Filtros : "ICB" "ABRAHAMSOHN, ISES DE ALMEIDA" Removidos: "Nunes, Maria Tereza" "MORAES, FLAVIO FAVA DE" "Coloquio da Sociedade de Microscopia Eletronica" "VACINAS" "Brasil" "Artes Médicas" Limpar

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  • Source: BioMed Research International. Unidade: ICB

    Subjects: PARASITOLOGIA, LEISHMANIA, LEISHMANIOSE CUTÂNEA

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      PIRES, Alause da Silva et al. Identification and biological characterization of Leishmania (Viannia) guyanensis isolated from a patient with tegumentary Leishmaniasis in Goiás, a nonendemic area for this species in Brazil. BioMed Research International, v. 2015, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/350764. Acesso em: 24 jun. 2024.
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      Pires, A. da S., Borges, A. F., Coelho, A. C., Dorta, M. L., Lino Junior, R. de S., Pereira, L. I. de A., et al. (2015). Identification and biological characterization of Leishmania (Viannia) guyanensis isolated from a patient with tegumentary Leishmaniasis in Goiás, a nonendemic area for this species in Brazil. BioMed Research International, 2015, 1-11. doi:10.1155/2015/350764
    • NLM

      Pires A da S, Borges AF, Coelho AC, Dorta ML, Lino Junior R de S, Pereira LI de A, Pinto SA, Oliveira AP de, Matos GG de, Abrahamsohn I de A, Uliana SRB, Lima GMC de A, Ribeiro-Dias F. Identification and biological characterization of Leishmania (Viannia) guyanensis isolated from a patient with tegumentary Leishmaniasis in Goiás, a nonendemic area for this species in Brazil [Internet]. BioMed Research International. 2015 ; 2015 1-11.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1155/2015/350764
    • Vancouver

      Pires A da S, Borges AF, Coelho AC, Dorta ML, Lino Junior R de S, Pereira LI de A, Pinto SA, Oliveira AP de, Matos GG de, Abrahamsohn I de A, Uliana SRB, Lima GMC de A, Ribeiro-Dias F. Identification and biological characterization of Leishmania (Viannia) guyanensis isolated from a patient with tegumentary Leishmaniasis in Goiás, a nonendemic area for this species in Brazil [Internet]. BioMed Research International. 2015 ; 2015 1-11.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1155/2015/350764
  • Source: Experimental Parasitology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FELIZARDO, Tânia Cristina et al. Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice. Experimental Parasitology, v. 130, n. 1, p. 48-57, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.exppara.2011.09.015. Acesso em: 24 jun. 2024.
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      Felizardo, T. C., Gaspar-Elsas, M. I. C., Lima, G. M. C. de A., & Abrahamsohn, I. de A. (2012). Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice. Experimental Parasitology, 130( 1), 48-57. doi:10.1016/j.exppara.2011.09.015
    • NLM

      Felizardo TC, Gaspar-Elsas MIC, Lima GMC de A, Abrahamsohn I de A. Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice [Internet]. Experimental Parasitology. 2012 ; 130( 1): 48-57.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.exppara.2011.09.015
    • Vancouver

      Felizardo TC, Gaspar-Elsas MIC, Lima GMC de A, Abrahamsohn I de A. Lack of signaling by IL-4 or by IL-4/IL-13 has more attenuating effects on Leishmania amazonensis dorsal skin – than on footpad-infected mice [Internet]. Experimental Parasitology. 2012 ; 130( 1): 48-57.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.exppara.2011.09.015
  • Source: PLOS ONE. Unidade: ICB

    Assunto: IMUNOLOGIA

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      ZAGO, Cláudia Augusta et al. Anti-IL-2 treatment impairs the expansion of Treg cell population during acute malaria and enhances the Th1 cell response at the chronic disease. PLOS ONE, v. 7, n. 1, p. 1-9, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0029894. Acesso em: 24 jun. 2024.
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      Zago, C. A., Bortoluci, K. R., Sardinha, L. R., Pretel, F. D., Castillo-Méndez, S. I., Rosário, A. P. F. do, et al. (2012). Anti-IL-2 treatment impairs the expansion of Treg cell population during acute malaria and enhances the Th1 cell response at the chronic disease. PLOS ONE, 7( 1), 1-9. doi:10.1371/journal.pone.0029894
    • NLM

