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Artigo de periodico
Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells (2021)
Saito, Renata F.; Rangel, Maria Cristina; Halman, Justin R.; Chandler, Morgan; Andrade, Luciana Nogueira de Sousa; Odete-bustos, Silvina; Furuya, Tatiane Katsue ; Carrasco, Alexis German Murillo; Chaves-filho, Adriano B.; Yoshinaga, Marcos Y.; Chammas, Roger
Source: Nanomedicine-nanotechnology biology and medicine. Unidade: FM
Subjects: RADIAÇÃO (ENERGIA RADIANTE), RADIOTERAPIA, APOPTOSE
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SAITO, Renata F. et al. Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells. Nanomedicine-nanotechnology biology and medicine, v. 36, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.nano.2021.102418. Acesso em: 16 nov. 2024.
APA
Saito, R. F., Rangel, M. C., Halman, J. R., Chandler, M., Andrade, L. N. de S., Odete-bustos, S., et al. (2021). Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells. Nanomedicine-nanotechnology biology and medicine, 36. doi:10.1016/j.nano.2021.102418
NLM
Saito RF, Rangel MC, Halman JR, Chandler M, Andrade LN de S, Odete-bustos S, Furuya TK, Carrasco AGM, Chaves-filho AB, Yoshinaga MY, Chammas R. Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells [Internet]. Nanomedicine-nanotechnology biology and medicine. 2021 ; 36[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.nano.2021.102418
Vancouver
Saito RF, Rangel MC, Halman JR, Chandler M, Andrade LN de S, Odete-bustos S, Furuya TK, Carrasco AGM, Chaves-filho AB, Yoshinaga MY, Chammas R. Simultaneous silencing of lysophosphatidylcholine acyltransferases 1-4 by nucleic acid nanoparticles (NANPs) improves radiation response of melanoma cells [Internet]. Nanomedicine-nanotechnology biology and medicine. 2021 ; 36[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.nano.2021.102418
Artigo de periodico
Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features (2020)
Bellana, D. L; Biscaiaa, S. M. P; Rossia, G. R; Cristala, A. M; Goncalvesa, J. P; Oliveiraa, C. C; Simasa, F. F; Sabryb, D. A; Rochab, H. A. O; Chammas, Roger
Source: Carbohydrate polymers. Unidade: FM
Subjects: ANIMAIS, APOPTOSE, QUÍMICA VERDE, CAMUNDONGOS, PROLIFERAÇÃO CELULAR
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BELLANA, D. L et al. Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features. Carbohydrate polymers, v. 250, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.carbpol.2020.116869. Acesso em: 16 nov. 2024.
APA
Bellana, D. L., Biscaiaa, S. M. P., Rossia, G. R., Cristala, A. M., Goncalvesa, J. P., Oliveiraa, C. C., et al. (2020). Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features. Carbohydrate polymers, 250. doi:10.1016/j.carbpol.2020.116869
NLM
Bellana DL, Biscaiaa SMP, Rossia GR, Cristala AM, Goncalvesa JP, Oliveiraa CC, Simasa FF, Sabryb DA, Rochab HAO, Chammas R. Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features [Internet]. Carbohydrate polymers. 2020 ; 250[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.carbpol.2020.116869
Vancouver
Bellana DL, Biscaiaa SMP, Rossia GR, Cristala AM, Goncalvesa JP, Oliveiraa CC, Simasa FF, Sabryb DA, Rochab HAO, Chammas R. Green does not always mean go: a sulfated galactan from Codium isthmocladum green seaweed reduces melanoma metastasis through direct regulation of malignancy features [Internet]. Carbohydrate polymers. 2020 ; 250[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.carbpol.2020.116869
Artigo de periodico
HPV-mediated resistance to TNF and TRAIL is characterized by global alterations in apoptosis regulatory factors, dysregulation of death receptors, and induction of ROS/RNS (2019)
Tomazella, Tatiane Karen Cabeça; Abreu, Alice de Mello; Andrette, Rafael; Lino, Vanesca de Souza ; Morale, Mirian Galliote ; Aguayo, Francisco ; Termini, Lara ; Villa, Luisa Lina ; Lepique, Ana Paula ; Boccardo, Enrique
Source: International Journal of Molecular Sciences. Unidades: FM, ICB
Subjects: HPV, CÉLULAS EPITELIAIS, ONCOPROTEÍNAS, APOPTOSE, NEOPLASIAS UTERINAS, INFECÇÕES POR PAPILLOMAVIRUS, CITOCINAS
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TOMAZELLA, Tatiane Karen Cabeça et al. HPV-mediated resistance to TNF and TRAIL is characterized by global alterations in apoptosis regulatory factors, dysregulation of death receptors, and induction of ROS/RNS. International Journal of Molecular Sciences, v. 20, n. 1, 2019Tradução . . Disponível em: https://doi.org/10.3390/ijms20010198. Acesso em: 16 nov. 2024.
