Filtros : "BENDHACK, LUSIANE MARIA" "Indexado no Medline" Removido: "Indexado no : Science Citation Index Expanded" Limpar

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  • Source: Vascualr Pharmacology. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, VASODILATAÇÃO, SEPSE

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    • ABNT

      ARAÚJO, Alice Valença et al. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP). Vascualr Pharmacology, v. 54, n. 1-2, p. 44-51, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2010.12.002. Acesso em: 17 out. 2024.
    • APA

      Araújo, A. V., Ferezin, C. Z., Rodrigues, G. J., Lunardi, C. N., Vercesi, J. A., Grando, M. D., et al. (2011). Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP). Vascualr Pharmacology, 54( 1-2), 44-51. doi:10.1016/j.vph.2010.12.002
    • NLM

      Araújo AV, Ferezin CZ, Rodrigues GJ, Lunardi CN, Vercesi JA, Grando MD, Bonaventura D, Bendhack LM. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) [Internet]. Vascualr Pharmacology. 2011 ; 54( 1-2): 44-51.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2010.12.002
    • Vancouver

      Araújo AV, Ferezin CZ, Rodrigues GJ, Lunardi CN, Vercesi JA, Grando MD, Bonaventura D, Bendhack LM. Prostacyclin, not only nitric oxide, is a mediator of the vasorelaxation induced by acetylcholine in aortas from rats submitted to cecal ligation and perforation (CLP) [Internet]. Vascualr Pharmacology. 2011 ; 54( 1-2): 44-51.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.vph.2010.12.002
  • Source: Archives of Toxicology. Unidades: FORP, FCFRP, FMRP

    Subjects: HIPERTENSÃO, CHUMBO, DOENÇAS CARDIOVASCULARES

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      RIZZI, Elen et al. Evidence of early involvement of matrix metalloproteinase-2 in lead-induced hypertension. Archives of Toxicology, v. 83, n. 5, p. 439-449, 2009Tradução . . Disponível em: https://doi.org/10.1007/s00204-008-0363-1. Acesso em: 17 out. 2024.
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      Rizzi, E., Castro, M. M., Fernandes, K., Barbosa Júnior, F., Arisi, G. M., Garcia-Cairasco, N., et al. (2009). Evidence of early involvement of matrix metalloproteinase-2 in lead-induced hypertension. Archives of Toxicology, 83( 5), 439-449. doi:10.1007/s00204-008-0363-1
    • NLM

      Rizzi E, Castro MM, Fernandes K, Barbosa Júnior F, Arisi GM, Garcia-Cairasco N, Bendhack LM, Tanus-Santos JE, Gerlach RF. Evidence of early involvement of matrix metalloproteinase-2 in lead-induced hypertension [Internet]. Archives of Toxicology. 2009 ; 83( 5): 439-449.[citado 2024 out. 17 ] Available from: https://doi.org/10.1007/s00204-008-0363-1
    • Vancouver

      Rizzi E, Castro MM, Fernandes K, Barbosa Júnior F, Arisi GM, Garcia-Cairasco N, Bendhack LM, Tanus-Santos JE, Gerlach RF. Evidence of early involvement of matrix metalloproteinase-2 in lead-induced hypertension [Internet]. Archives of Toxicology. 2009 ; 83( 5): 439-449.[citado 2024 out. 17 ] Available from: https://doi.org/10.1007/s00204-008-0363-1
  • Source: Vascular Pharmacology. Unidade: FCFRP

    Subjects: ENDOTÉLIO, AORTA, RETÍCULO SARCOPLÁSMICO

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      ROCHA, Matheus L. e BENDHACK, Lusiane Maria. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology, v. 50, n. 3-4, p. 98-103, 2009Tradução . . Acesso em: 17 out. 2024.
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      Rocha, M. L., & Bendhack, L. M. (2009). Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology, 50( 3-4), 98-103.
    • NLM

      Rocha ML, Bendhack LM. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology. 2009 ; 50( 3-4): 98-103.[citado 2024 out. 17 ]
    • Vancouver

