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SOUZA, William Danilo Fernandes de e FONSECA, Denise Morais da e SARTORI, Alexandrina. COVID-19 and Multiple Sclerosis: a complex relationship possibly aggravated by Low Vitamin D levels. Cells, v. 12, n. 5, p. 1-28, 2023Tradução . . Disponível em: https://doi.org/10.3390/cells12050684. Acesso em: 17 out. 2024.
APA
Souza, W. D. F. de, Fonseca, D. M. da, & Sartori, A. (2023). COVID-19 and Multiple Sclerosis: a complex relationship possibly aggravated by Low Vitamin D levels. Cells, 12( 5), 1-28. doi:10.3390/cells12050684
NLM
Souza WDF de, Fonseca DM da, Sartori A. COVID-19 and Multiple Sclerosis: a complex relationship possibly aggravated by Low Vitamin D levels [Internet]. Cells. 2023 ; 12( 5): 1-28.[citado 2024 out. 17 ] Available from: https://doi.org/10.3390/cells12050684
Vancouver
Souza WDF de, Fonseca DM da, Sartori A. COVID-19 and Multiple Sclerosis: a complex relationship possibly aggravated by Low Vitamin D levels [Internet]. Cells. 2023 ; 12( 5): 1-28.[citado 2024 out. 17 ] Available from: https://doi.org/10.3390/cells12050684
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BORIM, Patricia Aparecida et al. Effect of rapamycin on MOG-Reactive immune cells and lipopolysaccharide-activated microglia: an in vitro approach for screening new therapies for multiple sclerosis. Journal of Interferon & Cytokine Research, v. 42, n. 4, p. 1-8, 2022Tradução . . Disponível em: https://doi.org/10.1089/jir.2021.0206. Acesso em: 17 out. 2024.
APA
Borim, P. A., Mimura, L. A. N., Pezavento, S. F. G. Z., Polonio, C. M., Peron, J. P. S., Sartori, A., & Silva, T. F. de C. F. (2022). Effect of rapamycin on MOG-Reactive immune cells and lipopolysaccharide-activated microglia: an in vitro approach for screening new therapies for multiple sclerosis. Journal of Interferon & Cytokine Research, 42( 4), 1-8. doi:10.1089/jir.2021.0206
NLM
Borim PA, Mimura LAN, Pezavento SFGZ, Polonio CM, Peron JPS, Sartori A, Silva TF de CF. Effect of rapamycin on MOG-Reactive immune cells and lipopolysaccharide-activated microglia: an in vitro approach for screening new therapies for multiple sclerosis [Internet]. Journal of Interferon & Cytokine Research. 2022 ; 42( 4): 1-8.[citado 2024 out. 17 ] Available from: https://doi.org/10.1089/jir.2021.0206
Vancouver
Borim PA, Mimura LAN, Pezavento SFGZ, Polonio CM, Peron JPS, Sartori A, Silva TF de CF. Effect of rapamycin on MOG-Reactive immune cells and lipopolysaccharide-activated microglia: an in vitro approach for screening new therapies for multiple sclerosis [Internet]. Journal of Interferon & Cytokine Research. 2022 ; 42( 4): 1-8.[citado 2024 out. 17 ] Available from: https://doi.org/10.1089/jir.2021.0206
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FRAGOSO, Yara Dadalti et al. New relapse of multiple sclerosis and neuromyelitis optica as a potential adverse event of AstraZeneca AZD1222 vaccination for COVID-19. Multiple Sclerosis and Related Disorders, v. 57, p. 1-5, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.msard.2021.103321. Acesso em: 17 out. 2024.