APA
Tomazella, T. K. C., Abreu, A. de M., Andrette, R., Lino, V. de S., Morale, M. G., Aguayo, F., et al. (2019). HPV-mediated resistance to TNF and TRAIL is characterized by global alterations in apoptosis regulatory factors, dysregulation of death receptors, and induction of ROS/RNS. International Journal of Molecular Sciences, 20( 1). doi:10.3390/ijms20010198
NLM
Tomazella TKC, Abreu A de M, Andrette R, Lino V de S, Morale MG, Aguayo F, Termini L, Villa LL, Lepique AP, Boccardo E. HPV-mediated resistance to TNF and TRAIL is characterized by global alterations in apoptosis regulatory factors, dysregulation of death receptors, and induction of ROS/RNS [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 1):[citado 2024 nov. 16 ] Available from: https://doi.org/10.3390/ijms20010198
Vancouver
Tomazella TKC, Abreu A de M, Andrette R, Lino V de S, Morale MG, Aguayo F, Termini L, Villa LL, Lepique AP, Boccardo E. HPV-mediated resistance to TNF and TRAIL is characterized by global alterations in apoptosis regulatory factors, dysregulation of death receptors, and induction of ROS/RNS [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 1):[citado 2024 nov. 16 ] Available from: https://doi.org/10.3390/ijms20010198
Artigo de periodico
POP-Brazil study protocol: a nationwide cross-sectional evaluation of the prevalence and genotype distribution of human papillomavirus (HPV) in Brazil (2018)
Wendland, Eliana Marcia; Villa, Luisa Lina
Source: Bmj open. Unidade: FM
Subjects: HPV, VACINAS VIRAIS, APOPTOSE, INFLAMAÇÃO
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WENDLAND, Eliana Marcia e VILLA, Luisa Lina. POP-Brazil study protocol: a nationwide cross-sectional evaluation of the prevalence and genotype distribution of human papillomavirus (HPV) in Brazil. Bmj open, v. 8, n. 6, 2018Tradução . . Disponível em: https://doi.org/10.1136/bmjopen-2017-021170. Acesso em: 16 nov. 2024.
APA
Wendland, E. M., & Villa, L. L. (2018). POP-Brazil study protocol: a nationwide cross-sectional evaluation of the prevalence and genotype distribution of human papillomavirus (HPV) in Brazil. Bmj open, 8( 6). doi:10.1136/bmjopen-2017-021170
NLM
Wendland EM, Villa LL. POP-Brazil study protocol: a nationwide cross-sectional evaluation of the prevalence and genotype distribution of human papillomavirus (HPV) in Brazil [Internet]. Bmj open. 2018 ; 8( 6):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1136/bmjopen-2017-021170
Vancouver
Wendland EM, Villa LL. POP-Brazil study protocol: a nationwide cross-sectional evaluation of the prevalence and genotype distribution of human papillomavirus (HPV) in Brazil [Internet]. Bmj open. 2018 ; 8( 6):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1136/bmjopen-2017-021170
Artigo de periodico
PHLDA1 (pleckstrin homology-like domain, family A, member 1) knockdown promotes migration and invasion of MCF10A breast epithelial cells (2018)
Bonatto, Naieli; Carlini, Maria Jose; Garcia, Simone Aparecida de Bessa; Nagai, Maria Aparecida
Source: Cell adhesion & migration. Unidade: FM
Subjects: NEOPLASIAS, EXPRESSÃO GÊNICA, APOPTOSE
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BONATTO, Naieli et al. PHLDA1 (pleckstrin homology-like domain, family A, member 1) knockdown promotes migration and invasion of MCF10A breast epithelial cells. Cell adhesion & migration, v. 12, n. 1, p. 37-46, 2018Tradução . . Disponível em: http://observatorio.fm.usp.br/handle/OPI/25547. Acesso em: 16 nov. 2024.
APA
Bonatto, N., Carlini, M. J., Garcia, S. A. de B., & Nagai, M. A. (2018). PHLDA1 (pleckstrin homology-like domain, family A, member 1) knockdown promotes migration and invasion of MCF10A breast epithelial cells. Cell adhesion & migration, 12( 1), 37-46. doi:10.1080/19336918.2017.1313382
NLM
Bonatto N, Carlini MJ, Garcia SA de B, Nagai MA. PHLDA1 (pleckstrin homology-like domain, family A, member 1) knockdown promotes migration and invasion of MCF10A breast epithelial cells [Internet]. Cell adhesion & migration. 2018 ; 12( 1): 37-46.[citado 2024 nov. 16 ] Available from: http://observatorio.fm.usp.br/handle/OPI/25547
Vancouver
Bonatto N, Carlini MJ, Garcia SA de B, Nagai MA. PHLDA1 (pleckstrin homology-like domain, family A, member 1) knockdown promotes migration and invasion of MCF10A breast epithelial cells [Internet]. Cell adhesion & migration. 2018 ; 12( 1): 37-46.[citado 2024 nov. 16 ] Available from: http://observatorio.fm.usp.br/handle/OPI/25547
Artigo de periodico
Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death (2017)
Tortelli Junior, Tharcisio Citrangulo; Godoy, Lyris Martins Franco de; Souza, Gustavo Antonio de; Bonatto, Diego; Otake, Andreia Hanada; Saito, Renata de Freitas; Rosa, Jose Cesar; Greene, Lewis Joel; Chammas, Roger
Source: Oncotarget. Unidades: FMRP, FM
Subjects: APOPTOSE, NEOPLASIAS CUTÂNEAS, MANIFESTAÇÕES CUTÂNEAS, MELANOMA
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TORTELLI JUNIOR, Tharcisio Citrangulo et al. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death. Oncotarget, v. 8, n. 26, p. 43114-43129, 2017Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.17810. Acesso em: 16 nov. 2024.