      Zago CA, Bortoluci KR, Sardinha LR, Pretel FD, Castillo-Méndez SI, Rosário APF do, Hiyane MI, Muxel SM, Rodriguez-Málaga SM, Abrahamsohn I de A, Álvarez-Mosig JM, Lima MRD'I. Anti-IL-2 treatment impairs the expansion of Treg cell population during acute malaria and enhances the Th1 cell response at the chronic disease [Internet]. PLOS ONE. 2012 ; 7( 1): 1-9.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1371/journal.pone.0029894
    • Vancouver

      Zago CA, Bortoluci KR, Sardinha LR, Pretel FD, Castillo-Méndez SI, Rosário APF do, Hiyane MI, Muxel SM, Rodriguez-Málaga SM, Abrahamsohn I de A, Álvarez-Mosig JM, Lima MRD'I. Anti-IL-2 treatment impairs the expansion of Treg cell population during acute malaria and enhances the Th1 cell response at the chronic disease [Internet]. PLOS ONE. 2012 ; 7( 1): 1-9.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1371/journal.pone.0029894
  • Source: Microbes and Infection. Unidades: FMRP, IQ, ICB

    Subjects: DOENÇAS PARASITÁRIAS (EXPERIMENTOS), TRYPANOSOMA CRUZI, CITOCINAS

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      TORRECILHAS, Ana Claudia Trocoli et al. Trypanosoma cruzi: parasite shed vesicles increase heart parasitism and generate an intense inflammatory response. Microbes and Infection, v. 11, n. 1, p. 29-39, 2009Tradução . . Acesso em: 24 jun. 2024.
    • APA

      Torrecilhas, A. C. T., Tonelli, R. R., Pavanelli, W. R., Silva, J. S. da, Schumacher, R. I., Souza, W. de, et al. (2009). Trypanosoma cruzi: parasite shed vesicles increase heart parasitism and generate an intense inflammatory response. Microbes and Infection, 11( 1), 29-39.
    • NLM

      Torrecilhas ACT, Tonelli RR, Pavanelli WR, Silva JS da, Schumacher RI, Souza W de, Silva NLC e, Abrahamsohn I de A, Colli W, Alves MJM. Trypanosoma cruzi: parasite shed vesicles increase heart parasitism and generate an intense inflammatory response. Microbes and Infection. 2009 ; 11( 1): 29-39.[citado 2024 jun. 24 ]
    • Vancouver

      Torrecilhas ACT, Tonelli RR, Pavanelli WR, Silva JS da, Schumacher RI, Souza W de, Silva NLC e, Abrahamsohn I de A, Colli W, Alves MJM. Trypanosoma cruzi: parasite shed vesicles increase heart parasitism and generate an intense inflammatory response. Microbes and Infection. 2009 ; 11( 1): 29-39.[citado 2024 jun. 24 ]
  • Source: Parasitology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FELIZARDO, T. C. et al. Leishmania (Leishmania) amazonensis infection and dissemination in mice inoculated with stationary-phase or with purified metacyclic promastigotes. Parasitology, v. 134, n. 12, p. 1699-1707, 2007Tradução . . Disponível em: https://doi.org/10.1017/s0031182007003186. Acesso em: 24 jun. 2024.
    • APA

      Felizardo, T. C., Toma, L. S., Borges, N. B., Lima, G. M. C. A., & Abrahamsohn, I. de A. (2007). Leishmania (Leishmania) amazonensis infection and dissemination in mice inoculated with stationary-phase or with purified metacyclic promastigotes. Parasitology, 134( 12), 1699-1707. doi:10.1017/s0031182007003186
    • NLM

