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Artigo de periodico
The role of Nox2-derived ROS in the development of cognitive impairment after sepsis (2014)
Hernandes, Marina S.; D'Ávila, Joana C.; Trevelin, Silvia C.; Reis, Patricia A.; Kinjo, Erika R.; Lopes, Lucia Rossetti ; Castro-Faria-Neto, Hugo C.; Cunha, Fernando de Queiroz; Britto, Luiz Roberto Giorgetti de ; Bozza, Fernando A.
Source: Journal of Neuroinflammation. Unidades: ICB, FMRP
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
HERNANDES, Marina S. et al. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, v. 11, n. 01, p. 01-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-36. Acesso em: 07 set. 2024.
APA
Hernandes, M. S., D'Ávila, J. C., Trevelin, S. C., Reis, P. A., Kinjo, E. R., Lopes, L. R., et al. (2014). The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, 11( 01), 01-12. doi:10.1186/1742-2094-11-36
NLM
Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1186/1742-2094-11-36
Vancouver
Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1186/1742-2094-11-36
Artigo de periodico
Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus (2014)
Kinjo, Erika; Higa, Guilherme Shigueto Vilar; Morya, Edgard; Valle, Angela C.; Kihara, Alexandre H.; Britto, Luiz Roberto Giorgetti de
Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
KINJO, Erika et al. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus. PLOS ONE, v. 9, n. 10, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0109149. Acesso em: 07 set. 2024.
APA
Kinjo, E., Higa, G. S. V., Morya, E., Valle, A. C., Kihara, A. H., & Britto, L. R. G. de. (2014). Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus. PLOS ONE, 9( 10), 1-12. doi:10.1371/journal.pone.0109149
NLM
Kinjo E, Higa GSV, Morya E, Valle AC, Kihara AH, Britto LRG de. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus [Internet]. PLOS ONE. 2014 ; 9( 10): 1-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1371/journal.pone.0109149
Vancouver
Kinjo E, Higa GSV, Morya E, Valle AC, Kihara AH, Britto LRG de. Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus [Internet]. PLOS ONE. 2014 ; 9( 10): 1-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1371/journal.pone.0109149
Artigo de periodico
Leptin into the ventrolateral medulla facilitates chemorespiratory response in leptin-deficient (ob/ob) mice (2014)
Bassi, M.; Furuya, W. I.; Menani, J. V.; Colombari, D. S. A.; Carmo, J. M. do; Silva, A. A. da; Hall, J. E.; Moreira, Thiago dos Santos ; Wenker, I. C.; Mulkey, D. K.; Colombari, E.
Source: Acta Physiologica. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BASSI, M. et al. Leptin into the ventrolateral medulla facilitates chemorespiratory response in leptin-deficient (ob/ob) mice. Acta Physiologica, v. 211, n. 1, p. 240-248, 2014Tradução . . Disponível em: https://doi.org/10.1111/apha.12257. Acesso em: 07 set. 2024.
APA
Bassi, M., Furuya, W. I., Menani, J. V., Colombari, D. S. A., Carmo, J. M. do, Silva, A. A. da, et al. (2014). Leptin into the ventrolateral medulla facilitates chemorespiratory response in leptin-deficient (ob/ob) mice. Acta Physiologica, 211( 1), 240-248. doi:10.1111/apha.12257
NLM
Bassi M, Furuya WI, Menani JV, Colombari DSA, Carmo JM do, Silva AA da, Hall JE, Moreira T dos S, Wenker IC, Mulkey DK, Colombari E. Leptin into the ventrolateral medulla facilitates chemorespiratory response in leptin-deficient (ob/ob) mice [Internet]. Acta Physiologica. 2014 ; 211( 1): 240-248.[citado 2024 set. 07 ] Available from: https://doi.org/10.1111/apha.12257
Vancouver
Bassi M, Furuya WI, Menani JV, Colombari DSA, Carmo JM do, Silva AA da, Hall JE, Moreira T dos S, Wenker IC, Mulkey DK, Colombari E. Leptin into the ventrolateral medulla facilitates chemorespiratory response in leptin-deficient (ob/ob) mice [Internet]. Acta Physiologica. 2014 ; 211( 1): 240-248.[citado 2024 set. 07 ] Available from: https://doi.org/10.1111/apha.12257
Trabalho de evento-resumo
Intracellular pH in dorsal root ganglion neurons (2014)
Taniguchi, E. Y.; Cassola, Antonio Carlos
Source: Resumos. Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental (FeSBE). Unidade: ICB
Subjects: FISIOLOGIA, GÂNGLIOS, NEURÔNIOS
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ABNT
TANIGUCHI, E. Y. e CASSOLA, Antonio Carlos. Intracellular pH in dorsal root ganglion neurons. 2014, Anais.. Caxambu: FeSBE, 2014. . Acesso em: 07 set. 2024.