APA
Tortelli Junior, T. C., Godoy, L. M. F. de, Souza, G. A. de, Bonatto, D., Otake, A. H., Saito, R. de F., et al. (2017). Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death. Oncotarget, 8( 26), 43114-43129. doi:10.18632/oncotarget.17810
NLM
Tortelli Junior TC, Godoy LMF de, Souza GA de, Bonatto D, Otake AH, Saito R de F, Rosa JC, Greene LJ, Chammas R. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death [Internet]. Oncotarget. 2017 ; 8( 26): 43114-43129.[citado 2024 nov. 16 ] Available from: https://doi.org/10.18632/oncotarget.17810
Vancouver
Tortelli Junior TC, Godoy LMF de, Souza GA de, Bonatto D, Otake AH, Saito R de F, Rosa JC, Greene LJ, Chammas R. Accumulation of prohibitin is a common cellular response to different stressing stimuli and protects melanoma cells from ER stress and chemotherapy-induced cell death [Internet]. Oncotarget. 2017 ; 8( 26): 43114-43129.[citado 2024 nov. 16 ] Available from: https://doi.org/10.18632/oncotarget.17810
Artigo de periodico-carta/editorial
Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy (2017)
Chammas, Roger; Andrade, Luciana Nogueira de Sousa; Jancar, Sonia
Source: Nature reviews cancer. Unidades: FM, ICB
Subjects: APOPTOSE, NEOPLASIAS, ONCOGENES, CÉLULAS CULTIVADAS DE TUMOR
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CHAMMAS, Roger e ANDRADE, Luciana Nogueira de Sousa e JANCAR, Sonia. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/nrc.2017.15. Acesso em: 16 nov. 2024. , 2017
APA
Chammas, R., Andrade, L. N. de S., & Jancar, S. (2017). Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. doi:10.1038/nrc.2017.15
NLM
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/nrc.2017.15
Vancouver
Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy [Internet]. Nature reviews cancer. 2017 ; 17( 4):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/nrc.2017.15
Trabalho de evento-resumo periodico
Role of cellular and extracellular Par-4 expression in tumor formation and drug sensitivity (2017)
Oliveira Filho, Lourival A.; Carlini, Maria José; Nagai, Maria Aparecida
Source: Cancer Research. Conference titles: AACR Special Conference on Translational Control of Cancer: A New Frontier in Cancer Biology and Therapy. Unidade: FM
Subjects: APOPTOSE, BIOMARCADORES, EXPRESSÃO GÊNICA
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OLIVEIRA FILHO, Lourival A. e CARLINI, Maria José e NAGAI, Maria Aparecida. Role of cellular and extracellular Par-4 expression in tumor formation and drug sensitivity. Cancer Research. Baltimore: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://doi.org/10.1158/1538-7445. Acesso em: 16 nov. 2024. , 2017
APA
Oliveira Filho, L. A., Carlini, M. J., & Nagai, M. A. (2017). Role of cellular and extracellular Par-4 expression in tumor formation and drug sensitivity. Cancer Research. Baltimore: Faculdade de Medicina, Universidade de São Paulo. doi:10.1158/1538-7445
NLM
Oliveira Filho LA, Carlini MJ, Nagai MA. Role of cellular and extracellular Par-4 expression in tumor formation and drug sensitivity [Internet]. Cancer Research. 2017 ; 77( 6):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1158/1538-7445
Vancouver
Oliveira Filho LA, Carlini MJ, Nagai MA. Role of cellular and extracellular Par-4 expression in tumor formation and drug sensitivity [Internet]. Cancer Research. 2017 ; 77( 6):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1158/1538-7445
Artigo de periodico
Prostate apoptosis response 4 (PAR4) expression modulates WNT signaling pathways in MCF7 breast cancer cells: A possible mechanism underlying PAR4-mediated docetaxel chemosensitivity (2017)
Garcia, Simone Aparecida de Bessa; Pavanelli, Ana Carolina; Melo, Natália Cruz e; Nagai, Maria Aparecida
Source: International Journal of Molecular Medicine. Unidade: FM
Subjects: NEOPLASIAS MAMÁRIAS, BIOMARCADORES, APOPTOSE, EXPRESSÃO GÊNICA
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GARCIA, Simone Aparecida de Bessa et al. Prostate apoptosis response 4 (PAR4) expression modulates WNT signaling pathways in MCF7 breast cancer cells: A possible mechanism underlying PAR4-mediated docetaxel chemosensitivity. International Journal of Molecular Medicine, v. 39, n. 4, p. 809-818, 2017Tradução . . Disponível em: https://doi.org/10.3892/ijmm.2017.2900. Acesso em: 16 nov. 2024.