APA
Fragoso, Y. D., Gomes, S., Gonçalves, M. V. M., Mendes Junior, E., Oliveira, B. E. S. de, Rocha, C. F., et al. (2022). New relapse of multiple sclerosis and neuromyelitis optica as a potential adverse event of AstraZeneca AZD1222 vaccination for COVID-19. Multiple Sclerosis and Related Disorders, 57, 1-5. doi:10.1016/j.msard.2021.103321
NLM
Fragoso YD, Gomes S, Gonçalves MVM, Mendes Junior E, Oliveira BES de, Rocha CF, Santos GAC dos, Tauil CB, Araujo RV, Peron JPS. New relapse of multiple sclerosis and neuromyelitis optica as a potential adverse event of AstraZeneca AZD1222 vaccination for COVID-19 [Internet]. Multiple Sclerosis and Related Disorders. 2022 ; 57 1-5.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.msard.2021.103321
Vancouver
Fragoso YD, Gomes S, Gonçalves MVM, Mendes Junior E, Oliveira BES de, Rocha CF, Santos GAC dos, Tauil CB, Araujo RV, Peron JPS. New relapse of multiple sclerosis and neuromyelitis optica as a potential adverse event of AstraZeneca AZD1222 vaccination for COVID-19 [Internet]. Multiple Sclerosis and Related Disorders. 2022 ; 57 1-5.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.msard.2021.103321
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NUNES, Bruno Ghirotto. Caracterização do metabolismo e dinâmica mitocondrial em astrócitos derivados de células tronco pluripotentes induzidas de pacientes com esclerose múltipla . 2021. Dissertação (Mestrado) – Universidade de São Paulo, São Paulo, 2021. Disponível em: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-29042022-152032/. Acesso em: 17 out. 2024.
APA
Nunes, B. G. (2021). Caracterização do metabolismo e dinâmica mitocondrial em astrócitos derivados de células tronco pluripotentes induzidas de pacientes com esclerose múltipla (Dissertação (Mestrado). Universidade de São Paulo, São Paulo. Recuperado de https://www.teses.usp.br/teses/disponiveis/42/42133/tde-29042022-152032/
NLM
Nunes BG. Caracterização do metabolismo e dinâmica mitocondrial em astrócitos derivados de células tronco pluripotentes induzidas de pacientes com esclerose múltipla [Internet]. 2021 ;[citado 2024 out. 17 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-29042022-152032/
Vancouver
Nunes BG. Caracterização do metabolismo e dinâmica mitocondrial em astrócitos derivados de células tronco pluripotentes induzidas de pacientes com esclerose múltipla [Internet]. 2021 ;[citado 2024 out. 17 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-29042022-152032/
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ALVES, Natália Munhoz et al. Candida tropicalis systemic infection redirects Leukocyte infiltration to the kidneys attenuating encephalomyelitis. Journal of Fungi, v. 7, n. 9, p. 1-20, 2021Tradução . . Disponível em: https://doi.org/10.3390/jof7090757. Acesso em: 17 out. 2024.
APA
Alves, N. M., Mimura, L. A. N., Viero, R. M., Bagagli, E., Peron, J. P. S., Sartori, A., & Silva, T. F. de C. F. (2021). Candida tropicalis systemic infection redirects Leukocyte infiltration to the kidneys attenuating encephalomyelitis. Journal of Fungi, 7( 9), 1-20. doi:10.3390/jof7090757
NLM
Alves NM, Mimura LAN, Viero RM, Bagagli E, Peron JPS, Sartori A, Silva TF de CF. Candida tropicalis systemic infection redirects Leukocyte infiltration to the kidneys attenuating encephalomyelitis [Internet]. Journal of Fungi. 2021 ; 7( 9): 1-20.[citado 2024 out. 17 ] Available from: https://doi.org/10.3390/jof7090757
Vancouver
Alves NM, Mimura LAN, Viero RM, Bagagli E, Peron JPS, Sartori A, Silva TF de CF. Candida tropicalis systemic infection redirects Leukocyte infiltration to the kidneys attenuating encephalomyelitis [Internet]. Journal of Fungi. 2021 ; 7( 9): 1-20.[citado 2024 out. 17 ] Available from: https://doi.org/10.3390/jof7090757
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LEITE, Jefferson Antonio et al. Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders. British Journal of Pharmacology, v. 179, n. 8, p. 1496-1511, 2021Tradução . . Disponível em: https://doi.org/10.1111/bph.15570. Acesso em: 17 out. 2024.