APA
Taniguchi, E. Y., & Cassola, A. C. (2014). Intracellular pH in dorsal root ganglion neurons. In Resumos. Caxambu: FeSBE.
NLM
Taniguchi EY, Cassola AC. Intracellular pH in dorsal root ganglion neurons. Resumos. 2014 ;[citado 2024 set. 07 ]
Vancouver
Taniguchi EY, Cassola AC. Intracellular pH in dorsal root ganglion neurons. Resumos. 2014 ;[citado 2024 set. 07 ]
Artigo de periodico
Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood (2014)
Souza, K. de Picoli; Nunes, Maria Tereza
Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB
Subjects: FISIOLOGIA, HIPERTIREOIDISMO, EXPRESSÃO GÊNICA
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ABNT
SOUZA, K. de Picoli e NUNES, Maria Tereza. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood. Brazilian Journal of Medical and Biological Research, v. 47, n. 8, p. 670-678, 2014Tradução . . Disponível em: https://doi.org/10.1590/1414-431X20142875. Acesso em: 07 set. 2024.
APA
Souza, K. de P., & Nunes, M. T. (2014). Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood. Brazilian Journal of Medical and Biological Research, 47( 8), 670-678. doi:10.1590/1414-431X20142875
NLM
Souza K de P, Nunes MT. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood [Internet]. Brazilian Journal of Medical and Biological Research. 2014 ; 47( 8): 670-678.[citado 2024 set. 07 ] Available from: https://doi.org/10.1590/1414-431X20142875
Vancouver
Souza K de P, Nunes MT. Neonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood [Internet]. Brazilian Journal of Medical and Biological Research. 2014 ; 47( 8): 670-678.[citado 2024 set. 07 ] Available from: https://doi.org/10.1590/1414-431X20142875
Artigo de periodico
Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling (2014)
Couto, Gisele Kruger; Davel, Ana P; Brum, Patricia Chakur; Rossoni, Luciana Venturini
Source: Experimental physiology. Unidades: ICB, EEFE
Subjects: FISIOLOGIA, SISTEMA CARDIOVASCULAR
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ABNT
COUTO, Gisele Kruger et al. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, v. 99, n. 10, p. 1427–1438, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2014.079236. Acesso em: 07 set. 2024.
APA
Couto, G. K., Davel, A. P., Brum, P. C., & Rossoni, L. V. (2014). Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, 99( 10), 1427–1438. doi:10.1113/expphysiol.2014.079236
NLM
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 set. 07 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236
Vancouver
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 set. 07 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236
Trabalho de evento-resumo
Papel do acetato, ácido graxo de cadeia curta, na asma alérgica experimental (2014)
Mendes, E.; Terra, V. B.; Crisma, A. R.; Blotta, A. C.; Câmara, Niels Olsen Saraiva ; Lima, Wothan Tavares de ; Curi, Rui; Ferreira, C. M.
Source: Resumos. Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental (FeSBE). Unidade: ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, IMUNOLOGIA, ÁCIDOS GRAXOS, ASMA, CAMUNDONGOS, ANTI-INFLAMATÓRIOS
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ABNT
MENDES, E. et al. Papel do acetato, ácido graxo de cadeia curta, na asma alérgica experimental. 2014, Anais.. Caxambu: FeSBE, 2014. . Acesso em: 07 set. 2024.
APA
Mendes, E., Terra, V. B., Crisma, A. R., Blotta, A. C., Câmara, N. O. S., Lima, W. T. de, et al. (2014). Papel do acetato, ácido graxo de cadeia curta, na asma alérgica experimental. In Resumos. Caxambu: FeSBE.
NLM
Mendes E, Terra VB, Crisma AR, Blotta AC, Câmara NOS, Lima WT de, Curi R, Ferreira CM. Papel do acetato, ácido graxo de cadeia curta, na asma alérgica experimental. Resumos. 2014 ;[citado 2024 set. 07 ]
Vancouver
Mendes E, Terra VB, Crisma AR, Blotta AC, Câmara NOS, Lima WT de, Curi R, Ferreira CM. Papel do acetato, ácido graxo de cadeia curta, na asma alérgica experimental. Resumos. 2014 ;[citado 2024 set. 07 ]
Artigo de periodico
Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe (2014)
Roberto Sobrinho, Cleyton; Wenker, Ian C.; Poss, Erin M.; Takakura, Ana Carolina ; Moreira, Thiago dos Santos ; Mulkey, Daniel K.