APA
Garcia, S. A. de B., Pavanelli, A. C., Melo, N. C. e, & Nagai, M. A. (2017). Prostate apoptosis response 4 (PAR4) expression modulates WNT signaling pathways in MCF7 breast cancer cells: A possible mechanism underlying PAR4-mediated docetaxel chemosensitivity. International Journal of Molecular Medicine, 39( 4), 809-818. doi:10.3892/ijmm.2017.2900
NLM
Garcia SA de B, Pavanelli AC, Melo NC e, Nagai MA. Prostate apoptosis response 4 (PAR4) expression modulates WNT signaling pathways in MCF7 breast cancer cells: A possible mechanism underlying PAR4-mediated docetaxel chemosensitivity [Internet]. International Journal of Molecular Medicine. 2017 ; 39( 4): 809-818.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3892/ijmm.2017.2900
Vancouver
Garcia SA de B, Pavanelli AC, Melo NC e, Nagai MA. Prostate apoptosis response 4 (PAR4) expression modulates WNT signaling pathways in MCF7 breast cancer cells: A possible mechanism underlying PAR4-mediated docetaxel chemosensitivity [Internet]. International Journal of Molecular Medicine. 2017 ; 39( 4): 809-818.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3892/ijmm.2017.2900
Parte de monografia/livro
Alterações genéticas no câncer (2015)
Encinas, Giselly; Mazzotti, Tatiane Furuya; Nagai, Maria Aparecida
Source: Fundamentos da oncologia molecular. Unidade: FM
Subjects: TÉCNICAS CITOLÓGICAS, MUTAÇÃO GENÉTICA, GENES SUPRESSORES DE TUMOR, APOPTOSE
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ENCINAS, Giselly e MAZZOTTI, Tatiane Furuya e NAGAI, Maria Aparecida. Alterações genéticas no câncer. Fundamentos da oncologia molecular. Tradução . São Paulo: Atheneu, 2015. . . Acesso em: 16 nov. 2024.
APA
Encinas, G., Mazzotti, T. F., & Nagai, M. A. (2015). Alterações genéticas no câncer. In Fundamentos da oncologia molecular. São Paulo: Atheneu.
NLM
Encinas G, Mazzotti TF, Nagai MA. Alterações genéticas no câncer. In: Fundamentos da oncologia molecular. São Paulo: Atheneu; 2015. [citado 2024 nov. 16 ]
Vancouver
Encinas G, Mazzotti TF, Nagai MA. Alterações genéticas no câncer. In: Fundamentos da oncologia molecular. São Paulo: Atheneu; 2015. [citado 2024 nov. 16 ]
Artigo de periodico
HOXB7 mRNA is overexpressed in pancreatic ductal adenocarcinomas and its knockdown induces cell cycle arrest and apoptosis (2013)
Chile, Thais; Fortes, Maria Angela Henriques Zanella; Corrêa-Giannella, Maria Lúcia Cardillo; Brentani, Helena Paula; Maria, Durvanei Augusto; Puga, Renato David; Paula, Vanessa de Jesus R. de; Kubrusly, Marcia Saldanha; Novak, Estela Maria; Bacchella, Telésforo; Giorgi, Ricardo Rodrigues
Source: BMC Cancer. Unidade: FM
Subjects: NEOPLASIAS PANCREÁTICAS (GENÉTICA;CIRURGIA), APOPTOSE, ADENOCARCINOMA, EXPRESSÃO GÊNICA
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CHILE, Thais et al. HOXB7 mRNA is overexpressed in pancreatic ductal adenocarcinomas and its knockdown induces cell cycle arrest and apoptosis. BMC Cancer, v. 13, n. 451, p. (online), 2013Tradução . . Disponível em: https://doi.org/10.1186/1471-2407-13-451. Acesso em: 16 nov. 2024.