      Rocha ML, Bendhack LM. Relaxation evoked by extracellular 'Ca POT. 2+' in rat aorta is nerve-independent and involves sarcoplasmic reticulum and L-type 'Ca POT. 2+' channel. Vascular Pharmacology. 2009 ; 50( 3-4): 98-103.[citado 2024 out. 17 ]
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, ENDOTÉLIO VASCULAR, VASODILATAÇÃO

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    • ABNT

      BONAVENTURA, Daniella et al. A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta. Nitric Oxide, v. 18, n. 4, p. 287-295, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2008.02.004. Acesso em: 17 out. 2024.
    • APA

      Bonaventura, D., Lunardi, C. N., Rodrigues, G. J., Neto, M. A., & Bendhack, L. M. (2008). A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta. Nitric Oxide, 18( 4), 287-295. doi:10.1016/j.niox.2008.02.004
    • NLM

      Bonaventura D, Lunardi CN, Rodrigues GJ, Neto MA, Bendhack LM. A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta [Internet]. Nitric Oxide. 2008 ; 18( 4): 287-295.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2008.02.004
    • Vancouver

      Bonaventura D, Lunardi CN, Rodrigues GJ, Neto MA, Bendhack LM. A novel mechanism of vascular relaxation induced by sodium nitroprusside in the isolated rat aorta [Internet]. Nitric Oxide. 2008 ; 18( 4): 287-295.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2008.02.004
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, NEFROPATIAS, VITAMINA C

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      RODRIGUES, G. J. et al. Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats. Nitric Oxide, v. 18, n. 3, p. 176-183, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2007.12.002. Acesso em: 17 out. 2024.
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      Rodrigues, G. J., Lunardi, C. N., Lima, R. G., Santos, C. X., Laurindo, F. R. M., Silva, R. S. da, & Bendhack, L. M. (2008). Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats. Nitric Oxide, 18( 3), 176-183. doi:10.1016/j.niox.2007.12.002
    • NLM

      Rodrigues GJ, Lunardi CN, Lima RG, Santos CX, Laurindo FRM, Silva RS da, Bendhack LM. Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats [Internet]. Nitric Oxide. 2008 ; 18( 3): 176-183.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2007.12.002
    • Vancouver

      Rodrigues GJ, Lunardi CN, Lima RG, Santos CX, Laurindo FRM, Silva RS da, Bendhack LM. Vitamin C improves the effect of a new nitric oxide donor on the vascular smooth muscle from renal hypertensive rats [Internet]. Nitric Oxide. 2008 ; 18( 3): 176-183.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2007.12.002
  • Source: European Journal of Pharmacology. Unidades: FCFRP, FORP, FMRP

    Subjects: HIPERTENSÃO, DOENÇAS VASCULARES

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      MARTINEZ, Marcio L. L. et al. Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats. European Journal of Pharmacology, v. 591, n. 1-3, p. 224-230, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2008.06.096. Acesso em: 17 out. 2024.
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      Martinez, M. L. L., Castro, M. M., Rizzi, E., Fernandes, K., Demacq, C., Bendhack, L. M., et al. (2008). Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats. European Journal of Pharmacology, 591( 1-3), 224-230. doi:10.1016/j.ejphar.2008.06.096
    • NLM

      Martinez MLL, Castro MM, Rizzi E, Fernandes K, Demacq C, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats [Internet]. European Journal of Pharmacology. 2008 ; 591( 1-3): 224-230.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2008.06.096
    • Vancouver

      Martinez MLL, Castro MM, Rizzi E, Fernandes K, Demacq C, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine reduces matrix metalloproteinase-2 activity and reverses vascular dysfunction in renovascular hypertensive rats [Internet]. European Journal of Pharmacology. 2008 ; 591( 1-3): 224-230.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2008.06.096
  • Source: Journal of Pharmacy and Pharmacology. Unidades: FCFRP, EERP

