. 99, p. 23-33, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2017.09.003. Acesso em: 17 out. 2024.
APA
Ribeiro Junior, R. F., Fiorim, J., Marques, V. B., Ronconi, K. de S., Botelho, T., Grando, M. D., et al. (2017). Vascular activation of K+ channels and Na+-K+ ATPase activity of estrogen-deficient female rats. Vascular Pharmacology, 99, 23-33. doi:10.1016/j.vph.2017.09.003
NLM
Ribeiro Junior RF, Fiorim J, Marques VB, Ronconi K de S, Botelho T, Grando MD, Bendhack LM, Vassallo DV, Stefanon I. Vascular activation of K+ channels and Na+-K+ ATPase activity of estrogen-deficient female rats [Internet]. Vascular Pharmacology. 2017 ; 99 23-33.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2017.09.003
Vancouver
Ribeiro Junior RF, Fiorim J, Marques VB, Ronconi K de S, Botelho T, Grando MD, Bendhack LM, Vassallo DV, Stefanon I. Vascular activation of K+ channels and Na+-K+ ATPase activity of estrogen-deficient female rats [Internet]. Vascular Pharmacology. 2017 ; 99 23-33.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2017.09.003
Artigo de periodico
Blood pressure variability provokes vascular β-adrenoceptor desensitization in rats (2016)
Rocha, Matheus L.; Silva, Bruno R.; Lunardi, Claure Nain; Ramalho, Leandra Naíra Zambelli; Bendhack, Lusiane Maria
Source: Vascular Pharmacology. Unidades: FMRP, FCFRP
Subjects: PRESSÃO SANGUÍNEA, ADRENORECEPTORES, DESSENSIBILIZAÇÃO, DENERVAÇÃO
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ABNT
ROCHA, Matheus L. et al. Blood pressure variability provokes vascular β-adrenoceptor desensitization in rats. Vascular Pharmacology, v. 82, p. 82-89, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.05.007. Acesso em: 17 out. 2024.
APA
Rocha, M. L., Silva, B. R., Lunardi, C. N., Ramalho, L. N. Z., & Bendhack, L. M. (2016). Blood pressure variability provokes vascular β-adrenoceptor desensitization in rats. Vascular Pharmacology, 82, 82-89. doi:10.1016/j.vph.2016.05.007
NLM
Rocha ML, Silva BR, Lunardi CN, Ramalho LNZ, Bendhack LM. Blood pressure variability provokes vascular β-adrenoceptor desensitization in rats [Internet]. Vascular Pharmacology. 2016 ; 82 82-89.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2016.05.007
Vancouver
Rocha ML, Silva BR, Lunardi CN, Ramalho LNZ, Bendhack LM. Blood pressure variability provokes vascular β-adrenoceptor desensitization in rats [Internet]. Vascular Pharmacology. 2016 ; 82 82-89.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2016.05.007
Artigo de periodico
Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR (2016)
Perassa, Ligia A.; Graton, Murilo E.; Potje, Simone R.; Troiano, Jéssica A.; Lima, Mariana S.; Vale, Gabriel T.; Pereira, Ariana A.F.; Nakamune, Ana Claúdia M.S.; Sumida, Doris H.; Tirapelli, Carlos Renato ; Bendhack, Lusiane Maria; Antoniali, Cristina
Source: Vascular Pharmacology. Unidades: EERP, FCFRP
Subjects: ENDOTÉLIO VASCULAR, ÓXIDO NÍTRICO, PRESSÃO SANGUÍNEA, COMPOSTOS ORGÂNICOS, FARMACOLOGIA
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ABNT
PERASSA, Ligia A. et al. Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR. Vascular Pharmacology, v. 87, p. 38-48, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2016.06.005. Acesso em: 17 out. 2024.
APA
Perassa, L. A., Graton, M. E., Potje, S. R., Troiano, J. A., Lima, M. S., Vale, G. T., et al. (2016). Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR. Vascular Pharmacology, 87, 38-48. doi:10.1016/j.vph.2016.06.005
NLM
Perassa LA, Graton ME, Potje SR, Troiano JA, Lima MS, Vale GT, Pereira AAF, Nakamune ACMS, Sumida DH, Tirapelli CR, Bendhack LM, Antoniali C. Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR [Internet]. Vascular Pharmacology. 2016 ; 87 38-48.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2016.06.005
Vancouver
Perassa LA, Graton ME, Potje SR, Troiano JA, Lima MS, Vale GT, Pereira AAF, Nakamune ACMS, Sumida DH, Tirapelli CR, Bendhack LM, Antoniali C. Apocynin reduces blood pressure and restores the proper function of vascular endothelium in SHR [Internet]. Vascular Pharmacology. 2016 ; 87 38-48.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2016.06.005
Artigo de periodico
Ethanol induces vascular relaxation via redox-sensitive and nitric oxide-dependent pathways (2012)
Rocha, Juliana T.; Hipólito, Ulisses V.; Callera, Glaucia E.; Yogi, Alvaro; Neto Filho, Mario dos Anjos; Bendhack, Lusiane Maria; Touyz, Rhian M.; Tirapelli, Carlos Renato
Source: Vascular Pharmacology. Unidades: EERP, FCFRP
Subjects: ÓXIDO NÍTRICO, ETANOL, AORTA, VASODILATAÇÃO
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ABNT
ROCHA, Juliana T. et al. Ethanol induces vascular relaxation via redox-sensitive and nitric oxide-dependent pathways. Vascular Pharmacology, v. 56, n. 1-2, p. 74-83, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2011.11.006. Acesso em: 17 out. 2024.