APA
Chile, T., Fortes, M. A. H. Z., Corrêa-Giannella, M. L. C., Brentani, H. P., Maria, D. A., Puga, R. D., et al. (2013). HOXB7 mRNA is overexpressed in pancreatic ductal adenocarcinomas and its knockdown induces cell cycle arrest and apoptosis. BMC Cancer, 13( 451), (online). doi:10.1186/1471-2407-13-451
NLM
Chile T, Fortes MAHZ, Corrêa-Giannella MLC, Brentani HP, Maria DA, Puga RD, Paula V de JR de, Kubrusly MS, Novak EM, Bacchella T, Giorgi RR. HOXB7 mRNA is overexpressed in pancreatic ductal adenocarcinomas and its knockdown induces cell cycle arrest and apoptosis [Internet]. BMC Cancer. 2013 ; 13( 451): (online).[citado 2024 nov. 16 ] Available from: https://doi.org/10.1186/1471-2407-13-451
Vancouver
Chile T, Fortes MAHZ, Corrêa-Giannella MLC, Brentani HP, Maria DA, Puga RD, Paula V de JR de, Kubrusly MS, Novak EM, Bacchella T, Giorgi RR. HOXB7 mRNA is overexpressed in pancreatic ductal adenocarcinomas and its knockdown induces cell cycle arrest and apoptosis [Internet]. BMC Cancer. 2013 ; 13( 451): (online).[citado 2024 nov. 16 ] Available from: https://doi.org/10.1186/1471-2407-13-451
Artigo de periodico
A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production (2013)
Maria, Durvanei Augusto; Souza, Jean Gabriel de; Morais, Katia L. P.; Berra, Carolina Maria; Zampolli, Hamilton de Campos; Demasi, Marilene; Simons, Simone Michaela; Saito, Renata de Freitas; Chammas, Roger; Chudzinski-Tavassi, Ana Marisa
Source: Investigational New Drugs. Unidade: FM
Subjects: ESTRESSE, NEOPLASIAS (TERAPIA), MECANISMOS DE CONTROLE CELULAR, ADENOCARCINOMA, APOPTOSE
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MARIA, Durvanei Augusto et al. A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production. Investigational New Drugs, v. 31, n. 3, p. 493-505, 2013Tradução . . Disponível em: http://link.springer.com/article/10.1007/s10637-012-9871-1. Acesso em: 16 nov. 2024.
APA
Maria, D. A., Souza, J. G. de, Morais, K. L. P., Berra, C. M., Zampolli, H. de C., Demasi, M., et al. (2013). A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production. Investigational New Drugs, 31( 3), 493-505. doi:10.1007/s10637-012-9871-1
NLM
Maria DA, Souza JG de, Morais KLP, Berra CM, Zampolli H de C, Demasi M, Simons SM, Saito R de F, Chammas R, Chudzinski-Tavassi AM. A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production [Internet]. Investigational New Drugs. 2013 ; 31( 3): 493-505.[citado 2024 nov. 16 ] Available from: http://link.springer.com/article/10.1007/s10637-012-9871-1
Vancouver
Maria DA, Souza JG de, Morais KLP, Berra CM, Zampolli H de C, Demasi M, Simons SM, Saito R de F, Chammas R, Chudzinski-Tavassi AM. A novel proteasome inhibitor acting in mitochondrial dysfunction, ER stress and ROS production [Internet]. Investigational New Drugs. 2013 ; 31( 3): 493-505.[citado 2024 nov. 16 ] Available from: http://link.springer.com/article/10.1007/s10637-012-9871-1
Artigo de periodico
De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus (2013)
Borges, Beatriz E.; Teixeira, Verônica R.; Appel, Marcia H.; Steclan, Chelin A.; Rigo, Fernanda; Filipak Neto, Francisco; Ferreira, Ana M. da Costa; Chammas, Roger; Zanata, Silvio M.; Nakao, Lia S.
Source: Chemico-Biological Interactions. Unidades: IQ, FM
Subjects: MELANOMA, ESTRESSE OXIDATIVO, PROLIFERAÇÃO CELULAR, APOPTOSE, NEUROBLASTOMA, CITOFISIOLOGIA, METÁSTASE NEOPLÁSICA
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BORGES, Beatriz E. et al. De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus. Chemico-Biological Interactions, v. 206, n. 1, p. 37-46, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.cbi.2013.08.005. Acesso em: 16 nov. 2024.
APA
Borges, B. E., Teixeira, V. R., Appel, M. H., Steclan, C. A., Rigo, F., Filipak Neto, F., et al. (2013). De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus. Chemico-Biological Interactions, 206( 1), 37-46. doi:10.1016/j.cbi.2013.08.005
NLM
Borges BE, Teixeira VR, Appel MH, Steclan CA, Rigo F, Filipak Neto F, Ferreira AM da C, Chammas R, Zanata SM, Nakao LS. De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus [Internet]. Chemico-Biological Interactions. 2013 ; 206( 1): 37-46.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.cbi.2013.08.005
Vancouver
Borges BE, Teixeira VR, Appel MH, Steclan CA, Rigo F, Filipak Neto F, Ferreira AM da C, Chammas R, Zanata SM, Nakao LS. De novo galectin-3 expression influences the response of melanoma cells to isatin-Schiff base copper (II) complex-induced oxidative stimulus [Internet]. Chemico-Biological Interactions. 2013 ; 206( 1): 37-46.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.cbi.2013.08.005
Artigo de periodico
Differences in transcriptional effects of 1α,25 dihydroxyvitamin D3 on fibroblasts associated to breast carcinomas and from paired normal breast tissues (2013)
Campos, Laura Tojeiro; Brentani, Helena; Roela, Rosimeire Aparecida; Katayama, Maria Lucia Hirata; Lima, Leandro; Rolim, Cíntia Flores; Milani, Cíntia; Folgueira, Maria Aparecida Azevedo Koike; Brentani, Maria Mitzi
Source: The Journal of Steroid Biochemistry and Molecular Biology. Unidade: FM
Subjects: NEOPLASIAS MAMÁRIAS, PROLIFERAÇÃO CELULAR, APOPTOSE, CARCINOMA, BIOLOGIA MOLECULAR, EXPRESSÃO GÊNICA
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ABNT
CAMPOS, Laura Tojeiro et al. Differences in transcriptional effects of 1α,25 dihydroxyvitamin D3 on fibroblasts associated to breast carcinomas and from paired normal breast tissues. The Journal of Steroid Biochemistry and Molecular Biology, v. 133, n. 1, p. 12-24, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.jsbmb.2012.08.002. Acesso em: 16 nov. 2024.