    Assunto: FARMACOLOGIA

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      TIRAPELLI, Carlos Renato et al. Pimaradienoic acid inhibits vascular contraction and induces hypotension in normotensive rats. Journal of Pharmacy and Pharmacology, v. 60, n. 4, p. 453-459, 2008Tradução . . Disponível em: https://doi.org/10.1211/jpp.60.4.0007. Acesso em: 17 out. 2024.
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      Tirapelli, C. R., Neto, M. dos A., Bonaventura, D., Melo, M. C. C. de, Ambrosio, S. R., Oliveira, A. M. de, et al. (2008). Pimaradienoic acid inhibits vascular contraction and induces hypotension in normotensive rats. Journal of Pharmacy and Pharmacology, 60( 4), 453-459. doi:10.1211/jpp.60.4.0007
    • NLM

      Tirapelli CR, Neto M dos A, Bonaventura D, Melo MCC de, Ambrosio SR, Oliveira AM de, Bendhack LM, Costa FB da. Pimaradienoic acid inhibits vascular contraction and induces hypotension in normotensive rats [Internet]. Journal of Pharmacy and Pharmacology. 2008 ; 60( 4): 453-459.[citado 2024 out. 17 ] Available from: https://doi.org/10.1211/jpp.60.4.0007
    • Vancouver

      Tirapelli CR, Neto M dos A, Bonaventura D, Melo MCC de, Ambrosio SR, Oliveira AM de, Bendhack LM, Costa FB da. Pimaradienoic acid inhibits vascular contraction and induces hypotension in normotensive rats [Internet]. Journal of Pharmacy and Pharmacology. 2008 ; 60( 4): 453-459.[citado 2024 out. 17 ] Available from: https://doi.org/10.1211/jpp.60.4.0007
  • Source: European Journal of Pharmacology. Unidades: FMRP, FCFRP, FORP

    Subjects: DIABETES MELLITUS, METALOPROTEINASES, DOENÇAS CARDIOVASCULARES, ESTRESSE OXIDATIVO

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      MARTINEZ, Marcio L. L. et al. Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats. European Journal of Pharmacology, v. 599, n. 1-3, p. 110-116, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2008.10.007. Acesso em: 17 out. 2024.
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      Martinez, M. L. L., Rizzi, E., Castro, M. M., Fernandes, K., Bendhack, L. M., Gerlach, R. F., & Tanus-Santos, J. E. (2008). Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats. European Journal of Pharmacology, 599( 1-3), 110-116. doi:10.1016/j.ejphar.2008.10.007
    • NLM

      Martinez MLL, Rizzi E, Castro MM, Fernandes K, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats [Internet]. European Journal of Pharmacology. 2008 ; 599( 1-3): 110-116.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2008.10.007
    • Vancouver

      Martinez MLL, Rizzi E, Castro MM, Fernandes K, Bendhack LM, Gerlach RF, Tanus-Santos JE. Lercanidipine decreases vascular matrix metalloproteinase-2 activity and protects against vascular dysfunction in diabetic rats [Internet]. European Journal of Pharmacology. 2008 ; 599( 1-3): 110-116.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2008.10.007
  • Source: Journal of Inorganic Biochemistry. Unidades: FCFRP, FFCLRP

    Subjects: ÓXIDO NÍTRICO, FOTOQUÍMICA, BIOQUÍMICA INORGÂNICA

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      OLIVEIRA, Fabiana de Souza et al. The macrocyclic effect and vasodilation response based on the photoinduced nitric oxide release from trans-'[RuCl(tetraazamacrocycle)NO]POT. 2+'. Journal of Inorganic Biochemistry, v. 101, n. 2, p. 313-320, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.jinorgbio.2006.10.008. Acesso em: 17 out. 2024.
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      Oliveira, F. de S., Ferreira, K. Q., Bonaventura, D., Bendhack, L. M., Tedesco, A. C., Machado, S. de P., et al. (2007). The macrocyclic effect and vasodilation response based on the photoinduced nitric oxide release from trans-'[RuCl(tetraazamacrocycle)NO]POT. 2+'. Journal of Inorganic Biochemistry, 101( 2), 313-320. doi:10.1016/j.jinorgbio.2006.10.008
    • NLM