APA
Leite, J. A., Nunes, B. G., Targhetta, V. P., Lima, J. de, & Câmara, N. O. S. (2021). Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders. British Journal of Pharmacology, 179( 8), 1496-1511. doi:10.1111/bph.15570
NLM
Leite JA, Nunes BG, Targhetta VP, Lima J de, Câmara NOS. Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders [Internet]. British Journal of Pharmacology. 2021 ; 179( 8): 1496-1511.[citado 2024 out. 17 ] Available from: https://doi.org/10.1111/bph.15570
Vancouver
Leite JA, Nunes BG, Targhetta VP, Lima J de, Câmara NOS. Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders [Internet]. British Journal of Pharmacology. 2021 ; 179( 8): 1496-1511.[citado 2024 out. 17 ] Available from: https://doi.org/10.1111/bph.15570
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OLIVEIRA, Lilian Gomes de et al. Unraveling the link between mitochondrial dynamics and neuroinflammation. Frontiers in Immunology, v. 12, p. 1-14, 2021Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2021.624919. Acesso em: 17 out. 2024.
APA
Oliveira, L. G. de, Angelo, Y. de S., Iglesias, A. H., & Peron, J. P. S. (2021). Unraveling the link between mitochondrial dynamics and neuroinflammation. Frontiers in Immunology, 12, 1-14. doi:10.3389/fimmu.2021.624919
NLM
Oliveira LG de, Angelo Y de S, Iglesias AH, Peron JPS. Unraveling the link between mitochondrial dynamics and neuroinflammation [Internet]. Frontiers in Immunology. 2021 ; 12 1-14.[citado 2024 out. 17 ] Available from: https://doi.org/10.3389/fimmu.2021.624919
Vancouver
Oliveira LG de, Angelo Y de S, Iglesias AH, Peron JPS. Unraveling the link between mitochondrial dynamics and neuroinflammation [Internet]. Frontiers in Immunology. 2021 ; 12 1-14.[citado 2024 out. 17 ] Available from: https://doi.org/10.3389/fimmu.2021.624919
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SILVA, Thais Fernanda de Campos Fraga et al. Selenization of S. cerevisiae increases its protective potential in experimental autoimmune encephalomyelitis by triggering an intestinal immunomodulatory loop. Scientific Reports, v. 10, p. 17 , 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-79102-7. Acesso em: 17 out. 2024.
APA
Silva, T. F. de C. F., Mimura, L. A. N., Oliveira, L. R. C. de, Toledo, J. H. dos S., Borim, P. A., Pezavento, S. F. G. Z., et al. (2020). Selenization of S. cerevisiae increases its protective potential in experimental autoimmune encephalomyelitis by triggering an intestinal immunomodulatory loop. Scientific Reports, 10, 17 . doi:10.1038/s41598-020-79102-7
NLM
Silva TF de CF, Mimura LAN, Oliveira LRC de, Toledo JH dos S, Borim PA, Pezavento SFGZ, Alonso DP, Ribolla PEM, Oliveira CAF de, Fonseca DM da, Villablanca EJ, Sartori A. Selenization of S. cerevisiae increases its protective potential in experimental autoimmune encephalomyelitis by triggering an intestinal immunomodulatory loop [Internet]. Scientific Reports. 2020 ; 10 17 .[citado 2024 out. 17 ] Available from: https://doi.org/10.1038/s41598-020-79102-7
Vancouver
Silva TF de CF, Mimura LAN, Oliveira LRC de, Toledo JH dos S, Borim PA, Pezavento SFGZ, Alonso DP, Ribolla PEM, Oliveira CAF de, Fonseca DM da, Villablanca EJ, Sartori A. Selenization of S. cerevisiae increases its protective potential in experimental autoimmune encephalomyelitis by triggering an intestinal immunomodulatory loop [Internet]. Scientific Reports. 2020 ; 10 17 .[citado 2024 out. 17 ] Available from: https://doi.org/10.1038/s41598-020-79102-7
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SANT'ANNA, Morena Brazil Martins et al. The Crotoxin:SBA-15 complex down-regulates the incidence and intensity of experimental autoimmune encephalomyelitis through peripheral and central actions. Frontiers in Immunology, v. 11, p. 13, 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.591563. Acesso em: 17 out. 2024.