APA
Campos, L. T., Brentani, H., Roela, R. A., Katayama, M. L. H., Lima, L., Rolim, C. F., et al. (2013). Differences in transcriptional effects of 1α,25 dihydroxyvitamin D3 on fibroblasts associated to breast carcinomas and from paired normal breast tissues. The Journal of Steroid Biochemistry and Molecular Biology, 133( 1), 12-24. doi:10.1016/j.jsbmb.2012.08.002
NLM
Campos LT, Brentani H, Roela RA, Katayama MLH, Lima L, Rolim CF, Milani C, Folgueira MAAK, Brentani MM. Differences in transcriptional effects of 1α,25 dihydroxyvitamin D3 on fibroblasts associated to breast carcinomas and from paired normal breast tissues [Internet]. The Journal of Steroid Biochemistry and Molecular Biology. 2013 ; 133( 1): 12-24.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.jsbmb.2012.08.002
Vancouver
Campos LT, Brentani H, Roela RA, Katayama MLH, Lima L, Rolim CF, Milani C, Folgueira MAAK, Brentani MM. Differences in transcriptional effects of 1α,25 dihydroxyvitamin D3 on fibroblasts associated to breast carcinomas and from paired normal breast tissues [Internet]. The Journal of Steroid Biochemistry and Molecular Biology. 2013 ; 133( 1): 12-24.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.jsbmb.2012.08.002
Artigo de periodico
Insulin-like growth factor-1 and 17β-estradiol down-regulate prostate apoptosis response-4 expression in MCF-7 breast cancer cells (2012)
Casolari, Debora A.; Pereira, Michelly C.; Garcia, Simone A. de Bessa; Nagai, Maria A.
Source: International Journal of Molecular Medicine. Unidade: FM
Subjects: NEOPLASIAS MAMÁRIAS (GENÉTICA;MORTALIDADE), FATORES DE CRESCIMENTO, PRÓSTATA (REGULAÇÃO;(FISIOPATOLOGIA), APOPTOSE, EXPRESSÃO GÊNICA
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ABNT
CASOLARI, Debora A. et al. Insulin-like growth factor-1 and 17β-estradiol down-regulate prostate apoptosis response-4 expression in MCF-7 breast cancer cells. International Journal of Molecular Medicine, v. 28, n. 3, p. 337-342, 2012Tradução . . Disponível em: http://www.spandidos-publications.com/10.3892/ijmm.2011.691. Acesso em: 16 nov. 2024.
APA
Casolari, D. A., Pereira, M. C., Garcia, S. A. de B., & Nagai, M. A. (2012). Insulin-like growth factor-1 and 17β-estradiol down-regulate prostate apoptosis response-4 expression in MCF-7 breast cancer cells. International Journal of Molecular Medicine, 28( 3), 337-342. doi:10.3892/ijmm.2011.691
NLM
Casolari DA, Pereira MC, Garcia SA de B, Nagai MA. Insulin-like growth factor-1 and 17β-estradiol down-regulate prostate apoptosis response-4 expression in MCF-7 breast cancer cells [Internet]. International Journal of Molecular Medicine. 2012 ; 28( 3): 337-342.[citado 2024 nov. 16 ] Available from: http://www.spandidos-publications.com/10.3892/ijmm.2011.691
Vancouver
Casolari DA, Pereira MC, Garcia SA de B, Nagai MA. Insulin-like growth factor-1 and 17β-estradiol down-regulate prostate apoptosis response-4 expression in MCF-7 breast cancer cells [Internet]. International Journal of Molecular Medicine. 2012 ; 28( 3): 337-342.[citado 2024 nov. 16 ] Available from: http://www.spandidos-publications.com/10.3892/ijmm.2011.691
Artigo de periodico
Expression of Mitochondrial Non-coding RNAs (ncRNAs) Is Modulated by High Risk Human Papillomavirus (HPV) Oncogenes (2012)
Villota, Claudio; Campos, América; Vidaurre, Soledad; Oliveira-Cruz, Luciana; Boccardo, Enrique ; Burzio, Veronica A.; Varas, Manuel; Villegas, Jaime; Valenzuela, Pablo D. T.; Socías, Miguel; Roberts, Sally; Burzio, Luis O.