      Felizardo TC, Toma LS, Borges NB, Lima GMCA, Abrahamsohn I de A. Leishmania (Leishmania) amazonensis infection and dissemination in mice inoculated with stationary-phase or with purified metacyclic promastigotes [Internet]. Parasitology. 2007 ; 134( 12): 1699-1707.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1017/s0031182007003186
    • Vancouver

      Felizardo TC, Toma LS, Borges NB, Lima GMCA, Abrahamsohn I de A. Leishmania (Leishmania) amazonensis infection and dissemination in mice inoculated with stationary-phase or with purified metacyclic promastigotes [Internet]. Parasitology. 2007 ; 134( 12): 1699-1707.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1017/s0031182007003186
  • Source: Journal of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      COSTA, Vlaudia Maria Assis et al. Type I IFNs stimulate nitric oxide production and resistance to Trypanosoma cruzi infection1. Journal of Immunology, v. 177, p. 3193-3200, 2006Tradução . . Acesso em: 24 jun. 2024.
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      Costa, V. M. A., Torres, K. C. L., Mendonça, R. Z., Gresser, I., Gollob, K. J., & Abrahamsohn, I. de A. (2006). Type I IFNs stimulate nitric oxide production and resistance to Trypanosoma cruzi infection1. Journal of Immunology, 177, 3193-3200.
    • NLM

      Costa VMA, Torres KCL, Mendonça RZ, Gresser I, Gollob KJ, Abrahamsohn I de A. Type I IFNs stimulate nitric oxide production and resistance to Trypanosoma cruzi infection1. Journal of Immunology. 2006 ; 177 3193-3200.[citado 2024 jun. 24 ]
    • Vancouver

      Costa VMA, Torres KCL, Mendonça RZ, Gresser I, Gollob KJ, Abrahamsohn I de A. Type I IFNs stimulate nitric oxide production and resistance to Trypanosoma cruzi infection1. Journal of Immunology. 2006 ; 177 3193-3200.[citado 2024 jun. 24 ]
  • Source: Microbes and Infection. Unidade: ICB

    Assunto: IMUNOLOGIA

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      OLIVEIRA, Milton A. P. et al. Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania. Microbes and Infection, v. 7, n. 2, p. 213-223, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2004.10.013. Acesso em: 24 jun. 2024.
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      Oliveira, M. A. P., Tadokoro, C. E., Lima, G. M. C. A., Mosca, T., Vieira, L. Q., Leenen, P. J. M., & Abrahamsohn, I. A. (2005). Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania. Microbes and Infection, 7( 2), 213-223. doi:10.1016/j.micinf.2004.10.013
    • NLM

      Oliveira MAP, Tadokoro CE, Lima GMCA, Mosca T, Vieira LQ, Leenen PJM, Abrahamsohn IA. Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania [Internet]. Microbes and Infection. 2005 ; 7( 2): 213-223.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.micinf.2004.10.013
    • Vancouver

      Oliveira MAP, Tadokoro CE, Lima GMCA, Mosca T, Vieira LQ, Leenen PJM, Abrahamsohn IA. Macrophages at intermediate stage of maturation produce high levels of IL-12p40 upon stimulation with Leishmania [Internet]. Microbes and Infection. 2005 ; 7( 2): 213-223.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.micinf.2004.10.013
  • Source: Reproductive Biology and Endocrinology (Online). Unidade: ICB

    Subjects: IMUNOLOGIA, HISTOLOGIA

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      ALBIERI, Andréa et al. Interferon-gamma alters the phagocytic activity of the mouse trophoblast. Reproductive Biology and Endocrinology (Online), v. 3, n. 34, 2005Tradução . . Acesso em: 24 jun. 2024.
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      Albieri, A., Hoshida, M. S., Gagioti, S. M., Leanza, E. C., Abrahamsohn, I. de A., Croy, A., et al. (2005). Interferon-gamma alters the phagocytic activity of the mouse trophoblast. Reproductive Biology and Endocrinology (Online), 3( 34).
    • NLM