      Oliveira F de S, Ferreira KQ, Bonaventura D, Bendhack LM, Tedesco AC, Machado S de P, Tfouni E, Silva RS da. The macrocyclic effect and vasodilation response based on the photoinduced nitric oxide release from trans-'[RuCl(tetraazamacrocycle)NO]POT. 2+' [Internet]. Journal of Inorganic Biochemistry. 2007 ; 101( 2): 313-320.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.jinorgbio.2006.10.008
    • Vancouver

      Oliveira F de S, Ferreira KQ, Bonaventura D, Bendhack LM, Tedesco AC, Machado S de P, Tfouni E, Silva RS da. The macrocyclic effect and vasodilation response based on the photoinduced nitric oxide release from trans-'[RuCl(tetraazamacrocycle)NO]POT. 2+' [Internet]. Journal of Inorganic Biochemistry. 2007 ; 101( 2): 313-320.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.jinorgbio.2006.10.008
  • Source: Vascular Pharmacology. Unidade: FCFRP

    Subjects: VASODILATAÇÃO, RUTÊNIO, ÓXIDO NÍTRICO

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      LUNARDI, Claure N. et al. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology, v. 47, n. 2-3, p. 139-144, 2007Tradução . . Acesso em: 17 out. 2024.
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      Lunardi, C. N., Vercesi, J. A., Silva, R. S. da, & Bendhack, L. M. (2007). Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology, 47( 2-3), 139-144.
    • NLM

      Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology. 2007 ; 47( 2-3): 139-144.[citado 2024 out. 17 ]
    • Vancouver

      Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Vasorelaxation induced by the new nitric oxide donor cis-[Ru(Cl)'(bpy) IND.2'(NO)](P'F IND.6') is due to activation of 'K IND.Ca' by a cGMP-dependent pathway. Vascular Pharmacology. 2007 ; 47( 2-3): 139-144.[citado 2024 out. 17 ]
  • Source: Biological and Pharmaceutical Bulletin. Unidade: FCFRP

    Subjects: AORTA, CANAIS IÔNICOS

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      ROCHA, Matheus Lavorenti e BENDHACK, Lusiane Maria. Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin, v. 30, n. 11, p. 2098-2104, 2007Tradução . . Acesso em: 17 out. 2024.
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      Rocha, M. L., & Bendhack, L. M. (2007). Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin, 30( 11), 2098-2104.
    • NLM

      Rocha ML, Bendhack LM. Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin. 2007 ; 30( 11): 2098-2104.[citado 2024 out. 17 ]
    • Vancouver

      Rocha ML, Bendhack LM. Effects of 'K POT.+' channel modulators on oscillatory contractions in sinoartic denervated rat aortas. Biological and Pharmaceutical Bulletin. 2007 ; 30( 11): 2098-2104.[citado 2024 out. 17 ]
  • Source: Journal of Pharmacology and Experimental Therapeutics. Unidades: FMRP, FCFRP

    Subjects: ÓXIDO NÍTRICO, AORTA, VASODILATAÇÃO

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      RODRIGUES, G. J. et al. Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, v. 323, n. 3, p. 831-837, 2007Tradução . . Acesso em: 17 out. 2024.
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      Rodrigues, G. J., Restini, C. B., Lunardi, C. N., Moreira, J. E., Lima, R. G. de, Silva, R. S. da, & Bendhack, L. M. (2007). Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics, 323( 3), 831-837.
    • NLM

      Rodrigues GJ, Restini CB, Lunardi CN, Moreira JE, Lima RG de, Silva RS da, Bendhack LM. Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics. 2007 ; 323( 3): 831-837.[citado 2024 out. 17 ]
    • Vancouver

      Rodrigues GJ, Restini CB, Lunardi CN, Moreira JE, Lima RG de, Silva RS da, Bendhack LM. Caveolae dysfunction contributes to impaired relaxation induced by nitric oxide donor in aorta from renal hypertensive rats. Journal of Pharmacology and Experimental Therapeutics. 2007 ; 323( 3): 831-837.[citado 2024 out. 17 ]
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, RUTÊNIO, FARMACOLOGIA

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      LUNARDI, Claure Nain et al. Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells. Nitric Oxide, v. 15, p. 252-258, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2006.02.001. Acesso em: 17 out. 2024.
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      Lunardi, C. N., Cacciari, A. L., Silva, S. R., & Bendhack, L. M. (2006). Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells. Nitric Oxide, 15, 252-258. doi:10.1016/j.niox.2006.02.001
    • NLM