Source: Journal of Biological Chemistry. Unidade: FM
Subjects: RNA, EXPRESSÃO GÊNICA, BIOMARCADORES, ONCOGENES, PROLIFERAÇÃO CELULAR, METASTASE NEOPLÁSICA, PAPILLOMAVIRUS, APOPTOSE, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
VILLOTA, Claudio et al. Expression of Mitochondrial Non-coding RNAs (ncRNAs) Is Modulated by High Risk Human Papillomavirus (HPV) Oncogenes. Journal of Biological Chemistry, v. 287, n. 25, p. 21303-21315, 2012Tradução . . Disponível em: https://doi.org/10.1074/jbc.m111.326694. Acesso em: 16 nov. 2024.
APA
Villota, C., Campos, A., Vidaurre, S., Oliveira-Cruz, L., Boccardo, E., Burzio, V. A., et al. (2012). Expression of Mitochondrial Non-coding RNAs (ncRNAs) Is Modulated by High Risk Human Papillomavirus (HPV) Oncogenes. Journal of Biological Chemistry, 287( 25), 21303-21315. doi:10.1074/jbc.m111.326694
NLM
Villota C, Campos A, Vidaurre S, Oliveira-Cruz L, Boccardo E, Burzio VA, Varas M, Villegas J, Valenzuela PDT, Socías M, Roberts S, Burzio LO. Expression of Mitochondrial Non-coding RNAs (ncRNAs) Is Modulated by High Risk Human Papillomavirus (HPV) Oncogenes [Internet]. Journal of Biological Chemistry. 2012 ; 287( 25): 21303-21315.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1074/jbc.m111.326694
Vancouver
Villota C, Campos A, Vidaurre S, Oliveira-Cruz L, Boccardo E, Burzio VA, Varas M, Villegas J, Valenzuela PDT, Socías M, Roberts S, Burzio LO. Expression of Mitochondrial Non-coding RNAs (ncRNAs) Is Modulated by High Risk Human Papillomavirus (HPV) Oncogenes [Internet]. Journal of Biological Chemistry. 2012 ; 287( 25): 21303-21315.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1074/jbc.m111.326694
Artigo de periodico
Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA (2012)
Gimenez, Marcela; Marie, Suely Kazue Nagahashi; Oba-Shinjo, Sueli Mieko; Uno, Miyuki; Silva, Roseli da; Laure, Helen Julie; Izumi, Clarice; Otake, Andreia; Chammas, Roger; Rosa, José César
Source: Proteomics. Unidades: FM, FMRP
Subjects: FOSFOPROTEÍNAS, APOPTOSE, NEOPLASIAS CEREBRAIS, BIOQUÍMICA CELULAR
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ABNT
GIMENEZ, Marcela et al. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA. Proteomics, v. 12, n. 17, p. 2632-2640, 2012Tradução . . Disponível em: https://doi.org/10.1002/pmic.201200034. Acesso em: 16 nov. 2024.
APA
Gimenez, M., Marie, S. K. N., Oba-Shinjo, S. M., Uno, M., Silva, R. da, Laure, H. J., et al. (2012). Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA. Proteomics, 12( 17), 2632-2640. doi:10.1002/pmic.201200034
NLM
Gimenez M, Marie SKN, Oba-Shinjo SM, Uno M, Silva R da, Laure HJ, Izumi C, Otake A, Chammas R, Rosa JC. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA [Internet]. Proteomics. 2012 ; 12( 17): 2632-2640.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/pmic.201200034
Vancouver
Gimenez M, Marie SKN, Oba-Shinjo SM, Uno M, Silva R da, Laure HJ, Izumi C, Otake A, Chammas R, Rosa JC. Quantitative proteomic analysis and functional studies reveal that nucleophosmin is involved in cell death in glioblastoma cell line transfected with siRNA [Internet]. Proteomics. 2012 ; 12( 17): 2632-2640.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1002/pmic.201200034
Artigo de periodico
Comprehensive Analysis of Tissue Preservation and Recording Quality from Chronic Multielectrode Implants (2011)
Freire, Marco Aurelio M.; Morya, Edgard; Faber, Jean; Santos, Jose Ronaldo; Guimaraes, Joanilson S.; Lemos, Nelson A. M.; Sameshima, Koichi; Pereira, Antonio; Ribeiro, Sidarta; Nicolelis, Miguel A. L.
Source: PLOS ONE. Unidade: FM
Subjects: PROCEDIMENTOS NEUROCIRÚRGICOS, APOPTOSE, CÉREBRO (CIRURGIA;IMUNOLOGIA), IMUNOHISTOQUÍMICA, BIOMARCADORES, DOENÇAS NEURODEGENERATIVAS (REABILITAÇÃO), ELETRODO
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ABNT
FREIRE, Marco Aurelio M. et al. Comprehensive Analysis of Tissue Preservation and Recording Quality from Chronic Multielectrode Implants. PLOS ONE, v. 6, n. 11, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0027554. Acesso em: 16 nov. 2024.