      Albieri A, Hoshida MS, Gagioti SM, Leanza EC, Abrahamsohn I de A, Croy A, Ashkar AA, Bevilacqua EMAF. Interferon-gamma alters the phagocytic activity of the mouse trophoblast. Reproductive Biology and Endocrinology (Online). 2005 ; 3( 34):[citado 2024 jun. 24 ]
    • Vancouver

      Albieri A, Hoshida MS, Gagioti SM, Leanza EC, Abrahamsohn I de A, Croy A, Ashkar AA, Bevilacqua EMAF. Interferon-gamma alters the phagocytic activity of the mouse trophoblast. Reproductive Biology and Endocrinology (Online). 2005 ; 3( 34):[citado 2024 jun. 24 ]
  • Source: Clinical Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      BARROS-MAZON, Silvia de et al. Differential regulation of lymphoproliferative responses to Trypanosoma cruzi antigen in patients with the cardiac or indeterminate form of Chagas disease. Clinical Immunology, v. 111, p. 137-145, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.clim.2004.01.002. Acesso em: 24 jun. 2024.
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      Barros-Mazon, S. de, Guariento, M. E., Silva, C. A. da, Coffman, R. L., & Abrahamsohn, I. de A. (2004). Differential regulation of lymphoproliferative responses to Trypanosoma cruzi antigen in patients with the cardiac or indeterminate form of Chagas disease. Clinical Immunology, 111, 137-145. doi:10.1016/j.clim.2004.01.002
    • NLM

      Barros-Mazon S de, Guariento ME, Silva CA da, Coffman RL, Abrahamsohn I de A. Differential regulation of lymphoproliferative responses to Trypanosoma cruzi antigen in patients with the cardiac or indeterminate form of Chagas disease [Internet]. Clinical Immunology. 2004 ; 111 137-145.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.clim.2004.01.002
    • Vancouver

      Barros-Mazon S de, Guariento ME, Silva CA da, Coffman RL, Abrahamsohn I de A. Differential regulation of lymphoproliferative responses to Trypanosoma cruzi antigen in patients with the cardiac or indeterminate form of Chagas disease [Internet]. Clinical Immunology. 2004 ; 111 137-145.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.clim.2004.01.002
  • Source: Journal od Autoimmunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      TADOKORO, Carlos Eduardo et al. Experimental autoimmune encephalomyelitis can be prevented and cured by infection with Trypanosoma cruzi. Journal od Autoimmunity, v. 23, p. 103-115, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.jaut.2004.05.003. Acesso em: 24 jun. 2024.
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      Tadokoro, C. E., Vallochi, A. L., Rios, L. S., Martins, G. A., Schlesinger, D., Mosca, T., et al. (2004). Experimental autoimmune encephalomyelitis can be prevented and cured by infection with Trypanosoma cruzi. Journal od Autoimmunity, 23, 103-115. doi:10.1016/j.jaut.2004.05.003
    • NLM

      Tadokoro CE, Vallochi AL, Rios LS, Martins GA, Schlesinger D, Mosca T, Kuchroo VK, Rizzo LV, Abrahamsohn I de A. Experimental autoimmune encephalomyelitis can be prevented and cured by infection with Trypanosoma cruzi [Internet]. Journal od Autoimmunity. 2004 ; 23 103-115.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.jaut.2004.05.003
    • Vancouver

      Tadokoro CE, Vallochi AL, Rios LS, Martins GA, Schlesinger D, Mosca T, Kuchroo VK, Rizzo LV, Abrahamsohn I de A. Experimental autoimmune encephalomyelitis can be prevented and cured by infection with Trypanosoma cruzi [Internet]. Journal od Autoimmunity. 2004 ; 23 103-115.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/j.jaut.2004.05.003
  • Source: Clinical and Investigative Medicine. Conference titles: International Congress of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      GONDO, Mariza L. et al. TNFalpha production is down regulated by IL-10 in Chaga's disease patients. Clinical and Investigative Medicine. Montreal: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 24 jun. 2024. , 2004
    • APA

