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Artigo de periodico
Purinergic P2 and glutamate NMDA receptor coupling contributes to osmotically driven excitability in hypothalamic magnocellular neurosecretory neurons (2021)
Ferreira Neto, Hildebrando Candido; Antunes, Vagner Roberto ; Stern, Javier E.
Fonte: The Journal of Physiology. Unidade: ICB
Assuntos: FISIOLOGIA, NEURÔNIOS, HIPOTÁLAMO, GLUTAMATOS, BLOQUEADORES NEUROMUSCULARES, MEMBRANA PLASMÁTICA
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ABNT
FERREIRA NETO, Hildebrando Candido e ANTUNES, Vagner Roberto e STERN, Javier E. Purinergic P2 and glutamate NMDA receptor coupling contributes to osmotically driven excitability in hypothalamic magnocellular neurosecretory neurons. The Journal of Physiology, v. 599, n. 14, p. 3531-3547, 2021Tradução . . Disponível em: https://doi.org/10.1113/JP281411. Acesso em: 17 nov. 2024.
APA
Ferreira Neto, H. C., Antunes, V. R., & Stern, J. E. (2021). Purinergic P2 and glutamate NMDA receptor coupling contributes to osmotically driven excitability in hypothalamic magnocellular neurosecretory neurons. The Journal of Physiology, 599( 14), 3531-3547. doi:10.1113/JP281411
NLM
Ferreira Neto HC, Antunes VR, Stern JE. Purinergic P2 and glutamate NMDA receptor coupling contributes to osmotically driven excitability in hypothalamic magnocellular neurosecretory neurons [Internet]. The Journal of Physiology. 2021 ; 599( 14): 3531-3547.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1113/JP281411
Vancouver
Ferreira Neto HC, Antunes VR, Stern JE. Purinergic P2 and glutamate NMDA receptor coupling contributes to osmotically driven excitability in hypothalamic magnocellular neurosecretory neurons [Internet]. The Journal of Physiology. 2021 ; 599( 14): 3531-3547.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1113/JP281411
Artigo de periodico
Exercise training rescues the electrical activity of liver‐projecting DMNV neurones in response to oxytocin in spontaneously hypertensive rats (2021)
Santos, Karoline Martins dos ; Moraes, Davi José de Almeida; Moraes, Melina Pires da Silva; Antunes, Vagner Roberto
Fonte: Journal of Neuroendocrinology. Unidades: FMRP, ICB
Assuntos: FISIOLOGIA, EXERCÍCIO FÍSICO, NEURÔNIOS, FÍGADO, OCITOCINA, HIPOTÁLAMO, RATOS WISTAR, SISTEMA NERVOSO PARASSIMPÁTICO, GLICOSE
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ABNT
SANTOS, Karoline Martins dos et al. Exercise training rescues the electrical activity of liver‐projecting DMNV neurones in response to oxytocin in spontaneously hypertensive rats. Journal of Neuroendocrinology, v. 33, n. 5, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1111/jne.12977. Acesso em: 17 nov. 2024.
APA
Santos, K. M. dos, Moraes, D. J. de A., Moraes, M. P. da S., & Antunes, V. R. (2021). Exercise training rescues the electrical activity of liver‐projecting DMNV neurones in response to oxytocin in spontaneously hypertensive rats. Journal of Neuroendocrinology, 33( 5), 1-11. doi:10.1111/jne.12977
NLM
Santos KM dos, Moraes DJ de A, Moraes MP da S, Antunes VR. Exercise training rescues the electrical activity of liver‐projecting DMNV neurones in response to oxytocin in spontaneously hypertensive rats [Internet]. Journal of Neuroendocrinology. 2021 ; 33( 5): 1-11.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/jne.12977
Vancouver
Santos KM dos, Moraes DJ de A, Moraes MP da S, Antunes VR. Exercise training rescues the electrical activity of liver‐projecting DMNV neurones in response to oxytocin in spontaneously hypertensive rats [Internet]. Journal of Neuroendocrinology. 2021 ; 33( 5): 1-11.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1111/jne.12977
Artigo de periodico
Depletion of C1 neurons attenuates the salt-induced hypertension in unanesthetized rats (2020)
Ribeiro, Natalia; Sá, Renato W. Martins ; Antunes, Vagner Roberto
Fonte: Brain Research. Unidade: ICB
Assuntos: FISIOLOGIA, SAIS, INGESTÃO, CATECOLAMINAS, VASOPRESSINAS, PRESSÃO SANGUÍNEA, HIPERTENSÃO, RATOS, HIDROXILASE, ATIVAÇÃO ENZIMÁTICA, NEURÔNIOS
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ABNT
RIBEIRO, Natalia e SÁ, Renato W. Martins e ANTUNES, Vagner Roberto. Depletion of C1 neurons attenuates the salt-induced hypertension in unanesthetized rats. Brain Research, v. 1748, p. 10 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2020.147107. Acesso em: 17 nov. 2024.