      Lunardi CN, Cacciari AL, Silva SR, Bendhack LM. Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells [Internet]. Nitric Oxide. 2006 ; 15 252-258.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2006.02.001
    • Vancouver

      Lunardi CN, Cacciari AL, Silva SR, Bendhack LM. Cytosolic calcium concentration is reduced by photolysis of a nitrosyl ruthenium complex in vascular smooth muscle cells [Internet]. Nitric Oxide. 2006 ; 15 252-258.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2006.02.001
  • Source: Nitric Oxide. Unidade: FCFRP

    Subjects: ÓXIDO NÍTRICO, RUTÊNIO, VASODILATAÇÃO, FARMACOLOGIA

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      BONAVENTURA, Daniella et al. Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex. Nitric Oxide, v. 15, n. 4, p. 387-394, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2006.04.260. Acesso em: 17 out. 2024.
    • APA

      Bonaventura, D., Oliveira, F. de S., Lunardi, C. N., Vercesi, J. A., Silva, R. S. da, & Bendhack, L. M. (2006). Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex. Nitric Oxide, 15( 4), 387-394. doi:10.1016/j.niox.2006.04.260
    • NLM

      Bonaventura D, Oliveira F de S, Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex [Internet]. Nitric Oxide. 2006 ; 15( 4): 387-394.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2006.04.260
    • Vancouver

      Bonaventura D, Oliveira F de S, Lunardi CN, Vercesi JA, Silva RS da, Bendhack LM. Characterization of the mechanisms of action and nitric oxide species involved in the relaxationinduced by the ruthenium complex [Internet]. Nitric Oxide. 2006 ; 15( 4): 387-394.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.niox.2006.04.260
  • Source: Nitric Oxide. Unidades: FCFRP, FFCLRP

    Subjects: FARMACOLOGIA, CARDIOLOGIA

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      FEREZIN, Camila Z. et al. The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide, v. 13, p. 170-175, 2005Tradução . . Acesso em: 17 out. 2024.
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      Ferezin, C. Z., Oliveira, F. S., Silva, R. S. da, Simioni, A. R., Tedesco, A. C., & Bendhack, L. M. (2005). The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide, 13, 170-175.
    • NLM

      Ferezin CZ, Oliveira FS, Silva RS da, Simioni AR, Tedesco AC, Bendhack LM. The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide. 2005 ; 13 170-175.[citado 2024 out. 17 ]
    • Vancouver

      Ferezin CZ, Oliveira FS, Silva RS da, Simioni AR, Tedesco AC, Bendhack LM. The complex trans-'[RuCl([15]ane'N IND.4')NO]' POT. 2+' induces rat aorta relaxation by ultraviolet light irradiation. Nitric Oxide. 2005 ; 13 170-175.[citado 2024 out. 17 ]
  • Source: Clinical and experimental pharmacology and physiology. Unidade: FCFRP

    Subjects: FARMACOLOGIA, QUÍMICA INORGÂNICA

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    • ABNT

      BONAVENTURA, Daniella et al. Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation. Clinical and experimental pharmacology and physiology, v. 32, p. 478-481, 2005Tradução . . Acesso em: 17 out. 2024.
    • APA

      Bonaventura, D., Oliveira, F. S., Silva, R. S. da, & Bendhack, L. M. (2005). Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation. Clinical and experimental pharmacology and physiology, 32, 478-481.
    • NLM

      Bonaventura D, Oliveira FS, Silva RS da, Bendhack LM. Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation. Clinical and experimental pharmacology and physiology. 2005 ; 32 478-481.[citado 2024 out. 17 ]
    • Vancouver

      Bonaventura D, Oliveira FS, Silva RS da, Bendhack LM. Decreased vasodilation induced by a new nitric oxide donor in two kidney, one clip hypertensive rats is due to impaired 'K POT +' channel activation. Clinical and experimental pharmacology and physiology. 2005 ; 32 478-481.[citado 2024 out. 17 ]
  • Source: Vascular Pharmacology. Unidade: FCFRP