      Gondo, M. L., Guariento, M. E., Abrahamsohn, I. A., & Mazon, S. de B. (2004). TNFalpha production is down regulated by IL-10 in Chaga's disease patients. Clinical and Investigative Medicine. Montreal: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Gondo ML, Guariento ME, Abrahamsohn IA, Mazon S de B. TNFalpha production is down regulated by IL-10 in Chaga's disease patients. Clinical and Investigative Medicine. 2004 ;27 124B.[citado 2024 jun. 24 ]
    • Vancouver

      Gondo ML, Guariento ME, Abrahamsohn IA, Mazon S de B. TNFalpha production is down regulated by IL-10 in Chaga's disease patients. Clinical and Investigative Medicine. 2004 ;27 124B.[citado 2024 jun. 24 ]
  • Source: Infection and Immunity. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SBROGIO-ALMEIDA, M. E. et al. Host and bacterial factors affecting induction of immune responses to flagellin expressed by attenuated Salmonella vaccine strains. Infection and Immunity, v. 72, p. 2546-2555, 2004Tradução . . Acesso em: 24 jun. 2024.
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      Sbrogio-Almeida, M. E., Mosca, T., Massis, L. M., Abrahamsohn, I. de A., & Ferreira, L. C. de S. (2004). Host and bacterial factors affecting induction of immune responses to flagellin expressed by attenuated Salmonella vaccine strains. Infection and Immunity, 72, 2546-2555.
    • NLM

      Sbrogio-Almeida ME, Mosca T, Massis LM, Abrahamsohn I de A, Ferreira LC de S. Host and bacterial factors affecting induction of immune responses to flagellin expressed by attenuated Salmonella vaccine strains. Infection and Immunity. 2004 ; 72 2546-2555.[citado 2024 jun. 24 ]
    • Vancouver

      Sbrogio-Almeida ME, Mosca T, Massis LM, Abrahamsohn I de A, Ferreira LC de S. Host and bacterial factors affecting induction of immune responses to flagellin expressed by attenuated Salmonella vaccine strains. Infection and Immunity. 2004 ; 72 2546-2555.[citado 2024 jun. 24 ]
  • Source: Journal of Leukocyte Biology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      OLIVEIRA, M. A. P. et al. Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation. Journal of Leukocyte Biology, v. 74, p. 857-867, 2003Tradução . . Disponível em: https://doi.org/10.1189/jlb0302124. Acesso em: 24 jun. 2024.
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      Oliveira, M. A. P., Lima, G. M. C. de A., Shio, M. T., Leenen, P. J. M., & Abrahamsohn, I. de A. (2003). Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation. Journal of Leukocyte Biology, 74, 857-867. doi:10.1189/jlb0302124
    • NLM

      Oliveira MAP, Lima GMC de A, Shio MT, Leenen PJM, Abrahamsohn I de A. Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation [Internet]. Journal of Leukocyte Biology. 2003 ; 74 857-867.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1189/jlb0302124
    • Vancouver

      Oliveira MAP, Lima GMC de A, Shio MT, Leenen PJM, Abrahamsohn I de A. Immature macrophages derived from mouse bone marrow produce large amounts of IL-12p40 after LPS stimulation [Internet]. Journal of Leukocyte Biology. 2003 ; 74 857-867.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1189/jlb0302124
  • Source: Immunology and Cell Biology,. Unidade: ICB

    Assunto: IMUNOLOGIA

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      JACYSYN, Jacqueline F e ABRAHAMSOHN, Ises de Almeida e MACEDO, Mahasti Sahihi de. IL-4 from Th2-type cells suppresses induction of delayed-type hypersensitivity elicited shortly after immunization. Immunology and Cell Biology, v. 81, p. 424-430, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1440-1711.2003.01194.x. Acesso em: 24 jun. 2024.
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      Jacysyn, J. F., Abrahamsohn, I. de A., & Macedo, M. S. de. (2003). IL-4 from Th2-type cells suppresses induction of delayed-type hypersensitivity elicited shortly after immunization. Immunology and Cell Biology,, 81, 424-430. doi:10.1046/j.1440-1711.2003.01194.x
    • NLM