APA
Rocha, J. T., Hipólito, U. V., Callera, G. E., Yogi, A., Neto Filho, M. dos A., Bendhack, L. M., et al. (2012). Ethanol induces vascular relaxation via redox-sensitive and nitric oxide-dependent pathways. Vascular Pharmacology, 56( 1-2), 74-83. doi:10.1016/j.vph.2011.11.006
NLM
Rocha JT, Hipólito UV, Callera GE, Yogi A, Neto Filho M dos A, Bendhack LM, Touyz RM, Tirapelli CR. Ethanol induces vascular relaxation via redox-sensitive and nitric oxide-dependent pathways [Internet]. Vascular Pharmacology. 2012 ; 56( 1-2): 74-83.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2011.11.006
Vancouver
Rocha JT, Hipólito UV, Callera GE, Yogi A, Neto Filho M dos A, Bendhack LM, Touyz RM, Tirapelli CR. Ethanol induces vascular relaxation via redox-sensitive and nitric oxide-dependent pathways [Internet]. Vascular Pharmacology. 2012 ; 56( 1-2): 74-83.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2011.11.006
Artigo de periodico
Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells (2009)
Oliveira, Ana P. S.; Lunardi, Claure N.; Rodrigues, Gerson J.; Bendhack, Lusiane M.
Source: Vascular Pharmacology. Unidade: FCFRP
Subjects: VASODILATAÇÃO, ÓXIDO NÍTRICO, CÉLULAS ENDOTELIAIS
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ABNT
OLIVEIRA, Ana P. S. et al. Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells. Vascular Pharmacology, v. 50, n. 5-6, p. 153-159, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2008.11.010. Acesso em: 17 out. 2024.
APA
Oliveira, A. P. S., Lunardi, C. N., Rodrigues, G. J., & Bendhack, L. M. (2009). Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells. Vascular Pharmacology, 50( 5-6), 153-159. doi:10.1016/j.vph.2008.11.010
NLM
Oliveira APS, Lunardi CN, Rodrigues GJ, Bendhack LM. Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells [Internet]. Vascular Pharmacology. 2009 ; 50( 5-6): 153-159.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2008.11.010
Vancouver
Oliveira APS, Lunardi CN, Rodrigues GJ, Bendhack LM. Relaxation induced by calcium ionophore is impaired in carotid arteries from 2K-1C rats due to failed effect of nitric oxide on the smooth muscle cells [Internet]. Vascular Pharmacology. 2009 ; 50( 5-6): 153-159.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2008.11.010
Artigo de periodico
Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel (2009)
Rocha, Matheus L.; Bendhack, Lusiane Maria
Source: Vascular Pharmacology. Unidade: FCFRP
Subjects: ENDOTÉLIO, AORTA, RETÍCULO SARCOPLÁSMICO
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ABNT
ROCHA, Matheus L. e BENDHACK, Lusiane Maria. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology, v. 50, n. 3-4, p. 98-103, 2009Tradução . . Acesso em: 17 out. 2024.
APA
Rocha, M. L., & Bendhack, L. M. (2009). Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology, 50( 3-4), 98-103.
NLM
Rocha ML, Bendhack LM. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology. 2009 ; 50( 3-4): 98-103.[citado 2024 out. 17 ]
Vancouver
Rocha ML, Bendhack LM. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology. 2009 ; 50( 3-4): 98-103.[citado 2024 out. 17 ]
Artigo de periodico
Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway (2007)
Lunardi, Claure N.; Vercesi, Juliana Aparecida; Silva, Roberto Santana da; Bendhack, Lusiane Maria
Source: Vascular Pharmacology. Unidade: FCFRP
Subjects: VASODILATAÇÃO, RUTÊNIO, ÓXIDO NÍTRICO
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ABNT
LUNARDI, Claure N. et al. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology, v. 47, n. 2-3, p. 139-144, 2007Tradução . . Acesso em: 17 out. 2024.
APA
Lunardi, C. N., Vercesi, J. A., Silva, R. S. da, & Bendhack, L. M. (2007). Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology, 47( 2-3), 139-144.