APA
Sant'Anna, M. B. M., Giardini, A. C., Ribeiro, M. A. C., Lopes, F. S. R., Teixeira, N. B., Kimura, L. F., et al. (2020). The Crotoxin:SBA-15 complex down-regulates the incidence and intensity of experimental autoimmune encephalomyelitis through peripheral and central actions. Frontiers in Immunology, 11, 13. doi:10.3389/fimmu.2020.591563
NLM
Sant'Anna MBM, Giardini AC, Ribeiro MAC, Lopes FSR, Teixeira NB, Kimura LF, Buffalo MC, Ribeiro OG, Borrego A, Cabrera WHK, Zambelli VO, Sant'Anna OA, Piccolo G, Ferreira JCB. The Crotoxin:SBA-15 complex down-regulates the incidence and intensity of experimental autoimmune encephalomyelitis through peripheral and central actions [Internet]. Frontiers in Immunology. 2020 ; 11 13.[citado 2024 out. 17 ] Available from: https://doi.org/10.3389/fimmu.2020.591563
Vancouver
Sant'Anna MBM, Giardini AC, Ribeiro MAC, Lopes FSR, Teixeira NB, Kimura LF, Buffalo MC, Ribeiro OG, Borrego A, Cabrera WHK, Zambelli VO, Sant'Anna OA, Piccolo G, Ferreira JCB. The Crotoxin:SBA-15 complex down-regulates the incidence and intensity of experimental autoimmune encephalomyelitis through peripheral and central actions [Internet]. Frontiers in Immunology. 2020 ; 11 13.[citado 2024 out. 17 ] Available from: https://doi.org/10.3389/fimmu.2020.591563
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FREITAS, Carla Longo de. Propriedades imunomoduladoras de células tronco mesenquimais estromais no modelo de encefalomielite experimental autoimune. 2019. Tese (Doutorado) – Universidade de São Paulo, São Paulo, 2019. Disponível em: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-08102021-101808/. Acesso em: 17 out. 2024.
APA
Freitas, C. L. de. (2019). Propriedades imunomoduladoras de células tronco mesenquimais estromais no modelo de encefalomielite experimental autoimune (Tese (Doutorado). Universidade de São Paulo, São Paulo. Recuperado de https://www.teses.usp.br/teses/disponiveis/42/42133/tde-08102021-101808/
NLM
Freitas CL de. Propriedades imunomoduladoras de células tronco mesenquimais estromais no modelo de encefalomielite experimental autoimune [Internet]. 2019 ;[citado 2024 out. 17 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-08102021-101808/
Vancouver
Freitas CL de. Propriedades imunomoduladoras de células tronco mesenquimais estromais no modelo de encefalomielite experimental autoimune [Internet]. 2019 ;[citado 2024 out. 17 ] Available from: https://www.teses.usp.br/teses/disponiveis/42/42133/tde-08102021-101808/
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CARVALHO, Robert Honorato Fraga et al. [11C]PIB PET imaging can detect white and grey matter demyelination in a non-human primate model of progressive multiple sclerosis. Multiple Sclerosis and Related Disorders, v. 35, p. 108-115, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.msard.2019.07.020. Acesso em: 17 out. 2024.
APA
Carvalho, R. H. F., Gregório, C. C. R., Maria, S. C., Garcez, A. T., Duran, F. L. de S., Marques, F. L. N., et al. (2019). [11C]PIB PET imaging can detect white and grey matter demyelination in a non-human primate model of progressive multiple sclerosis. Multiple Sclerosis and Related Disorders, 35, 108-115. doi:10.1016/j.msard.2019.07.020
NLM
Carvalho RHF, Gregório CCR, Maria SC, Garcez AT, Duran FL de S, Marques FLN, Mello LE, Busatto Filho G, Vries EFJ de, Britto LRG de, Buchpiguel CA, Faria D de P. [11C]PIB PET imaging can detect white and grey matter demyelination in a non-human primate model of progressive multiple sclerosis [Internet]. Multiple Sclerosis and Related Disorders. 2019 ; 35 108-115.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.msard.2019.07.020
Vancouver
Carvalho RHF, Gregório CCR, Maria SC, Garcez AT, Duran FL de S, Marques FLN, Mello LE, Busatto Filho G, Vries EFJ de, Britto LRG de, Buchpiguel CA, Faria D de P. [11C]PIB PET imaging can detect white and grey matter demyelination in a non-human primate model of progressive multiple sclerosis [Internet]. Multiple Sclerosis and Related Disorders. 2019 ; 35 108-115.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.msard.2019.07.020
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SOARES, Jaine L. S. e OLIVEIRA, Enedina Maria Lobato de e PONTILLO, Alessandra. Variants in NLRP3 and NLRC4 inflammasome associate with susceptibility and severity of multiple sclerosis. Multiple Sclerosis and Related Disorders, v. 29, p. 26-34, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.msard.2019.01.023. Acesso em: 17 out. 2024.