APA
Freire, M. A. M., Morya, E., Faber, J., Santos, J. R., Guimaraes, J. S., Lemos, N. A. M., et al. (2011). Comprehensive Analysis of Tissue Preservation and Recording Quality from Chronic Multielectrode Implants. PLOS ONE, 6( 11). doi:10.1371/journal.pone.0027554
NLM
Freire MAM, Morya E, Faber J, Santos JR, Guimaraes JS, Lemos NAM, Sameshima K, Pereira A, Ribeiro S, Nicolelis MAL. Comprehensive Analysis of Tissue Preservation and Recording Quality from Chronic Multielectrode Implants [Internet]. PLOS ONE. 2011 ; 6( 11):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1371/journal.pone.0027554
Vancouver
Freire MAM, Morya E, Faber J, Santos JR, Guimaraes JS, Lemos NAM, Sameshima K, Pereira A, Ribeiro S, Nicolelis MAL. Comprehensive Analysis of Tissue Preservation and Recording Quality from Chronic Multielectrode Implants [Internet]. PLOS ONE. 2011 ; 6( 11):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1371/journal.pone.0027554
Artigo de periodico
Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection (2011)
Oliveira, Felipe L.; Brand, Camila; Paula, Adelzon A.; Arcanjo, Kátia D.; Hsu, Daniel K.; Liu, Fu-Tong; Takiya, Christina M.; Borojevic, Radovan; Chammas, Roger; El-Cheikh, Márcia C.
Source: PLOS ONE. Unidade: FM
Subjects: ESQUISTOSSOMOSE MANSONI (PARASITOLOGIA), APOPTOSE, BIOMARCADORES, CAMUNDONGOS, CÉLULAS DENTRÍTICAS, INFLAMAÇÃO, CITOMETRIA DE FLUXO, FAGÓCITOS, IMUNOHISTOQUÍMICA, LINFÓCITOS
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ABNT
OLIVEIRA, Felipe L. et al. Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection. PLOS ONE, v. 6, n. 5, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0019216. Acesso em: 16 nov. 2024.
APA
Oliveira, F. L., Brand, C., Paula, A. A., Arcanjo, K. D., Hsu, D. K., Liu, F. -T., et al. (2011). Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection. PLOS ONE, 6( 5). doi:10.1371/journal.pone.0019216
NLM
Oliveira FL, Brand C, Paula AA, Arcanjo KD, Hsu DK, Liu F-T, Takiya CM, Borojevic R, Chammas R, El-Cheikh MC. Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection [Internet]. PLOS ONE. 2011 ; 6( 5):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1371/journal.pone.0019216
Vancouver
Oliveira FL, Brand C, Paula AA, Arcanjo KD, Hsu DK, Liu F-T, Takiya CM, Borojevic R, Chammas R, El-Cheikh MC. Lack of Galectin-3 Disturbs Mesenteric Lymph Node Homeostasis and B Cell Niches in the Course of Schistosoma mansoni Infection [Internet]. PLOS ONE. 2011 ; 6( 5):[citado 2024 nov. 16 ] Available from: https://doi.org/10.1371/journal.pone.0019216
Artigo de periodico
Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer (2010)
Nagai, Maria Aparecida; Gerhard, Renê; Salaorni, Sibeli; Fregnani, José Humberto Tavares; Nonogaki, Suely; Mourão Netto, Mário; Soares, Fernando Augusto
Source: International journal of oncology. Unidades: FM, FO
Subjects: GENES SUPRESSORES, APOPTOSE, NEOPLASIAS MAMÁRIAS (GENÉTICA), PROGNÓSTICO, IMUNOHISTOQUÍMICA, BIOMARCADORES
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ABNT
NAGAI, Maria Aparecida et al. Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer. International journal of oncology, v. 37, n. 1, p. 41-49, 2010Tradução . . Disponível em: https://doi.org/10.3892/ijo_00000651. Acesso em: 16 nov. 2024.
APA
Nagai, M. A., Gerhard, R., Salaorni, S., Fregnani, J. H. T., Nonogaki, S., Mourão Netto, M., & Soares, F. A. (2010). Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer. International journal of oncology, 37( 1), 41-49. doi:10.3892/ijo_00000651
NLM
Nagai MA, Gerhard R, Salaorni S, Fregnani JHT, Nonogaki S, Mourão Netto M, Soares FA. Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer [Internet]. International journal of oncology. 2010 ; 37( 1): 41-49.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3892/ijo_00000651
Vancouver
Nagai MA, Gerhard R, Salaorni S, Fregnani JHT, Nonogaki S, Mourão Netto M, Soares FA. Down-regulation of the candidate tumor suppressor gene PAR-4 is associated with poor prognosis in breast cancer [Internet]. International journal of oncology. 2010 ; 37( 1): 41-49.[citado 2024 nov. 16 ] Available from: https://doi.org/10.3892/ijo_00000651

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