NLM
Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology. 2007 ; 47( 2-3): 139-144.[citado 2024 out. 17 ]
Vancouver
Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology. 2007 ; 47( 2-3): 139-144.[citado 2024 out. 17 ]
Artigo de periodico
Comparison of the mechanisms underlying the relaxation induced by two nitric oxide donors: sodium nitroprusside and a new ruthenium complex (2007)
Bonaventura, Daniella; Lima, Renata Galvão de; Vercesi, Juliana A.; Silva, Roberto Santana da; Bendhack, Lusiane Maria
Source: Vascular Pharmacology. Unidade: FCFRP
Assunto: FARMACOLOGIA
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ABNT
BONAVENTURA, Daniella et al. Comparison of the mechanisms underlying the relaxation induced by two nitric oxide donors: sodium nitroprusside and a new ruthenium complex. Vascular Pharmacology, v. 46, n. 3, p. 215-222, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2006.10.002. Acesso em: 17 out. 2024.
APA
Bonaventura, D., Lima, R. G. de, Vercesi, J. A., Silva, R. S. da, & Bendhack, L. M. (2007). Comparison of the mechanisms underlying the relaxation induced by two nitric oxide donors: sodium nitroprusside and a new ruthenium complex. Vascular Pharmacology, 46( 3), 215-222. doi:10.1016/j.vph.2006.10.002
NLM
Bonaventura D, Lima RG de, Vercesi JA, Silva RS da, Bendhack LM. Comparison of the mechanisms underlying the relaxation induced by two nitric oxide donors: sodium nitroprusside and a new ruthenium complex [Internet]. Vascular Pharmacology. 2007 ; 46( 3): 215-222.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2006.10.002
Vancouver
Bonaventura D, Lima RG de, Vercesi JA, Silva RS da, Bendhack LM. Comparison of the mechanisms underlying the relaxation induced by two nitric oxide donors: sodium nitroprusside and a new ruthenium complex [Internet]. Vascular Pharmacology. 2007 ; 46( 3): 215-222.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2006.10.002
Artigo de periodico
Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation (2005)
Rascado, Ricardo Radighieri; Bendhack, Lusiane Maria
Source: Vascular Pharmacology. Unidade: FCFRP
Assunto: FARMACOLOGIA
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ABNT
RASCADO, Ricardo Radighieri e BENDHACK, Lusiane Maria. Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology, v. 42, p. 63-68, 2005Tradução . . Acesso em: 17 out. 2024.
APA
Rascado, R. R., & Bendhack, L. M. (2005). Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology, 42, 63-68.
NLM
Rascado RR, Bendhack LM. Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology. 2005 ; 42 63-68.[citado 2024 out. 17 ]
Vancouver
Rascado RR, Bendhack LM. Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology. 2005 ; 42 63-68.[citado 2024 out. 17 ]
Artigo de periodico
Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms (2004)
Molin, Josiane Cristina; Bendhack, Lusiane Maria
Source: Vascular Pharmacology. Unidade: FCFRP
Subjects: FARMACOLOGIA, CARDIOLOGIA
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ABNT
MOLIN, Josiane Cristina e BENDHACK, Lusiane Maria. Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms. Vascular Pharmacology, v. 42, p. 1-6, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2004.11.006. Acesso em: 17 out. 2024.
APA
Molin, J. C., & Bendhack, L. M. (2004). Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms. Vascular Pharmacology, 42, 1-6. doi:10.1016/j.vph.2004.11.006
NLM
Molin JC, Bendhack LM. Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms [Internet]. Vascular Pharmacology. 2004 ; 42 1-6.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2004.11.006
Vancouver
Molin JC, Bendhack LM. Clonidine induces rat aorta relaxation by nitric oxide-dependent and -independent mechanisms [Internet]. Vascular Pharmacology. 2004 ; 42 1-6.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2004.11.006
Artigo de periodico
Endothelin-1-induced contraction is impaired in the tail artery of renal hypertensive rats (2002)
Linder, Áurea Elizabeth; Bendhack, Lusiane Maria
Source: Vascular Pharmacology. Unidade: FCFRP
Assunto: FARMACOLOGIA
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ABNT
LINDER, Áurea Elizabeth e BENDHACK, Lusiane Maria. Endothelin-1-induced contraction is impaired in the tail artery of renal hypertensive rats. Vascular Pharmacology, v. 39, p. 77-82, 2002Tradução . . Disponível em: https://doi.org/10.1016/s1537-1891(02)00282-3. Acesso em: 17 out. 2024.
APA
Linder, Á. E., & Bendhack, L. M. (2002). Endothelin-1-induced contraction is impaired in the tail artery of renal hypertensive rats. Vascular Pharmacology, 39, 77-82. doi:10.1016/s1537-1891(02)00282-3
NLM
Linder ÁE, Bendhack LM. Endothelin-1-induced contraction is impaired in the tail artery of renal hypertensive rats [Internet]. Vascular Pharmacology. 2002 ; 39 77-82.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/s1537-1891(02)00282-3
Vancouver
Linder ÁE, Bendhack LM. Endothelin-1-induced contraction is impaired in the tail artery of renal hypertensive rats [Internet]. Vascular Pharmacology. 2002 ; 39 77-82.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/s1537-1891(02)00282-3

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