Source: Journal of Physiology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
ROBERTO SOBRINHO, Cleyton et al. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, v. 15, p. 1309-1323, 2014Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2013.268490. Acesso em: 07 set. 2024.
APA
Roberto Sobrinho, C., Wenker, I. C., Poss, E. M., Takakura, A. C., Moreira, T. dos S., & Mulkey, D. K. (2014). Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, 15, 1309-1323. doi:10.1113/jphysiol.2013.268490
NLM
Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 set. 07 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
Vancouver
Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 set. 07 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
Artigo de periodico
The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure (2014)
Ruchaya, P. J.; Antunes, Vagner Roberto ; Paton, J. F. R.; Murphy, D.; Yao, S. T.
Source: Experimental Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
RUCHAYA, P. J. et al. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure. Experimental Physiology, v. 99, n. 1, p. 111-122, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2013.075432. Acesso em: 07 set. 2024.
APA
Ruchaya, P. J., Antunes, V. R., Paton, J. F. R., Murphy, D., & Yao, S. T. (2014). The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure. Experimental Physiology, 99( 1), 111-122. doi:10.1113/expphysiol.2013.075432
NLM
Ruchaya PJ, Antunes VR, Paton JFR, Murphy D, Yao ST. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure [Internet]. Experimental Physiology. 2014 ; 99( 1): 111-122.[citado 2024 set. 07 ] Available from: https://doi.org/10.1113/expphysiol.2013.075432
Vancouver
Ruchaya PJ, Antunes VR, Paton JFR, Murphy D, Yao ST. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure [Internet]. Experimental Physiology. 2014 ; 99( 1): 111-122.[citado 2024 set. 07 ] Available from: https://doi.org/10.1113/expphysiol.2013.075432
Artigo de periodico
Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells (2014)
Martín-Rufián, Mercedes; Nascimento-Gomes, Renata; Higuero, Ana; Crisma, Amanda R.; Campos-Sandoval, José A.; Gómez-Garcia, María C.; Cardona, Carolina; Cheng, Tzuling; Lobo, Carolina; Segura, Juan A.; Alonso, Francisco J.; Szeliga, Monika; Albrecht, Jan; Curi, Rui; Márquez, Javier; Colquhoun, Alison ; DeBerardinis, Ralph J.; Matés, José M.
Source: Journal of Molecular Medicine. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA
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ABNT
MARTÍN-RUFIÁN, Mercedes et al. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, v. 92, n. 3, p. 277-290, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-013-1105-2. Acesso em: 07 set. 2024.
APA
Martín-Rufián, M., Nascimento-Gomes, R., Higuero, A., Crisma, A. R., Campos-Sandoval, J. A., Gómez-Garcia, M. C., et al. (2014). Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, 92( 3), 277-290. doi:10.1007/s00109-013-1105-2
NLM
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 set. 07 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Vancouver
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 set. 07 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Artigo de periodico
Rat retina shows robust circadian expression of clock and clock output genes in explant culture (2014)
Buonfiglio, Daniella; Malan, André; Sandu, Cristina; Jaegger, Catherine; Cipolla-Neto, José; Hicks, David; Felder-Schmittbuhl, Marie-Paule
Source: Molecular Vision. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BUONFIGLIO, Daniella et al. Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision, v. 20, p. 742-752, 2014Tradução . . Acesso em: 07 set. 2024.
APA
Buonfiglio, D., Malan, A., Sandu, C., Jaegger, C., Cipolla-Neto, J., Hicks, D., & Felder-Schmittbuhl, M. -P. (2014). Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision, 20, 742-752.