APA
Soares, J. L. S., Oliveira, E. M. L. de, & Pontillo, A. (2019). Variants in NLRP3 and NLRC4 inflammasome associate with susceptibility and severity of multiple sclerosis. Multiple Sclerosis and Related Disorders, 29, 26-34. doi:10.1016/j.msard.2019.01.023
NLM
Soares JLS, Oliveira EML de, Pontillo A. Variants in NLRP3 and NLRC4 inflammasome associate with susceptibility and severity of multiple sclerosis [Internet]. Multiple Sclerosis and Related Disorders. 2019 ; 29 26-34.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.msard.2019.01.023
Vancouver
Soares JLS, Oliveira EML de, Pontillo A. Variants in NLRP3 and NLRC4 inflammasome associate with susceptibility and severity of multiple sclerosis [Internet]. Multiple Sclerosis and Related Disorders. 2019 ; 29 26-34.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.msard.2019.01.023
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SANTOS, Nilton Barreto dos et al. High dose of dexamethasone protects against EAE-induced motor deficits but impairs learning/memory in C57BL/6 mice. Scientific Reports, v. 9, p. 1-13, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-43217-3. Acesso em: 17 out. 2024.
APA
Santos, N. B. dos, Novaes, L. S., Dragunas, G., Rodrigues, J. R., Brandão, W. N., Camarini, R., et al. (2019). High dose of dexamethasone protects against EAE-induced motor deficits but impairs learning/memory in C57BL/6 mice. Scientific Reports, 9, 1-13. doi:10.1038/s41598-019-43217-3
NLM
Santos NB dos, Novaes LS, Dragunas G, Rodrigues JR, Brandão WN, Camarini R, Peron JPS, Munhoz CD. High dose of dexamethasone protects against EAE-induced motor deficits but impairs learning/memory in C57BL/6 mice [Internet]. Scientific Reports. 2019 ; 9 1-13.[citado 2024 out. 17 ] Available from: https://doi.org/10.1038/s41598-019-43217-3
Vancouver
Santos NB dos, Novaes LS, Dragunas G, Rodrigues JR, Brandão WN, Camarini R, Peron JPS, Munhoz CD. High dose of dexamethasone protects against EAE-induced motor deficits but impairs learning/memory in C57BL/6 mice [Internet]. Scientific Reports. 2019 ; 9 1-13.[citado 2024 out. 17 ] Available from: https://doi.org/10.1038/s41598-019-43217-3
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BRANDÃO, Wesley Nogueira et al. Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice. International Immunopharmacology, v. 75, p. 1-7, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.105809. Acesso em: 17 out. 2024.
APA
Brandão, W. N., Andersen, M. L., Palermo-Neto, J., Peron, J. P. S., & Zager, A. (2019). Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice. International Immunopharmacology, 75, 1-7. doi:10.1016/j.intimp.2019.105809
NLM
Brandão WN, Andersen ML, Palermo-Neto J, Peron JPS, Zager A. Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice [Internet]. International Immunopharmacology. 2019 ; 75 1-7.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.intimp.2019.105809
Vancouver
Brandão WN, Andersen ML, Palermo-Neto J, Peron JPS, Zager A. Therapeutic treatment with Modafinil decreases the severity of experimental autoimmune encephalomyelitis in mice [Internet]. International Immunopharmacology. 2019 ; 75 1-7.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.intimp.2019.105809
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VASCONCELOS, Andrea Rodrigues et al. Nrf2/ARE pathway modulation by dietary energy regulation in neurological disorders. Frontiers in Pharmacology, v. 10, 2019Tradução . . Disponível em: https://doi.org/10.3389/fphar.2019.00033. Acesso em: 17 out. 2024.
