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Artigo de periodico
Redox properties of the adenoside triphosphate-sensitive 'K POT. +' channel in brain mitochondria (2008)
Fornazari, Maynara; Paula, Juliana Gabriela de; Castilho, Roger Frigério; Kowaltowski, Alicia Juliana
Source: Journal of Neuroscience Research. Unidade: IQ
Subjects: MITOCÔNDRIAS, BIOQUÍMICA, ISQUEMIA, CÁLCIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FORNAZARI, Maynara et al. Redox properties of the adenoside triphosphate-sensitive 'K POT. +' channel in brain mitochondria. Journal of Neuroscience Research, v. 86, n. 7, p. 1548-1556, 2008Tradução . . Disponível em: https://doi.org/10.1002/jnr.21614. Acesso em: 15 set. 2024.
APA
Fornazari, M., Paula, J. G. de, Castilho, R. F., & Kowaltowski, A. J. (2008). Redox properties of the adenoside triphosphate-sensitive 'K POT. +' channel in brain mitochondria. Journal of Neuroscience Research, 86( 7), 1548-1556. doi:10.1002/jnr.21614
NLM
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of the adenoside triphosphate-sensitive 'K POT. +' channel in brain mitochondria [Internet]. Journal of Neuroscience Research. 2008 ;86( 7): 1548-1556.[citado 2024 set. 15 ] Available from: https://doi.org/10.1002/jnr.21614
Vancouver
Fornazari M, Paula JG de, Castilho RF, Kowaltowski AJ. Redox properties of the adenoside triphosphate-sensitive 'K POT. +' channel in brain mitochondria [Internet]. Journal of Neuroscience Research. 2008 ;86( 7): 1548-1556.[citado 2024 set. 15 ] Available from: https://doi.org/10.1002/jnr.21614
Artigo de periodico
Mode of cembranoid action on embryonic muscle acetylcholine receptor (2008)
Ulrich, Henning ; Akk, G.; Nery, Arthur Andrade; Trujillo, Cleber Augusto; Rodriguez, A. D.; Eterovic, Vesna A.
Source: Journal of Neuroscience Research. Unidade: IQ
Assunto: BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ULRICH, Henning et al. Mode of cembranoid action on embryonic muscle acetylcholine receptor. Journal of Neuroscience Research, v. 86, n. 1, p. 93-107, 2008Tradução . . Disponível em: https://doi.org/10.1002/jnr.21468. Acesso em: 15 set. 2024.
APA
Ulrich, H., Akk, G., Nery, A. A., Trujillo, C. A., Rodriguez, A. D., & Eterovic, V. A. (2008). Mode of cembranoid action on embryonic muscle acetylcholine receptor. Journal of Neuroscience Research, 86( 1), 93-107. doi:10.1002/jnr.21468
NLM
Ulrich H, Akk G, Nery AA, Trujillo CA, Rodriguez AD, Eterovic VA. Mode of cembranoid action on embryonic muscle acetylcholine receptor [Internet]. Journal of Neuroscience Research. 2008 ;86( 1): 93-107.[citado 2024 set. 15 ] Available from: https://doi.org/10.1002/jnr.21468
Vancouver
Ulrich H, Akk G, Nery AA, Trujillo CA, Rodriguez AD, Eterovic VA. Mode of cembranoid action on embryonic muscle acetylcholine receptor [Internet]. Journal of Neuroscience Research. 2008 ;86( 1): 93-107.[citado 2024 set. 15 ] Available from: https://doi.org/10.1002/jnr.21468
Artigo de periodico
Interactions between bacteriophage DNA and cationic biomimetic particles (2008)
Rosa, Heloísa; Petri, Denise Freitas Siqueira ; Carmona-Ribeiro, Ana Maria
Source: Journal of Physical Chemistry B. Unidade: IQ
Subjects: BIOQUÍMICA, DNA, BIOTECNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSA, Heloísa e PETRI, Denise Freitas Siqueira e CARMONA-RIBEIRO, Ana Maria. Interactions between bacteriophage DNA and cationic biomimetic particles. Journal of Physical Chemistry B, v. 112, n. 51, p. 16422-16430, 2008Tradução . . Disponível em: http://pubs.acs.org/doi/pdf/10.1021/jp806992f. Acesso em: 15 set. 2024.
APA
Rosa, H., Petri, D. F. S., & Carmona-Ribeiro, A. M. (2008). Interactions between bacteriophage DNA and cationic biomimetic particles. Journal of Physical Chemistry B, 112( 51), 16422-16430. Recuperado de http://pubs.acs.org/doi/pdf/10.1021/jp806992f
NLM
Rosa H, Petri DFS, Carmona-Ribeiro AM. Interactions between bacteriophage DNA and cationic biomimetic particles [Internet]. Journal of Physical Chemistry B. 2008 ; 112( 51): 16422-16430.[citado 2024 set. 15 ] Available from: http://pubs.acs.org/doi/pdf/10.1021/jp806992f
Vancouver
Rosa H, Petri DFS, Carmona-Ribeiro AM. Interactions between bacteriophage DNA and cationic biomimetic particles [Internet]. Journal of Physical Chemistry B. 2008 ; 112( 51): 16422-16430.[citado 2024 set. 15 ] Available from: http://pubs.acs.org/doi/pdf/10.1021/jp806992f
Artigo de periodico
Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells (2008)
Sartori, Adriano; Garay-Malpartida, Humberto Miguel ; Forni, Maria F.; Schumacher, Robert Ivan; Dutra, Fernando; Sogayar, Mari Cleide ; Bechara, Etelvino José Henriques
Source: Chemical Research in Toxicology. Unidades: EACH, IQ
Subjects: ESTRESSE OXIDATIVO, DIABETES MELLITUS, BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARTORI, Adriano et al. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Chemical Research in Toxicology, v. 21, n. 9, p. 1841-1850, 2008Tradução . . Disponível em: https://doi.org/10.1021/tx8001753. Acesso em: 15 set. 2024.
APA
Sartori, A., Garay-Malpartida, H. M., Forni, M. F., Schumacher, R. I., Dutra, F., Sogayar, M. C., & Bechara, E. J. H. (2008). Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells. Chemical Research in Toxicology, 21( 9), 1841-1850. doi:10.1021/tx8001753
NLM
Sartori A, Garay-Malpartida HM, Forni MF, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells [Internet]. Chemical Research in Toxicology. 2008 ; 21( 9): 1841-1850.[citado 2024 set. 15 ] Available from: https://doi.org/10.1021/tx8001753
Vancouver
Sartori A, Garay-Malpartida HM, Forni MF, Schumacher RI, Dutra F, Sogayar MC, Bechara EJH. Aminoacetone, a putative endogenous source of methylglyoxal, causes oxidative stress and death to insulin-producing RINm5f cells [Internet]. Chemical Research in Toxicology. 2008 ; 21( 9): 1841-1850.[citado 2024 set. 15 ] Available from: https://doi.org/10.1021/tx8001753

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