NLM
Buonfiglio D, Malan A, Sandu C, Jaegger C, Cipolla-Neto J, Hicks D, Felder-Schmittbuhl M-P. Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision. 2014 ; 20 742-752.[citado 2024 set. 07 ]
Vancouver
Buonfiglio D, Malan A, Sandu C, Jaegger C, Cipolla-Neto J, Hicks D, Felder-Schmittbuhl M-P. Rat retina shows robust circadian expression of clock and clock output genes in explant culture. Molecular Vision. 2014 ; 20 742-752.[citado 2024 set. 07 ]
Artigo de periodico
Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide (2014)
Festuccia, William Tadeu Lara ; Pouliot, Philippe; Bakan, Inan; Sabatini, David M.; Laplante, Mathieu
Source: PLOS ONE. Unidade: ICB
Subjects: FISIOLOGIA, MACRÓFAGOS, LIPOLISSACARIDEOS, INFLAMAÇÃO
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ABNT
FESTUCCIA, William Tadeu Lara et al. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide. PLOS ONE, v. 9, n. 4, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0095432. Acesso em: 07 set. 2024.
APA
Festuccia, W. T. L., Pouliot, P., Bakan, I., Sabatini, D. M., & Laplante, M. (2014). Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide. PLOS ONE, 9( 4), 1-12. doi:10.1371/journal.pone.0095432
NLM
Festuccia WTL, Pouliot P, Bakan I, Sabatini DM, Laplante M. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide [Internet]. PLOS ONE. 2014 ; 9( 4): 1-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1371/journal.pone.0095432
Vancouver
Festuccia WTL, Pouliot P, Bakan I, Sabatini DM, Laplante M. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide [Internet]. PLOS ONE. 2014 ; 9( 4): 1-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1371/journal.pone.0095432
Artigo de periodico
An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages (2014)
Moreira, Vanessa; Lomonte, Bruno; Vinolo, Marco Aurélio Ramirez; Curi, Rui; Gutiérrez, José María; Teixeira, Catarina
Source: Mediators of Inflammation. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MOREIRA, Vanessa et al. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, v. 2014, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/105879. Acesso em: 07 set. 2024.
APA
Moreira, V., Lomonte, B., Vinolo, M. A. R., Curi, R., Gutiérrez, J. M., & Teixeira, C. (2014). An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages. Mediators of Inflammation, 2014, 1-10. doi:10.1155/2014/105879
NLM
Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 set. 07 ] Available from: https://doi.org/10.1155/2014/105879
Vancouver
Moreira V, Lomonte B, Vinolo MAR, Curi R, Gutiérrez JM, Teixeira C. An Asp49 phospholipase A2 from snake venom induces cyclooxygenase-2 expression and prostaglandin E2 production via activation of NF-κB, p38MAPK, and PKC in macrophages [Internet]. Mediators of Inflammation. 2014 ; 2014 1-10.[citado 2024 set. 07 ] Available from: https://doi.org/10.1155/2014/105879
Artigo de periodico
Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites (2014)
Moraes, Paulo Alexandre; Yonamine, Caio Yogi; Pinto Junior, Danilo Correa; Esteves, João Victor Del Conti; Machado, Ubiratan Fabres ; Mori, Rosana Cristina
Source: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORAES, Paulo Alexandre et al. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, v. 114, n. 1, p. 36-44, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.07.040. Acesso em: 07 set. 2024.
APA
Moraes, P. A., Yonamine, C. Y., Pinto Junior, D. C., Esteves, J. V. D. C., Machado, U. F., & Mori, R. C. (2014). Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, 114( 1), 36-44. doi:10.1016/j.lfs.2014.07.040
NLM
Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040
Vancouver
Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040
Artigo de periodico
Evidence from simultaneous intracellular- and surface-pH transients that carbonic anhydrase IV enhances CO2 fluxes across Xenopus oocyte plasma membranes (2014)
Musa-Aziz, Raif ; Occhipinti, Rossana; Boron, Walter F.
Source: American Journal of Physiology Society. Cell Physiology. Unidade: ICB
Subjects: FISIOLOGIA, MEMBRANA PLASMÁTICA, XENOPUS
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ABNT
MUSA-AZIZ, Raif e OCCHIPINTI, Rossana e BORON, Walter F. Evidence from simultaneous intracellular- and surface-pH transients that carbonic anhydrase IV enhances CO2 fluxes across Xenopus oocyte plasma membranes. American Journal of Physiology Society. Cell Physiology, v. 307, n. 9, p. C814-C840, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00050.2014. Acesso em: 07 set. 2024.