      Jacysyn JF, Abrahamsohn I de A, Macedo MS de. IL-4 from Th2-type cells suppresses induction of delayed-type hypersensitivity elicited shortly after immunization [Internet]. Immunology and Cell Biology,. 2003 ; 81 424-430.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1046/j.1440-1711.2003.01194.x
    • Vancouver

      Jacysyn JF, Abrahamsohn I de A, Macedo MS de. IL-4 from Th2-type cells suppresses induction of delayed-type hypersensitivity elicited shortly after immunization [Internet]. Immunology and Cell Biology,. 2003 ; 81 424-430.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1046/j.1440-1711.2003.01194.x
  • Source: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      SILVA, Ana Paula Galvão da e JACYSYN, Jacqueline F. e ABRAHAMSOHN, Ises de Almeida. Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response. Immunology, v. 108, p. 230-237, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2003.01571.x. Acesso em: 24 jun. 2024.
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      Silva, A. P. G. da, Jacysyn, J. F., & Abrahamsohn, I. de A. (2003). Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response. Immunology, 108, 230-237. doi:10.1046/j.1365-2567.2003.01571.x
    • NLM

      Silva APG da, Jacysyn JF, Abrahamsohn I de A. Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response [Internet]. Immunology. 2003 ; 108 230-237.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1046/j.1365-2567.2003.01571.x
    • Vancouver

      Silva APG da, Jacysyn JF, Abrahamsohn I de A. Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response [Internet]. Immunology. 2003 ; 108 230-237.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1046/j.1365-2567.2003.01571.x
  • Source: Abstract. Conference titles: Annual Meeting of Federation of Clinical Immunology Societis - (FOCIS). Unidade: ICB

    Assunto: IMUNOLOGIA

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      TADOKORO, C E. et al. The effect of t. cruzi infection over eae development. 2002, Anais.. San Francisco: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2002. . Acesso em: 24 jun. 2024.
    • APA

      Tadokoro, C. E., Vellochi, A. L., Rios, L. S., Martins, G. A., Rizzo, L. V., & Abrahamsohn, I. de A. (2002). The effect of t. cruzi infection over eae development. In Abstract. San Francisco: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Tadokoro CE, Vellochi AL, Rios LS, Martins GA, Rizzo LV, Abrahamsohn I de A. The effect of t. cruzi infection over eae development. Abstract. 2002 ;[citado 2024 jun. 24 ]
    • Vancouver

      Tadokoro CE, Vellochi AL, Rios LS, Martins GA, Rizzo LV, Abrahamsohn I de A. The effect of t. cruzi infection over eae development. Abstract. 2002 ;[citado 2024 jun. 24 ]
  • Source: Microbes and Infection. Unidade: ICB

    Subjects: FARMACOLOGIA, IMUNOLOGIA

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      BALESTIERI, Filomena M P. et al. Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages. Microbes and Infection, v. 4, p. 23-29, 2002Tradução . . Disponível em: https://doi.org/10.1016/s1286-4579(01)01505-2. Acesso em: 24 jun. 2024.
    • APA

      Balestieri, F. M. P., Queiroz, A. R. P., Scavone, C., Costa, V. M. A., Barral-Netto, M., & Abrahamsohn, I. de A. (2002). Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages. Microbes and Infection, 4, 23-29. doi:10.1016/s1286-4579(01)01505-2
    • NLM

      Balestieri FMP, Queiroz ARP, Scavone C, Costa VMA, Barral-Netto M, Abrahamsohn I de A. Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages [Internet]. Microbes and Infection. 2002 ;4 23-29.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/s1286-4579(01)01505-2
    • Vancouver