APA
Vasconcelos, A. R., Santos, N. B. dos, Scavone, C., & Munhoz, C. D. (2019). Nrf2/ARE pathway modulation by dietary energy regulation in neurological disorders. Frontiers in Pharmacology, 10. doi:10.3389/fphar.2019.00033
NLM
Vasconcelos AR, Santos NB dos, Scavone C, Munhoz CD. Nrf2/ARE pathway modulation by dietary energy regulation in neurological disorders [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2024 out. 17 ] Available from: https://doi.org/10.3389/fphar.2019.00033
Vancouver
Vasconcelos AR, Santos NB dos, Scavone C, Munhoz CD. Nrf2/ARE pathway modulation by dietary energy regulation in neurological disorders [Internet]. Frontiers in Pharmacology. 2019 ; 10[citado 2024 out. 17 ] Available from: https://doi.org/10.3389/fphar.2019.00033
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RAVELLI, Katherine Garcia et al. Nox2-dependent neuroinflammation in an EAE model of multiple sclerosis. Translational Research, v. 10, p. 1-9, 2019Tradução . . Disponível em: https://doi.org/10.1515/tnsci-2019-0001. Acesso em: 17 out. 2024.
APA
Ravelli, K. G., Santos, G. D., Santos, N. B. dos, Nogueira, D. A., Campos, A. C. P., Pagano, R. de L., et al. (2019). Nox2-dependent neuroinflammation in an EAE model of multiple sclerosis. Translational Research, 10, 1-9. doi:10.1515/tnsci-2019-0001
NLM
Ravelli KG, Santos GD, Santos NB dos, Nogueira DA, Campos ACP, Pagano R de L, Hernandes MS, Munhoz CD, Britto LRG de. Nox2-dependent neuroinflammation in an EAE model of multiple sclerosis [Internet]. Translational Research. 2019 ; 10 1-9.[citado 2024 out. 17 ] Available from: https://doi.org/10.1515/tnsci-2019-0001
Vancouver
Ravelli KG, Santos GD, Santos NB dos, Nogueira DA, Campos ACP, Pagano R de L, Hernandes MS, Munhoz CD, Britto LRG de. Nox2-dependent neuroinflammation in an EAE model of multiple sclerosis [Internet]. Translational Research. 2019 ; 10 1-9.[citado 2024 out. 17 ] Available from: https://doi.org/10.1515/tnsci-2019-0001
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
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TRONCOSO, Lian Lopes et al. CCR5Δ32 – A piece of protection in the inflammatory puzzle of multiple sclerosis susceptibility. Human Immunology, v. 79, p. 621-626, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.humimm.2018.04.015. Acesso em: 17 out. 2024.
APA
Troncoso, L. L., Oliveira, E. M. L. de, Finkelszteinjnm Alessandro,, Scheineider, S., & Chies, J. A. B. (2018). CCR5Δ32 – A piece of protection in the inflammatory puzzle of multiple sclerosis susceptibility. Human Immunology, 79, 621-626. doi:10.1016/j.humimm.2018.04.015
NLM
Troncoso LL, Oliveira EML de, Finkelszteinjnm Alessandro, Scheineider S, Chies JAB. CCR5Δ32 – A piece of protection in the inflammatory puzzle of multiple sclerosis susceptibility [Internet]. Human Immunology. 2018 ; 79 621-626.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.humimm.2018.04.015
Vancouver
Troncoso LL, Oliveira EML de, Finkelszteinjnm Alessandro, Scheineider S, Chies JAB. CCR5Δ32 – A piece of protection in the inflammatory puzzle of multiple sclerosis susceptibility [Internet]. Human Immunology. 2018 ; 79 621-626.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.humimm.2018.04.015
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BATALHOTE, Rafaela Florindo Pestana Ferrão. Estudos da transmissão neuromuscular e do músculo esquelético em um modelo experimental de Esclerose Múltipla em camundongos. 2017. Tese (Doutorado) – Universidade de São Paulo, São Paulo, 2017. Disponível em: http://www.teses.usp.br/teses/disponiveis/42/42136/tde-14072017-152319/. Acesso em: 17 out. 2024.