    Assunto: FARMACOLOGIA

    How to cite
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    • ABNT

      RASCADO, Ricardo Radighieri e BENDHACK, Lusiane Maria. Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology, v. 42, p. 63-68, 2005Tradução . . Acesso em: 17 out. 2024.
    • APA

      Rascado, R. R., & Bendhack, L. M. (2005). Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology, 42, 63-68.
    • NLM

      Rascado RR, Bendhack LM. Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology. 2005 ; 42 63-68.[citado 2024 out. 17 ]
    • Vancouver

      Rascado RR, Bendhack LM. Activation of 'alfa IND 2-adrenoceptors' is necessary to induce nitric oxide release in isoprenaline-induced relaxation. Vascular Pharmacology. 2005 ; 42 63-68.[citado 2024 out. 17 ]
  • Source: European Journal of Pharmacology. Unidade: FCFRP

    Subjects: FARMACOLOGIA, CARDIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      MOLIN, Josiane C. e SGUILLA, Fabiana S. e BENDHACK, Lusiane Maria. Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium. European Journal of Pharmacology, v. 522, p. 94-99, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2005.08.034. Acesso em: 17 out. 2024.
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      Molin, J. C., Sguilla, F. S., & Bendhack, L. M. (2005). Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium. European Journal of Pharmacology, 522, 94-99. doi:10.1016/j.ejphar.2005.08.034
    • NLM

      Molin JC, Sguilla FS, Bendhack LM. Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium [Internet]. European Journal of Pharmacology. 2005 ; 522 94-99.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2005.08.034
    • Vancouver

      Molin JC, Sguilla FS, Bendhack LM. Decreased contraction to phenylephrine by ouabain in 2K-1C rat aorta is modulated by the endothelium [Internet]. European Journal of Pharmacology. 2005 ; 522 94-99.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.ejphar.2005.08.034
  • Source: Comparative Biochemistry and Physiology Part C. Unidades: FCFRP, FMRP

    Subjects: TOXICOLOGIA, FARMACOLOGIA CLÍNICA

    Acesso à fonteDOIHow to cite
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      VASCONCELOS, Flávio et al. Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines. Comparative Biochemistry and Physiology Part C, v. 141, p. 85-92, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.cca.2005.05.012. Acesso em: 17 out. 2024.
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      Vasconcelos, F., Lanchote, V. L., Bendhack, L. M., Giglio, J. R., Sampaio, S. V., & Arantes, E. C. (2005). Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines. Comparative Biochemistry and Physiology Part C, 141, 85-92. doi:10.1016/j.cca.2005.05.012
    • NLM

      Vasconcelos F, Lanchote VL, Bendhack LM, Giglio JR, Sampaio SV, Arantes EC. Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines [Internet]. Comparative Biochemistry and Physiology Part C. 2005 ; 141 85-92.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.cca.2005.05.012
    • Vancouver

      Vasconcelos F, Lanchote VL, Bendhack LM, Giglio JR, Sampaio SV, Arantes EC. Effects of voltage-gated 'Na POT.+' channel toxins from Tityus serrulatus venom on rat arterial blood pressure and plasma catecholamines [Internet]. Comparative Biochemistry and Physiology Part C. 2005 ; 141 85-92.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.cca.2005.05.012
  • Source: Pharmacology. Unidade: FCFRP

    Assunto: FARMACOLOGIA

    How to cite
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    • ABNT

      GRIFONI, Samira de Campos e BENDHACK, Lusiane Maria. Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology, v. 70, p. 160-168, 2004Tradução . . Acesso em: 17 out. 2024.
    • APA

      Grifoni, S. de C., & Bendhack, L. M. (2004). Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology, 70, 160-168.
    • NLM

      Grifoni S de C, Bendhack LM. Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology. 2004 ; 70 160-168.[citado 2024 out. 17 ]
    • Vancouver

      Grifoni S de C, Bendhack LM. Functional study of the '[Ca POT.2+] IND i' signaling pathway in aortas of L-NAME-Hypertensive rats. Pharmacology. 2004 ; 70 160-168.[citado 2024 out. 17 ]

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