      Balestieri FMP, Queiroz ARP, Scavone C, Costa VMA, Barral-Netto M, Abrahamsohn I de A. Leishmania (L.) amazonensis-induced inhibition of nitric oxide synthesis in host macrophages [Internet]. Microbes and Infection. 2002 ;4 23-29.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1016/s1286-4579(01)01505-2
  • Source: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      GRISOTTO, M G et al. Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules. Immunology, v. 102, p. 209-217, 2001Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2001.01170.x. Acesso em: 24 jun. 2024.
    • APA

      Grisotto, M. G., Lima, M. R. D. 'I., Marinho, C. R. F., Tadokoro, C. E., Abrahamsohn, I. de A., & Alvarez, J. M. (2001). Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules. Immunology, 102, 209-217. doi:10.1046/j.1365-2567.2001.01170.x
    • NLM

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahamsohn I de A, Alvarez JM. Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules [Internet]. Immunology. 2001 ;102 209-217.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01170.x
    • Vancouver

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahamsohn I de A, Alvarez JM. Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules [Internet]. Immunology. 2001 ;102 209-217.[citado 2024 jun. 24 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01170.x
  • Source: Scandinavian Journal of Immunology. Conference titles: International Congress of Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      GRISOTTO, M et al. Most parasite specific CD8+ cells in Trypanosoma cruzi-infected chronic mice are down regulated for TCR and CD8 Molecules. Scandinavian Journal of Immunology. Stockholm: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 24 jun. 2024. , 2001
    • APA

      Grisotto, M., Lima, M. R. D. 'I., Marinho, C., Tadokoro, M., Abrahamson, I., & Mosig, J. M. A. (2001). Most parasite specific CD8+ cells in Trypanosoma cruzi-infected chronic mice are down regulated for TCR and CD8 Molecules. Scandinavian Journal of Immunology. Stockholm: Instituto de Ciências Biomédicas, Universidade de São Paulo.
    • NLM

      Grisotto M, Lima MRD'I, Marinho C, Tadokoro M, Abrahamson I, Mosig JMA. Most parasite specific CD8+ cells in Trypanosoma cruzi-infected chronic mice are down regulated for TCR and CD8 Molecules. Scandinavian Journal of Immunology. 2001 ;[citado 2024 jun. 24 ]
    • Vancouver

      Grisotto M, Lima MRD'I, Marinho C, Tadokoro M, Abrahamson I, Mosig JMA. Most parasite specific CD8+ cells in Trypanosoma cruzi-infected chronic mice are down regulated for TCR and CD8 Molecules. Scandinavian Journal of Immunology. 2001 ;[citado 2024 jun. 24 ]
  • Source: Scandinavian Journal of Immunology. Unidade: ICB

    Subjects: IMUNOLOGIA, MICROBIOLOGIA

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      GRISOTTO, Marcos Grigolin et al. Most parasite-specific CD+ cells in Trypanosoma cruzi infected chronic mice are down-regulated for TCRsalpha-beta and CDB molecules. Scandinavian Journal of Immunology, 2001Tradução . . Acesso em: 24 jun. 2024.
    • APA

      Grisotto, M. G., Lima, M. R. D. 'I., Marinho, C. R. F., Tadokoro, C. E., Abrahansohn, I. de A., & Mosig, J. M. (2001). Most parasite-specific CD+ cells in Trypanosoma cruzi infected chronic mice are down-regulated for TCRsalpha-beta and CDB molecules. Scandinavian Journal of Immunology.
    • NLM

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahansohn I de A, Mosig JM. Most parasite-specific CD+ cells in Trypanosoma cruzi infected chronic mice are down-regulated for TCRsalpha-beta and CDB molecules. Scandinavian Journal of Immunology. 2001 ;[citado 2024 jun. 24 ]
    • Vancouver

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahansohn I de A, Mosig JM. Most parasite-specific CD+ cells in Trypanosoma cruzi infected chronic mice are down-regulated for TCRsalpha-beta and CDB molecules. Scandinavian Journal of Immunology. 2001 ;[citado 2024 jun. 24 ]

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