APA
Musa-Aziz, R., Occhipinti, R., & Boron, W. F. (2014). Evidence from simultaneous intracellular- and surface-pH transients that carbonic anhydrase IV enhances CO2 fluxes across Xenopus oocyte plasma membranes. American Journal of Physiology Society. Cell Physiology, 307( 9), C814-C840. doi:10.1152/ajpcell.00050.2014
NLM
Musa-Aziz R, Occhipinti R, Boron WF. Evidence from simultaneous intracellular- and surface-pH transients that carbonic anhydrase IV enhances CO2 fluxes across Xenopus oocyte plasma membranes [Internet]. American Journal of Physiology Society. Cell Physiology. 2014 ; 307( 9): C814-C840.[citado 2024 set. 07 ] Available from: https://doi.org/10.1152/ajpcell.00050.2014
Vancouver
Musa-Aziz R, Occhipinti R, Boron WF. Evidence from simultaneous intracellular- and surface-pH transients that carbonic anhydrase IV enhances CO2 fluxes across Xenopus oocyte plasma membranes [Internet]. American Journal of Physiology Society. Cell Physiology. 2014 ; 307( 9): C814-C840.[citado 2024 set. 07 ] Available from: https://doi.org/10.1152/ajpcell.00050.2014
Artigo de periodico
Functional mapping of the circuits involved in the expression of contextual fear responses in socially defeated animals (2014)
Faturi, C. B.; Rangel Jr, Miguel José; Baldo, Marcus Vinícius Chrysóstomo ; Canteras, Newton Sabino
Source: Brain Structural Function. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FATURI, C. B. et al. Functional mapping of the circuits involved in the expression of contextual fear responses in socially defeated animals. Brain Structural Function, v. 219, n. 3, p. 931-946, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00429-013-0544-4. Acesso em: 07 set. 2024.
APA
Faturi, C. B., Rangel Jr, M. J., Baldo, M. V. C., & Canteras, N. S. (2014). Functional mapping of the circuits involved in the expression of contextual fear responses in socially defeated animals. Brain Structural Function, 219( 3), 931-946. doi:10.1007/s00429-013-0544-4
NLM
Faturi CB, Rangel Jr MJ, Baldo MVC, Canteras NS. Functional mapping of the circuits involved in the expression of contextual fear responses in socially defeated animals [Internet]. Brain Structural Function. 2014 ; 219( 3): 931-946.[citado 2024 set. 07 ] Available from: https://doi.org/10.1007/s00429-013-0544-4
Vancouver
Faturi CB, Rangel Jr MJ, Baldo MVC, Canteras NS. Functional mapping of the circuits involved in the expression of contextual fear responses in socially defeated animals [Internet]. Brain Structural Function. 2014 ; 219( 3): 931-946.[citado 2024 set. 07 ] Available from: https://doi.org/10.1007/s00429-013-0544-4
Artigo de periodico
PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats (2014)
Festuccia, William Tadeu Lara ; Blanchard, Pierre-Gilles; Belchior, Thiago; Chimin, Patrícia; Paschoal, Vivian A.; Magdalon, Juliana; Hirabara, Sandro M.; Simões, Daniel; St-Pierre, Philippe; Marette, André; Deshaies, Yves; Carpinelli, Angelo Rafael
Source: American Journal of Physiology. Endocrinology and Metabolism. Unidade: ICB
Subjects: FISIOLOGIA, RATOS, GLIC0SE, MONOSSACARIDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FESTUCCIA, William Tadeu Lara et al. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats. American Journal of Physiology. Endocrinology and Metabolism, v. 9, p. E1046-E1054, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00683.2013. Acesso em: 07 set. 2024.
APA
Festuccia, W. T. L., Blanchard, P. -G., Belchior, T., Chimin, P., Paschoal, V. A., Magdalon, J., et al. (2014). PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats. American Journal of Physiology. Endocrinology and Metabolism, 9, E1046-E1054. doi:10.1152/ajpendo.00683.2013
NLM
Festuccia WTL, Blanchard P-G, Belchior T, Chimin P, Paschoal VA, Magdalon J, Hirabara SM, Simões D, St-Pierre P, Marette A, Deshaies Y, Carpinelli AR. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 9 E1046-E1054.[citado 2024 set. 07 ] Available from: https://doi.org/10.1152/ajpendo.00683.2013
Vancouver
Festuccia WTL, Blanchard P-G, Belchior T, Chimin P, Paschoal VA, Magdalon J, Hirabara SM, Simões D, St-Pierre P, Marette A, Deshaies Y, Carpinelli AR. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 9 E1046-E1054.[citado 2024 set. 07 ] Available from: https://doi.org/10.1152/ajpendo.00683.2013
Artigo de periodico
Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes (2014)
Lehnen, Tatian Ederich; Lehnen, Alexandre Machado; Tavares, Angela Maria Vicente; Belló-Klein, Adriane; Markoski, Melissa Medeiros; Machado, Ubiratan Fabres ; Schaan, Beatriz
Source: Clinical and Experimental Pharmacology & Physiology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEHNEN, Tatian Ederich et al. Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes. Clinical and Experimental Pharmacology & Physiology, v. 41, n. 12, p. 986-994, 2014Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.12313. Acesso em: 07 set. 2024.