APA
Ribeiro, N., Sá, R. W. M., & Antunes, V. R. (2020). Depletion of C1 neurons attenuates the salt-induced hypertension in unanesthetized rats. Brain Research, 1748, 10 . doi:10.1016/j.brainres.2020.147107
NLM
Ribeiro N, Sá RWM, Antunes VR. Depletion of C1 neurons attenuates the salt-induced hypertension in unanesthetized rats [Internet]. Brain Research. 2020 ; 1748 10 .[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.brainres.2020.147107
Vancouver
Ribeiro N, Sá RWM, Antunes VR. Depletion of C1 neurons attenuates the salt-induced hypertension in unanesthetized rats [Internet]. Brain Research. 2020 ; 1748 10 .[citado 2024 nov. 17 ] Available from: https://doi.org/10.1016/j.brainres.2020.147107
Artigo de periodico
Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat (2018)
Moreira, Thiago dos Santos ; Antunes, Vagner Roberto ; Falquetto, Bárbara ; Gonzalez, Nephtali Marina
Fonte: Journal of Hypertension. Unidade: ICB
Assuntos: PRESSÃO SANGUÍNEA, FREQUÊNCIA CARDÍACA, RATOS WISTAR, NEURÔNIOS, GÂNGLIOS, TRONCO CEREBRAL, SISTEMA NERVOSO CENTRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOREIRA, Thiago dos Santos et al. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat. Journal of Hypertension, v. 36, n. 12, p. 2444-2452, 2018Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000001871. Acesso em: 17 nov. 2024.
APA
Moreira, T. dos S., Antunes, V. R., Falquetto, B., & Gonzalez, N. M. (2018). Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat. Journal of Hypertension, 36( 12), 2444-2452. doi:10.1097/HJH.0000000000001871
NLM
Moreira T dos S, Antunes VR, Falquetto B, Gonzalez NM. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat [Internet]. Journal of Hypertension. 2018 ; 36( 12): 2444-2452.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1097/HJH.0000000000001871
Vancouver
Moreira T dos S, Antunes VR, Falquetto B, Gonzalez NM. Long-term stimulation of cardiac vagal preganglionic neurons reduces blood pressure in the spontaneously hypertensive rat [Internet]. Journal of Hypertension. 2018 ; 36( 12): 2444-2452.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1097/HJH.0000000000001871
Artigo de periodico
ATP stimulates rat hypothalamic sympathetic neurons by enhancing AMPA receptor-mediated currents (2015)
Ferreira Neto, Hildebrando Candido; Antunes, Vagner Roberto ; Stern, Javier Eduardo
Fonte: Journal of Neurophysiology. Unidade: ICB
Assuntos: FISIOLOGIA, HIPOTÁLAMO, RATOS, NEURÔNIOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA NETO, Hildebrando Candido e ANTUNES, Vagner Roberto e STERN, Javier Eduardo. ATP stimulates rat hypothalamic sympathetic neurons by enhancing AMPA receptor-mediated currents. Journal of Neurophysiology, v. 114, n. 1, p. 159-169, 2015Tradução . . Disponível em: https://doi.org/10.1152/jn.01011.2014. Acesso em: 17 nov. 2024.
APA
Ferreira Neto, H. C., Antunes, V. R., & Stern, J. E. (2015). ATP stimulates rat hypothalamic sympathetic neurons by enhancing AMPA receptor-mediated currents. Journal of Neurophysiology, 114( 1), 159-169. doi:10.1152/jn.01011.2014
NLM
Ferreira Neto HC, Antunes VR, Stern JE. ATP stimulates rat hypothalamic sympathetic neurons by enhancing AMPA receptor-mediated currents [Internet]. Journal of Neurophysiology. 2015 ; 114( 1): 159-169.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1152/jn.01011.2014
Vancouver
Ferreira Neto HC, Antunes VR, Stern JE. ATP stimulates rat hypothalamic sympathetic neurons by enhancing AMPA receptor-mediated currents [Internet]. Journal of Neurophysiology. 2015 ; 114( 1): 159-169.[citado 2024 nov. 17 ] Available from: https://doi.org/10.1152/jn.01011.2014

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