APA
Batalhote, R. F. P. F. (2017). Estudos da transmissão neuromuscular e do músculo esquelético em um modelo experimental de Esclerose Múltipla em camundongos (Tese (Doutorado). Universidade de São Paulo, São Paulo. Recuperado de http://www.teses.usp.br/teses/disponiveis/42/42136/tde-14072017-152319/
NLM
Batalhote RFPF. Estudos da transmissão neuromuscular e do músculo esquelético em um modelo experimental de Esclerose Múltipla em camundongos [Internet]. 2017 ;[citado 2024 out. 17 ] Available from: http://www.teses.usp.br/teses/disponiveis/42/42136/tde-14072017-152319/
Vancouver
Batalhote RFPF. Estudos da transmissão neuromuscular e do músculo esquelético em um modelo experimental de Esclerose Múltipla em camundongos [Internet]. 2017 ;[citado 2024 out. 17 ] Available from: http://www.teses.usp.br/teses/disponiveis/42/42136/tde-14072017-152319/
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSSATO, Cristiano et al. Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis. Biologicals, v. 49, p. 62-68, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.biologicals.2017.06.007. Acesso em: 17 out. 2024.
APA
Rossato, C., Brandão, W. N., Castro, S. B. R. de, Almeida, D. C. de, Maranduba, C. M. da C., Câmara, N. O. S., et al. (2017). Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis. Biologicals, 49, 62-68. doi:10.1016/j.biologicals.2017.06.007
NLM
Rossato C, Brandão WN, Castro SBR de, Almeida DC de, Maranduba CM da C, Câmara NOS, Peron JPS, Silva FS. Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis [Internet]. Biologicals. 2017 ; 49 62-68.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.biologicals.2017.06.007
Vancouver
Rossato C, Brandão WN, Castro SBR de, Almeida DC de, Maranduba CM da C, Câmara NOS, Peron JPS, Silva FS. Stem cells from human-exfoliated deciduous teeth reduce tissue- infiltrating inflammatory cells improving clinical signs in experimental autoimmune encephalomyelitis [Internet]. Biologicals. 2017 ; 49 62-68.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.biologicals.2017.06.007
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZAGER, Adriano et al. Maternal immune activation in late gestation increases neuroinflammation and aggravates experimental autoimmune encephalomyelitis in the offspring. Brain, Behavior, and Immunity, v. 43, p. 159-171, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2014.07.021. Acesso em: 17 out. 2024.
APA
Zager, A., Peron, J. P. S., Mennecier, G., Rodrigues, S. C., Aloia, T. P. A., & Palermo-Neto, J. (2015). Maternal immune activation in late gestation increases neuroinflammation and aggravates experimental autoimmune encephalomyelitis in the offspring. Brain, Behavior, and Immunity, 43, 159-171. doi:10.1016/j.bbi.2014.07.021
NLM
Zager A, Peron JPS, Mennecier G, Rodrigues SC, Aloia TPA, Palermo-Neto J. Maternal immune activation in late gestation increases neuroinflammation and aggravates experimental autoimmune encephalomyelitis in the offspring [Internet]. Brain, Behavior, and Immunity. 2015 ; 43 159-171.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.bbi.2014.07.021
Vancouver
Zager A, Peron JPS, Mennecier G, Rodrigues SC, Aloia TPA, Palermo-Neto J. Maternal immune activation in late gestation increases neuroinflammation and aggravates experimental autoimmune encephalomyelitis in the offspring [Internet]. Brain, Behavior, and Immunity. 2015 ; 43 159-171.[citado 2024 out. 17 ] Available from: https://doi.org/10.1016/j.bbi.2014.07.021