APA
Lehnen, T. E., Lehnen, A. M., Tavares, A. M. V., Belló-Klein, A., Markoski, M. M., Machado, U. F., & Schaan, B. (2014). Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes. Clinical and Experimental Pharmacology & Physiology, 41( 12), 986-994. doi:10.1111/1440-1681.12313
NLM
Lehnen TE, Lehnen AM, Tavares AMV, Belló-Klein A, Markoski MM, Machado UF, Schaan B. Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes [Internet]. Clinical and Experimental Pharmacology & Physiology. 2014 ; 41( 12): 986-994.[citado 2024 set. 07 ] Available from: https://doi.org/10.1111/1440-1681.12313
Vancouver
Lehnen TE, Lehnen AM, Tavares AMV, Belló-Klein A, Markoski MM, Machado UF, Schaan B. Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes [Internet]. Clinical and Experimental Pharmacology & Physiology. 2014 ; 41( 12): 986-994.[citado 2024 set. 07 ] Available from: https://doi.org/10.1111/1440-1681.12313
Artigo de periodico
The in vitro maintenance of clock genes expression within the rat pineal gland under standard and norepinephrine-synchronized stimulation (2014)
Silva, Jássica Andrade; Cipolla-Neto, José; Peliciari-Garcia, Rodrigo Antonio
Source: Neuroscience Research. Unidade: ICB
Subjects: FISIOLOGIA, GLÂNDULA PINEAL, RATOS, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Jássica Andrade e CIPOLLA-NETO, José e PELICIARI-GARCIA, Rodrigo Antonio. The in vitro maintenance of clock genes expression within the rat pineal gland under standard and norepinephrine-synchronized stimulation. Neuroscience Research, v. 81-82, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.neures.2014.03.005. Acesso em: 07 set. 2024.
APA
Silva, J. A., Cipolla-Neto, J., & Peliciari-Garcia, R. A. (2014). The in vitro maintenance of clock genes expression within the rat pineal gland under standard and norepinephrine-synchronized stimulation. Neuroscience Research, 81-82, 1-10. doi:10.1016/j.neures.2014.03.005
NLM
Silva JA, Cipolla-Neto J, Peliciari-Garcia RA. The in vitro maintenance of clock genes expression within the rat pineal gland under standard and norepinephrine-synchronized stimulation [Internet]. Neuroscience Research. 2014 ; 81-82 1-10.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.neures.2014.03.005
Vancouver
Silva JA, Cipolla-Neto J, Peliciari-Garcia RA. The in vitro maintenance of clock genes expression within the rat pineal gland under standard and norepinephrine-synchronized stimulation [Internet]. Neuroscience Research. 2014 ; 81-82 1-10.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.neures.2014.03.005
Artigo de periodico
Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis (2014)
Machado, Ubiratan Fabres ; Giannella, Maria Lúcia Cardillo Côrrea
Source: Expert Opinion on Emerging Drugs. Unidades: ICB, FM
Subjects: FISIOLOGIA, GLICOSE, DIABETES MELLITUS, HEMOGLOBINAS, MEDICAMENTO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MACHADO, Ubiratan Fabres e GIANNELLA, Maria Lúcia Cardillo Côrrea. Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis. Expert Opinion on Emerging Drugs, v. 19, n. 1, p. 5-9, 2014Tradução . . Disponível em: https://doi.org/10.1517/14728214.2014.875530. Acesso em: 07 set. 2024.
APA
Machado, U. F., & Giannella, M. L. C. C. (2014). Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis. Expert Opinion on Emerging Drugs, 19( 1), 5-9. doi:10.1517/14728214.2014.875530
NLM
Machado UF, Giannella MLCC. Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis [Internet]. Expert Opinion on Emerging Drugs. 2014 ; 19( 1): 5-9.[citado 2024 set. 07 ] Available from: https://doi.org/10.1517/14728214.2014.875530
Vancouver
Machado UF, Giannella MLCC. Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis [Internet]. Expert Opinion on Emerging Drugs. 2014 ; 19( 1): 5-9.[citado 2024 set. 07 ] Available from: https://doi.org/10.1517/14728214.2014.875530

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